Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2
- Journal
- Nature Communications
In The Last Decade
doi.org/10.1038/ncomms4477 →Countries where authors are citing Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2
This map shows the geographic impact of Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2 with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2 more than expected).
Fields of papers citing Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2
This network shows the impact of Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2.
About Syntenin-ALIX exosome biogenesis and budding into multivesicular bodies are controlled by ARF6 and PLD2
This paper, published in 2014, received 437 indexed citations . Written by Rania Ghossoub, Frédérique Lembo, Aude Rubio, Jérôme Bouchet, Nicolas Vitale, Josef Slavík, Miroslav Machala and Pascale Zimmermann covering the research area of Molecular Biology and Cell Biology. It is primarily cited by scholars working on Molecular Biology (406 citations), Cancer Research (231 citations) and Immunology (61 citations). Published in Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1038/ncomms4477.