Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension
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- Nature Genetics
In The Last Decade
doi.org/10.1038/79226 →Countries where authors are citing Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension
This map shows the geographic impact of Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension more than expected).
Fields of papers citing Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension
This network shows the impact of Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension.
About Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension
This paper, published in 2000, received 1.1k indexed citations . Written by Kirk B. Lane, Rajiv D. Machado, Michael W. Pauciulo, Jennifer R. Thomson, John A. Phillips, James E. Loyd, William C. Nichols and Richard C. Trembath covering the research area of Molecular Biology and Pulmonary and Respiratory Medicine. It is primarily cited by scholars working on Pulmonary and Respiratory Medicine (932 citations), Cardiology and Cardiovascular Medicine (341 citations) and Molecular Biology (281 citations). Published in Nature Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1038/79226.