Yumiko Fujimura

6.9k total citations
10 papers, 312 citations indexed

About

Yumiko Fujimura is a scholar working on Molecular Biology, Immunology and Surgery. According to data from OpenAlex, Yumiko Fujimura has authored 10 papers receiving a total of 312 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Immunology and 3 papers in Surgery. Recurrent topics in Yumiko Fujimura's work include Gut microbiota and health (4 papers), Immune Cell Function and Interaction (3 papers) and Digestive system and related health (2 papers). Yumiko Fujimura is often cited by papers focused on Gut microbiota and health (4 papers), Immune Cell Function and Interaction (3 papers) and Digestive system and related health (2 papers). Yumiko Fujimura collaborates with scholars based in Japan, Germany and United States. Yumiko Fujimura's co-authors include Koji Hase, Daisuke Takahashi, Yuuki Obata, Yukihiro Furusawa, Hiroshi Ohno, Tatsuya Morita, Shigenobu Yonemura, Jun Kunisawa, Yutaka Nakamura and Hiroshi Kiyono and has published in prestigious journals such as SHILAP Revista de lepidopterología, The Journal of Immunology and Gastroenterology.

In The Last Decade

Yumiko Fujimura

8 papers receiving 309 citations

Peers

Yumiko Fujimura

MB

IMMUN

SURGE

GENET

FS

Udit Dalwadi Canada

Feilong Guo China

Laetitia Charrier‐Hisamuddin United States

Huan Song China

Victoria Godfrey United States

Hyun Woo South Korea

Ayatoshi Andou Japan

Benedikt Hild Germany

Pavel Filipčík New Zealand

Ruibing Yang China

Udit Dalwadi Canada

Yumiko Fujimura

276 ×1.4

MB

48 ×0.6

IMMUN

49 ×1.0

SURGE

38 ×0.9

GENET

32 ×0.8

FS

Citations per year, relative to Yumiko Fujimura Yumiko Fujimura (= 1×) peers Udit Dalwadi

Countries citing papers authored by Yumiko Fujimura

Since Specialization

Citations

This map shows the geographic impact of Yumiko Fujimura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yumiko Fujimura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yumiko Fujimura more than expected).

Fields of papers citing papers by Yumiko Fujimura

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yumiko Fujimura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yumiko Fujimura. The network helps show where Yumiko Fujimura may publish in the future.

Co-authorship network of co-authors of Yumiko Fujimura

This figure shows the co-authorship network connecting the top 25 collaborators of Yumiko Fujimura. A scholar is included among the top collaborators of Yumiko Fujimura based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yumiko Fujimura. Yumiko Fujimura is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Kimura, Shunsuke, et al.. (2025). Tear duct M cells exacerbate allergic conjunctivitis by facilitating germinal-center reactions. Mucosal Immunology. 18(3). 549–561.

2.

Fujimura, Yumiko, et al.. (2023). α-Glucosidase inhibitors boost gut immunity by inducing IgA responses in Peyer’s patches. Frontiers in Immunology. 14. 1277637–1277637. 1 indexed citations

3.

Nakamura, Yutaka, et al.. (2022). Spi-B alleviates food allergy by securing mucosal barrier and immune tolerance in the intestine. SHILAP Revista de lepidopterología. 3. 996657–996657. 1 indexed citations

4.

Akiyama, Masahiro, Hiroki Sato, Yumiko Fujimura, et al.. (2021). Seaweed Dietary Fiber Sodium Alginate Suppresses the Migration of Colonic Inflammatory Monocytes and Diet-Induced Metabolic Syndrome via the Gut Microbiota. Nutrients. 13(8). 2812–2812. 19 indexed citations

5.

Nakamura, Yutaka, Hitomi Mimuro, Jun Kunisawa, et al.. (2020). Microfold cell-dependent antigen transport alleviates infectious colitis by inducing antigen-specific cellular immunity. Mucosal Immunology. 13(4). 679–690. 29 indexed citations

6.

Yamada, Takahiro, Shingo Hino, Hideki Iijima, et al.. (2019). Mucin O-glycans facilitate symbiosynthesis to maintain gut immune homeostasis. EBioMedicine. 48. 513–525. 80 indexed citations

7.

Obata, Yuuki, Yutaka Nakamura, Yumiko Fujimura, et al.. (2019). Commensal-bacteria-derived butyrate promotes the T-cell-independent IgA response in the colon. International Immunology. 32(4). 243–258. 72 indexed citations

8.

Obata, Yuuki, Shunsuke Kimura, Gaku Nakato, et al.. (2014). Epithelial–stromal interaction via N otch signaling is essential for the full maturation of gut‐associated lymphoid tissues. EMBO Reports. 15(12). 1297–1304. 11 indexed citations

9.

Hase, Koji, Fubito Nakatsu, Kazushi Sugihara, et al.. (2013). AP-1B−Mediated Protein Sorting Regulates Polarity and Proliferation of Intestinal Epithelial Cells in Mice. Gastroenterology. 145(3). 625–635. 43 indexed citations

10.

Obata, Yuuki, Daisuke Takahashi, Masashi Ebisawa, et al.. (2012). Epithelial Cell-Intrinsic Notch Signaling Plays an Essential Role in the Maintenance of Gut Immune Homeostasis. The Journal of Immunology. 188(5). 2427–2436. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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