Zhongsen Ma

702 total citations
19 papers, 541 citations indexed

About

Zhongsen Ma is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Oncology. According to data from OpenAlex, Zhongsen Ma has authored 19 papers receiving a total of 541 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Pulmonary and Respiratory Medicine, 7 papers in Molecular Biology and 4 papers in Oncology. Recurrent topics in Zhongsen Ma's work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (5 papers), Asthma and respiratory diseases (3 papers) and Medical Imaging and Pathology Studies (3 papers). Zhongsen Ma is often cited by papers focused on Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (5 papers), Asthma and respiratory diseases (3 papers) and Medical Imaging and Pathology Studies (3 papers). Zhongsen Ma collaborates with scholars based in China and United States. Zhongsen Ma's co-authors include Jian Fu, Rong Gao, Sreerama Shetty, Shaomin Shi, Bingdi Yan, Jinyan Yu, Yuxin Hu, Jiao Chen, Tiangang Ma and Junling Yang and has published in prestigious journals such as American Journal of Physiology-Endocrinology and Metabolism, Oncotarget and American Journal of Physiology-Lung Cellular and Molecular Physiology.

In The Last Decade

Zhongsen Ma

19 papers receiving 534 citations

Peers

Zhongsen Ma

PRM

EPIDE

IMMUN

PHYSI

Ge Jiang China

Bowen Li China

Antonietta Liotti Italy

Yonghong Shi China

Giovanna Priante Italy

Xiuzhen Li China

Reny Joseph United States

Ignacio Norambuena‐Soto Chile

Weili Liu China

Ge Jiang China

Zhongsen Ma

341 ×1.3

173 ×1.1

PRM

68 ×0.8

EPIDE

126 ×1.7

IMMUN

103 ×1.6

PHYSI

Citations per year, relative to Zhongsen Ma Zhongsen Ma (= 1×) peers Ge Jiang

Countries citing papers authored by Zhongsen Ma

Since Specialization

Citations

This map shows the geographic impact of Zhongsen Ma's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Zhongsen Ma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Zhongsen Ma more than expected).

Fields of papers citing papers by Zhongsen Ma

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Zhongsen Ma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Zhongsen Ma. The network helps show where Zhongsen Ma may publish in the future.

Co-authorship network of co-authors of Zhongsen Ma

This figure shows the co-authorship network connecting the top 25 collaborators of Zhongsen Ma. A scholar is included among the top collaborators of Zhongsen Ma based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Zhongsen Ma. Zhongsen Ma is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Yan, Bingdi, Zhongsen Ma, Shaomin Shi, et al.. (2017). Sulforaphane prevents bleomycin-induced pulmonary fibrosis in mice by inhibiting oxidative stress via nuclear factor erythroid 2-related factor-2 activation. Molecular Medicine Reports. 15(6). 4005–4014. 45 indexed citations

Shi, Shaomin, et al.. (2017). HPIP silencing inhibits TGF-β1-induced EMT in lung cancer cells. International Journal of Molecular Medicine. 39(2). 479–483. 25 indexed citations

Shi, Shaomin, Ping Tan, Bingdi Yan, et al.. (2016). ER stress and autophagy are involved in the apoptosis induced by cisplatin in human lung cancer cells. Oncology Reports. 35(5). 2606–2614. 90 indexed citations

Yan, Bingdi, Shanshan Meng, Jin Ren, et al.. (2016). Asthma control and severe exacerbations in patients with moderate or severe asthma in Jilin Province, China: a multicenter cross-sectional survey. BMC Pulmonary Medicine. 16(1). 130–130. 14 indexed citations

Ren, Jin, et al.. (2016). Approaches to prevent the patients with chronic airway diseases from exacerbation in the haze weather.. PubMed. 8(1). E1–7. 28 indexed citations

Yu, Jinyan, et al.. (2016). HDAC6 inhibition prevents TNF-α-induced caspase 3 activation in lung endothelial cell and maintains cell-cell junctions. Oncotarget. 7(34). 54714–54722. 23 indexed citations

Yu, Jinyan, et al.. (2016). Selective HDAC6 inhibition prevents TNF-α-induced lung endothelial cell barrier disruption and endotoxin-induced pulmonary edema. American Journal of Physiology-Lung Cellular and Molecular Physiology. 311(1). L39–L47. 44 indexed citations

Kong, Lingfei, Jiaming Liu, Zhongsen Ma, et al.. (2015). Randomized, Multicenter Clinical Study of Tratinterol Hydrochloride Tablets for the Treatment of Bronchial Asthma. Clinical Therapeutics. 37(6). 1248–1258. 2 indexed citations

Gao, Rong, Zhongsen Ma, Jinyan Yu, et al.. (2015). Deletion of Src family kinase Lyn aggravates endotoxin-induced lung inflammation. American Journal of Physiology-Lung Cellular and Molecular Physiology. 309(11). L1376–L1381. 8 indexed citations

10.

Gao, Rong, Zhongsen Ma, Yuxin Hu, et al.. (2015). Sirt1 restrains lung inflammasome activation in a murine model of sepsis. American Journal of Physiology-Lung Cellular and Molecular Physiology. 308(8). L847–L853. 102 indexed citations

11.

Li, Bo, Zhongsen Ma, Xuesong Xu, et al.. (2015). Multi-organ Involvement of Sweet's Syndrome: A Case Report and Literature Review. Internal Medicine. 54(3). 339–343. 7 indexed citations

12.

Ma, Tiangang, et al.. (2014). A Pedigree with Pulmonary Alveolar Microlithiasis: A Clinical Case Report and Literature Review. Cell Biochemistry and Biophysics. 70(1). 565–572. 14 indexed citations

13.

Chen, Peng, Xiuli Wang, Zhongsen Ma, et al.. (2012). Knockdown of HMGN5 Expression by RNA Interference Induces Cell Cycle Arrest in Human Lung Cancer Cells. Asian Pacific Journal of Cancer Prevention. 13(7). 3223–3228. 7 indexed citations

14.

Liu, Ling, et al.. (2012). Comparative Study of Trans‐Oral and Trans‐Tracheal Intratracheal Instillations in a Murine Model of Acute Lung Injury. The Anatomical Record. 295(9). 1513–1519. 7 indexed citations

15.

Dong, Chunling, Guifang Wang, Bo Li, et al.. (2011). Anti-asthmatic agents alleviate pulmonary edema by upregulating AQP1 and AQP5 expression in the lungs of mice with OVA-induced asthma. Respiratory Physiology & Neurobiology. 181(1). 21–28. 62 indexed citations

16.

Yang, Junling, Yi Tan, Zhongsen Ma, et al.. (2011). Angiotensin II plays a critical role in diabetic pulmonary fibrosis most likely via activation of NADPH oxidase-mediated nitrosative damage. American Journal of Physiology-Endocrinology and Metabolism. 301(1). E132–E144. 51 indexed citations

17.

Ma, Zhongsen, et al.. (2008). [Preparation and identification of monoclonal antibodies against human DcR3].. PubMed. 24(2). 139–41. 2 indexed citations

18.

Zhang, Yue, et al.. (2008). [Three cases of pulmonary alveolar microlithiasis in a family and clinical analysis].. PubMed. 31(2). 112–5. 2 indexed citations

19.

Yang, Yang, et al.. (2008). [Detection of SLC34A2 in patients with pulmonary alveolar microlithiasis and the effect of SLC34A2 on transportation of calcium and phosphate in human alveolar epithelial cells].. PubMed. 31(12). 908–11. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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