Zeynab Najafova

1.1k total citations
17 papers, 703 citations indexed

About

Zeynab Najafova is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Zeynab Najafova has authored 17 papers receiving a total of 703 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 4 papers in Oncology and 2 papers in Cancer Research. Recurrent topics in Zeynab Najafova's work include Ubiquitin and proteasome pathways (5 papers), Epigenetics and DNA Methylation (5 papers) and Protein Degradation and Inhibitors (5 papers). Zeynab Najafova is often cited by papers focused on Ubiquitin and proteasome pathways (5 papers), Epigenetics and DNA Methylation (5 papers) and Protein Degradation and Inhibitors (5 papers). Zeynab Najafova collaborates with scholars based in Germany, United States and United Kingdom. Zeynab Najafova's co-authors include Steven A. Johnsen, Sankari Nagarajan, Tareq Hossan, Stefan Knapp, Matthias Dobbelstein, Upasana Bedi, Florian Wegwitz, Vijayalakshmi Kari, Eric Hesse and Moustapha Kassem and has published in prestigious journals such as Nucleic Acids Research, Molecular Cell and Cancer Research.

In The Last Decade

Zeynab Najafova

15 papers receiving 701 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Zeynab Najafova Germany	13	637	177	66	60	57	17	703
Brendon Ladd United States	11	489 0.8×	109 0.6×	106 1.6×	63 1.1×	31 0.5×	19	584
Stacey M. Frumm United States	7	588 0.9×	126 0.7×	123 1.9×	44 0.7×	130 2.3×	12	724
Senthilkumar Cinghu United States	12	706 1.1×	136 0.8×	156 2.4×	54 0.9×	38 0.7×	16	842
Julie Ross Canada	10	373 0.6×	86 0.5×	51 0.8×	39 0.7×	67 1.2×	14	497
Roel H. Wilting Netherlands	4	443 0.7×	130 0.7×	86 1.3×	31 0.5×	28 0.5×	4	547
Bandana Sharma United States	8	662 1.0×	283 1.6×	100 1.5×	58 1.0×	37 0.6×	16	833
Edmond Chipumuro United States	9	707 1.1×	296 1.7×	112 1.7×	57 0.9×	35 0.6×	14	930
Sarah Bailey United States	10	585 0.9×	146 0.8×	128 1.9×	130 2.2×	40 0.7×	16	667
Zongling Ji United Kingdom	12	491 0.8×	117 0.7×	62 0.9×	52 0.9×	17 0.3×	20	596
Robyn T. Sussman United States	11	331 0.5×	160 0.9×	106 1.6×	44 0.7×	16 0.3×	24	468

Countries citing papers authored by Zeynab Najafova

Since Specialization

Citations

This map shows the geographic impact of Zeynab Najafova's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Zeynab Najafova with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Zeynab Najafova more than expected).

Fields of papers citing papers by Zeynab Najafova

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Zeynab Najafova. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Zeynab Najafova. The network helps show where Zeynab Najafova may publish in the future.

Co-authorship network of co-authors of Zeynab Najafova

This figure shows the co-authorship network connecting the top 25 collaborators of Zeynab Najafova. A scholar is included among the top collaborators of Zeynab Najafova based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Zeynab Najafova. Zeynab Najafova is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Bernardini, Chiara, Zeynab Najafova, Steven A. Johnsen, et al.. (2025). CRISPR GENome and epigenome engineering improves loss-of-function genetic-screening approaches. Cell Reports Methods. 5(6). 101078–101078.

Ge, Wei, et al.. (2025). PHF19 drives the formation of PRC2 clusters to enhance motility in TNBC cells. Cell Reports. 44(10). 116391–116391.

Ahmad, Mubashir, Melanie Haffner‐Luntzer, Astrid Schoppa, et al.. (2024). Mechanical induction of osteoanabolic Wnt1 promotes osteoblast differentiation via Plat. The FASEB Journal. 38(4). e23489–e23489. 2 indexed citations

Kutschat, Ana P., Feda H. Hamdan, Xin Wang, et al.. (2021). STIM1 Mediates Calcium-Dependent Epigenetic Reprogramming in Pancreatic Cancer. Cancer Research. 81(11). 2943–2955. 11 indexed citations

Najafova, Zeynab, Peng Liu, Florian Wegwitz, et al.. (2020). RNF40 exerts stage-dependent functions in differentiating osteoblasts and is essential for bone cell crosstalk. Cell Death and Differentiation. 28(2). 700–714. 19 indexed citations

Piquereau, Jérôme, Lydie Nadal‐Desbarats, Sandra Même, et al.. (2019). Novel role of Tieg1 in muscle metabolism and mitochondrial oxidative capacities. Acta Physiologica. 228(3). e13394–e13394. 16 indexed citations

Kari, Vijayalakshmi, Julia Kitz, Frank Krämer, et al.. (2019). The histone methyltransferase DOT1L is required for proper DNA damage response, DNA repair, and modulates chemotherapy responsiveness. Clinical Epigenetics. 11(1). 4–4. 49 indexed citations

Dickmanns, Antje, et al.. (2018). CDK4 inhibition diminishes p53 activation by MDM2 antagonists. Cell Death and Disease. 9(9). 918–918. 23 indexed citations

Xie, Wanhua, Sankari Nagarajan, Simon J. Baumgart, et al.. (2017). RNF40 regulates gene expression in an epigenetic context-dependent manner. Genome biology. 18(1). 32–32. 42 indexed citations

10.

Mishra, Vivek Kumar, Florian Wegwitz, Robyn Laura Kosinsky, et al.. (2017). Histone deacetylase class-I inhibition promotes epithelial gene expression in pancreatic cancer cells in a BRD4- and MYC-dependent manner. Nucleic Acids Research. 45(11). 6334–6349. 65 indexed citations

11.

Baumgart, Simon J., Zeynab Najafova, Tareq Hossan, et al.. (2017). CHD1 regulates cell fate determination by activation of differentiation-induced genes. Nucleic Acids Research. 45(13). 7722–7735. 24 indexed citations

12.

Najafova, Zeynab, Roberto Tirado-Magallanes, Malayannan Subramaniam, et al.. (2016). BRD4 localization to lineage-specific enhancers is associated with a distinct transcription factor repertoire. Nucleic Acids Research. 45(1). 127–141. 77 indexed citations

13.

Nagarajan, Sankari, Upasana Bedi, Feda H. Hamdan, et al.. (2016). BRD4 promotes p63 and GRHL3 expression downstream of FOXO in mammary epithelial cells. Nucleic Acids Research. 45(6). gkw1276–gkw1276. 24 indexed citations

14.

Wienken, Magdalena, et al.. (2016). Cooperation of Nutlin-3a and a Wip1 inhibitor to induce p53 activity. Oncotarget. 7(22). 31623–31638. 37 indexed citations

15.

Wienken, Magdalena, Antje Dickmanns, Alice Nemajerová, et al.. (2015). MDM2 Associates with Polycomb Repressor Complex 2 and Enhances Stemness-Promoting Chromatin Modifications Independent of p53. Molecular Cell. 61(1). 68–83. 76 indexed citations

16.

Nagarajan, Sankari, Tareq Hossan, Malik Alawi, et al.. (2014). Bromodomain Protein BRD4 Is Required for Estrogen Receptor-Dependent Enhancer Activation and Gene Transcription. Cell Reports. 8(2). 460–469. 130 indexed citations

17.

Karpiuk, Oleksandra, Zeynab Najafova, Frank Krämer, et al.. (2012). The Histone H2B Monoubiquitination Regulatory Pathway Is Required for Differentiation of Multipotent Stem Cells. Molecular Cell. 46(5). 705–713. 108 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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