Yuko Nakamichi

3.7k total citations · 1 hit paper
46 papers, 2.6k citations indexed

About

Yuko Nakamichi is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Yuko Nakamichi has authored 46 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Molecular Biology, 22 papers in Oncology and 10 papers in Pathology and Forensic Medicine. Recurrent topics in Yuko Nakamichi's work include Bone Metabolism and Diseases (31 papers), Bone health and treatments (21 papers) and Vitamin D Research Studies (10 papers). Yuko Nakamichi is often cited by papers focused on Bone Metabolism and Diseases (31 papers), Bone health and treatments (21 papers) and Vitamin D Research Studies (10 papers). Yuko Nakamichi collaborates with scholars based in Japan, Austria and United States. Yuko Nakamichi's co-authors include Nobuyuki Udagawa, Naoyuki Takahashi, Yasuhiro Kobayashi, Hisataka Yasuda, Teruhito Yamashita, Masanori Koide, Midori Nakamura, Shunsuke Uehara, Shigeaki Kato and Toshihide Mizoguchi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and The Journal of Cell Biology.

In The Last Decade

Yuko Nakamichi

44 papers receiving 2.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Osteoclast differentiation by RANKL and OPG signaling pathways

2020 480 citations Nobuyuki Udagawa, Masanori Koide et al. Journal of Bone and Mineral Metabolism profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Yuko Nakamichi Japan	27	1.6k	776	468	390	319	46	2.6k
Simon W. Fox United Kingdom	23	1.5k 1.0×	946 1.2×	613 1.3×	329 0.8×	230 0.7×	40	2.5k
Sakamuri V. Reddy United States	32	1.3k 0.8×	1.0k 1.4×	402 0.9×	199 0.5×	212 0.7×	58	2.9k
Dana Gaddy United States	26	1.4k 0.9×	785 1.0×	454 1.0×	161 0.4×	267 0.8×	74	2.6k
Mikihito Hayashi Japan	17	2.3k 1.4×	1.1k 1.4×	614 1.3×	377 1.0×	196 0.6×	36	3.2k
Julia F. Charles United States	23	2.1k 1.4×	473 0.6×	322 0.7×	338 0.9×	244 0.8×	61	3.0k
Dorit Naot New Zealand	35	1.6k 1.0×	792 1.0×	776 1.7×	145 0.4×	271 0.8×	85	3.9k
Karen E. Callon New Zealand	39	1.7k 1.1×	986 1.3×	859 1.8×	216 0.6×	242 0.8×	98	4.0k
Jameel Iqbal United States	28	1.5k 1.0×	755 1.0×	833 1.8×	204 0.5×	416 1.3×	72	3.4k
Timothy Chambers United Kingdom	32	2.6k 1.6×	1.4k 1.8×	856 1.8×	433 1.1×	311 1.0×	58	4.0k
Changjun Li China	34	1.9k 1.2×	393 0.5×	399 0.9×	356 0.9×	187 0.6×	84	3.5k

Countries citing papers authored by Yuko Nakamichi

Since Specialization

Citations

This map shows the geographic impact of Yuko Nakamichi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuko Nakamichi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuko Nakamichi more than expected).

Fields of papers citing papers by Yuko Nakamichi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuko Nakamichi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuko Nakamichi. The network helps show where Yuko Nakamichi may publish in the future.

Co-authorship network of co-authors of Yuko Nakamichi

This figure shows the co-authorship network connecting the top 25 collaborators of Yuko Nakamichi. A scholar is included among the top collaborators of Yuko Nakamichi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yuko Nakamichi. Yuko Nakamichi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hiraga, Toru, Teruhito Yamashita, Toshihide Mizoguchi, et al.. (2025). Identification of novel macrophages and bone morphogenetic protein signals causing ectopic calcification and impairing muscle regeneration. iScience. 28(7). 112841–112841.

Fukuda, Shinji, Hisayo Nishida‐Fukuda, Takuma Sato, et al.. (2024). Osteoprotegerin secretion and its inhibition by RANKL in osteoblastic cells visualized using bioluminescence imaging. Bone. 191. 117319–117319.

Nakamichi, Yuko, et al.. (2023). The vitamin D receptor in osteoblastic cells but not secreted parathyroid hormone is crucial for soft tissue calcification induced by the proresorptive activity of 1,25(OH)2D3. The Journal of Steroid Biochemistry and Molecular Biology. 232. 106351–106351. 3 indexed citations

Mizuno, Takafumi, Tohru Hosoyama, Makiko Tomida, et al.. (2022). Influence of vitamin D on sarcopenia pathophysiology: A longitudinal study in humans and basic research in knockout mice. Journal of Cachexia Sarcopenia and Muscle. 13(6). 2961–2973. 31 indexed citations

Tsuruda, Toshihiro, Atsushi Yamashita, Masanori Koide, et al.. (2022). Angiotensin II Induces Aortic Rupture and Dissection in Osteoprotegerin‐Deficient Mice. Journal of the American Heart Association. 11(8). e025336–e025336. 11 indexed citations

Soga, Kohei, Shotaro Nakamura, Nobuyuki Udagawa, et al.. (2021). Effector memory CD4+T cells in mesenteric lymph nodes mediate bone loss in food-allergic enteropathy model mice, creating IL-4 dominance. Mucosal Immunology. 14(6). 1335–1346. 5 indexed citations

Udagawa, Nobuyuki, Masanori Koide, Midori Nakamura, et al.. (2020). Osteoclast differentiation by RANKL and OPG signaling pathways. Journal of Bone and Mineral Metabolism. 39(1). 19–26. 480 indexed citations breakdown →

Tsuruda, Toshihiro, Nobuyuki Udagawa, Yuko Nakamichi, et al.. (2019). Blockade of the angiotensin II type 1 receptor increases bone mineral density and left ventricular contractility in a mouse model of juvenile Paget disease. European Journal of Pharmacology. 859. 172519–172519. 3 indexed citations

Nakamichi, Yuko, Nobuyuki Udagawa, Toshihide Mizoguchi, et al.. (2017). VDR in Osteoblast‐Lineage Cells Primarily Mediates Vitamin D Treatment‐Induced Increase in Bone Mass by Suppressing Bone Resorption. Journal of Bone and Mineral Research. 32(6). 1297–1308. 63 indexed citations

10.

Nakamichi, Yuko, Nobuyuki Udagawa, Tatsuo Suda, & Naoyuki Takahashi. (2017). Mechanisms involved in bone resorption regulated by vitamin D. The Journal of Steroid Biochemistry and Molecular Biology. 177. 70–76. 48 indexed citations

11.

Nakamichi, Yuko & Naoyuki Takahashi. (2015). [Current Topics on Vitamin D. The role of active forms of vitamin D in regulation of bone remodeling].. PubMed. 25(3). 395–402. 2 indexed citations

12.

Okamoto, Masanori, Nobuyuki Udagawa, Shunsuke Uehara, et al.. (2014). Noncanonical Wnt5a enhances Wnt/β-catenin signaling during osteoblastogenesis. Scientific Reports. 4(1). 4493–4493. 124 indexed citations

13.

Nakamichi, Yuko, Shunsuke Uehara, Midori Nakamura, et al.. (2013). Roles of cathelicidin‐related antimicrobial peptide in murine osteoclastogenesis. Immunology. 140(3). 344–351. 28 indexed citations

14.

Harada, Suguru, Toshihide Mizoguchi, Yasuhiro Kobayashi, et al.. (2011). Daily administration of eldecalcitol (ED-71), an active vitamin D analog, increases bone mineral density by suppressing RANKL expression in mouse trabecular bone. Journal of Bone and Mineral Research. 27(2). 461–473. 81 indexed citations

15.

Takahashi, Masahiro, Toshihide Mizoguchi, Shunsuke Uehara, et al.. (2008). Docetaxel inhibits bone resorption through suppression of osteoclast formation and function in different manners. Journal of Bone and Mineral Metabolism. 27(1). 24–35. 11 indexed citations

16.

Nakamichi, Yuko, Nobuyuki Udagawa, Yasuhiro Kobayashi, et al.. (2007). Osteoprotegerin Reduces the Serum Level of Receptor Activator of NF-κB Ligand Derived from Osteoblasts. The Journal of Immunology. 178(1). 192–200. 42 indexed citations

17.

Udagawa, Nobuyuki, Yohei Yamamoto, Yuko Nakamichi, Midori Nakamura, & Naoyuki Takahashi. (2007). Osteoblasts Play Important Roles in Osteoclastogenesis through Offering the Critical Microenvironment for the Action of RANKL. 43. 195–200. 2 indexed citations

18.

Yamada, Atsushi, Masamichi Takami, Tadaharu Kawawa, et al.. (2007). Interleukin‐4 inhibition of osteoclast differentiation is stronger than that of interleukin‐13 and they are equivalent for induction of osteoprotegerin production from osteoblasts. Immunology. 120(4). 573–579. 74 indexed citations

19.

Nakamichi, Yuko & Nobuyuki Udagawa. (2006). [Role of OPG in regulation of bone remodeling].. PubMed. 16(9). 1463–68. 2 indexed citations

20.

Yang, Shuhua, Naoyuki Takahashi, Teruhito Yamashita, et al.. (2005). Muramyl Dipeptide Enhances Osteoclast Formation Induced by Lipopolysaccharide, IL-1α, and TNF-α through Nucleotide-Binding Oligomerization Domain 2-Mediated Signaling in Osteoblasts. The Journal of Immunology. 175(3). 1956–1964. 64 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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