Zhenming Xu

3.8k total citations
53 papers, 2.9k citations indexed

About

Zhenming Xu is a scholar working on Immunology, Molecular Biology and Epidemiology. According to data from OpenAlex, Zhenming Xu has authored 53 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Immunology, 19 papers in Molecular Biology and 11 papers in Epidemiology. Recurrent topics in Zhenming Xu's work include T-cell and B-cell Immunology (26 papers), Immune Cell Function and Interaction (23 papers) and Ubiquitin and proteasome pathways (6 papers). Zhenming Xu is often cited by papers focused on T-cell and B-cell Immunology (26 papers), Immune Cell Function and Interaction (23 papers) and Ubiquitin and proteasome pathways (6 papers). Zhenming Xu collaborates with scholars based in United States, China and Australia. Zhenming Xu's co-authors include Paolo Casali, Hong Zan, Egest J. Pone, Thach Mai, T. S. Benedict Yen, Min Chen, Victor E. Buckwold, J H Ou, Ahmed A. Al‐Qahtani and Jinsong Zhang and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Zhenming Xu

53 papers receiving 2.8k citations

Peers

Zhenming Xu
Ulrike Seifert Germany
Laura Runkel United States
Zhonghua Lin China
Ranjit Ray United States
Kazumoto Murata Japan
Pil Soo Sung South Korea
Hans Christian Spangenberg Germany
Mina Nakagawa Japan
Isabel Fofana France
Lynn B. Dustin United States
Ulrike Seifert Germany
Zhenming Xu
Citations per year, relative to Zhenming Xu Zhenming Xu (= 1×) peers Ulrike Seifert

Countries citing papers authored by Zhenming Xu

Since Specialization
Citations

This map shows the geographic impact of Zhenming Xu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Zhenming Xu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Zhenming Xu more than expected).

Fields of papers citing papers by Zhenming Xu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Zhenming Xu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Zhenming Xu. The network helps show where Zhenming Xu may publish in the future.

Co-authorship network of co-authors of Zhenming Xu

This figure shows the co-authorship network connecting the top 25 collaborators of Zhenming Xu. A scholar is included among the top collaborators of Zhenming Xu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Zhenming Xu. Zhenming Xu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhou, Yulai, et al.. (2024). A humanized mouse that mounts mature class-switched, hypermutated and neutralizing antibody responses. Nature Immunology. 25(8). 1489–1506. 23 indexed citations
2.
Hong, Juyeong, Ji Hoon Lee, Zhao Zhang, et al.. (2022). PRC2-Mediated Epigenetic Suppression of Type I IFN-STAT2 Signaling Impairs Antitumor Immunity in Luminal Breast Cancer. Cancer Research. 82(24). 4624–4640. 14 indexed citations
3.
Afrasiabi, Ali, Hamid Alinejad‐Rokny, Mostafa Rahnama, et al.. (2022). The low abundance of CpG in the SARS-CoV-2 genome is not an evolutionarily signature of ZAP. Scientific Reports. 12(1). 2420–2420. 5 indexed citations
4.
Yan, Hui, María E. Fernández, Shuai Wu, et al.. (2020). B Cell Endosomal RAB7 Promotes TRAF6 K63 Polyubiquitination and NF-κB Activation for Antibody Class-Switching. The Journal of Immunology. 204(5). 1146–1157. 9 indexed citations
5.
Wang, Rui, Zheng Lou, Julia R. Taylor, et al.. (2016). Small Molecule Inhibition of Rab7 Impairs B Cell Class Switching and Plasma Cell Survival To Dampen the Autoantibody Response in Murine Lupus. The Journal of Immunology. 197(10). 3792–3805. 23 indexed citations
6.
Pone, Egest J., Zheng Lou, Milton L. Greenberg, et al.. (2014). B cell TLR1/2, TLR4, TLR7 and TLR9 interact in induction of class switch DNA recombination: Modulation by BCR and CD40, and relevance to T-independent antibody responses. Autoimmunity. 48(1). 1–12. 50 indexed citations
7.
Li, Guideng, Hong Zan, Zhenming Xu, & Paolo Casali. (2013). Epigenetics of the antibody response. Trends in Immunology. 34(9). 460–470. 65 indexed citations
8.
Xu, Zhenming, Hong Zan, Egest J. Pone, Thach Mai, & Paolo Casali. (2012). Immunoglobulin class-switch DNA recombination: induction, targeting and beyond. Nature reviews. Immunology. 12(7). 517–531. 300 indexed citations
9.
Mai, Thach, Hong Zan, Jinsong Zhang, et al.. (2011). Estrogen receptors bind to and activate the promoter of the HoxC4 gene to potentiate HoxC4-mediated AID induction, immunoglobulin class-switch DNA recombination and somatic hypermutation (62.8). The Journal of Immunology. 186(1_Supplement). 62.8–62.8. 1 indexed citations
10.
11.
Pone, Egest J., Hong Zan, Jinsong Zhang, et al.. (2010). Toll-Like Receptors and B-Cell Receptors Synergize to Induce Immunoglobulin Class-Switch DNA Recombination: Relevance to Microbial Antibody Responses. Critical Reviews in Immunology. 30(1). 1–29. 107 indexed citations
12.
Xu, Zhenming, Zsolt Zoltán Fülöp, Guikai Wu, et al.. (2010). 14-3-3 adaptor proteins recruit AID to 5′-AGCT-3′–rich switch regions for class switch recombination. Nature Structural & Molecular Biology. 17(9). 1124–1135. 111 indexed citations
13.
Al‐Qahtani, Ahmed A., Zhenming Xu, Hong Zan, Craig M. Walsh, & Paolo Casali. (2008). A role for DRAK2 in the germinal center reaction and the antibody response. Autoimmunity. 41(5). 341–352. 9 indexed citations
14.
Xu, Zhenming, Hong Zan, Zsuzsanna Pál, & Paolo Casali. (2007). DNA Replication to Aid Somatic Hypermutation. Advances in experimental medicine and biology. 596. 111–127. 7 indexed citations
15.
Xu, Zhenming, Egest J. Pone, Ahmed A. Al‐Qahtani, et al.. (2007). Regulation of aicda Expression and AID Activity: Relevance to Somatic Hypermutation and Class Switch DNA Recombination. Critical Reviews in Immunology. 27(4). 367–397. 79 indexed citations
16.
Casali, Paolo, Zsuzsanna Pál, Zhenming Xu, & Hong Zan. (2006). DNA repair in antibody somatic hypermutation. Trends in Immunology. 27(7). 313–321. 61 indexed citations
17.
Xu, Zhenming, Zsolt Zoltán Fülöp, Yuan Zhong, et al.. (2005). DNA Lesions and Repair in Immunoglobulin Class Switch Recombination and Somatic Hypermutation. Annals of the New York Academy of Sciences. 1050(1). 146–162. 41 indexed citations
18.
Hu, Ronggui, Jun Sheng, Xin Qi, et al.. (2005). The N-end rule pathway as a nitric oxide sensor controlling the levels of multiple regulators. Nature. 437(7061). 981–986. 247 indexed citations
19.
Zan, Hong, Naoko Shima, Zhenming Xu, et al.. (2005). The translesion DNA polymerase θ plays a dominant role in immunoglobulin gene somatic hypermutation. The EMBO Journal. 24(21). 3757–3769. 102 indexed citations
20.
Kojima, Hiroshige, Kelly Kaita, Zhenming Xu, et al.. (2003). The absence of up-regulation of telomerase activity during regeneration after partial hepatectomy in hepatitis B virus X gene transgenic mice. Journal of Hepatology. 39(2). 262–268. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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