CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis

622 indexed citations

Abstract

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This paper, published in 2016, received 622 indexed citations. Written by Ting Zhang, Yan Zhang, Jin Li, Yimei Wang, Fengxiang Lv, Yuli Liu, Wen Zheng, Haibao Shang, Jun Zhang and Mao Zhang covering the research area of Molecular Biology and Pathology and Forensic Medicine. It is primarily cited by scholars working on Molecular Biology (422 citations), Cardiology and Cardiovascular Medicine (160 citations) and Pathology and Forensic Medicine (116 citations). Published in Nature Medicine.

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Countries where authors are citing CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis

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This map shows the geographic impact of CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis more than expected).

Fields of papers citing CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis

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Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis.

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This paper is also available at doi.org/10.1038/nm.4017.

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