Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma
Abstract
loading...
About
In The Last Decade
doi.org/10.1038/nm1517 →Countries where authors are citing Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma
This map shows the geographic impact of Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma more than expected).
Fields of papers citing Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma
This network shows the impact of Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1038/nm1517.