Yukiko Aikawa

968 total citations
21 papers, 719 citations indexed

About

Yukiko Aikawa is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Yukiko Aikawa has authored 21 papers receiving a total of 719 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 16 papers in Hematology and 3 papers in Genetics. Recurrent topics in Yukiko Aikawa's work include Acute Myeloid Leukemia Research (16 papers), Protein Degradation and Inhibitors (5 papers) and Epigenetics and DNA Methylation (4 papers). Yukiko Aikawa is often cited by papers focused on Acute Myeloid Leukemia Research (16 papers), Protein Degradation and Inhibitors (5 papers) and Epigenetics and DNA Methylation (4 papers). Yukiko Aikawa collaborates with scholars based in Japan, United Kingdom and United States. Yukiko Aikawa's co-authors include Issay Kitabayashi, Takuo Katsumoto, Shinobu Ueda, Takahiro Ochiya, Atsushi Iwama, Hitoshi Ichikawa, Haruhiko Koseki, Nobuyuki Takakura, M. Lienhard Schmitz and Kazutsune Yamagata and has published in prestigious journals such as Journal of Biological Chemistry, Nature Medicine and Genes & Development.

In The Last Decade

Yukiko Aikawa

19 papers receiving 708 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yukiko Aikawa Japan 11 582 216 103 85 66 21 719
Luipa Khandker United States 7 419 0.7× 169 0.8× 78 0.8× 54 0.6× 44 0.7× 9 547
Miao-Chia Lo United States 15 560 1.0× 164 0.8× 47 0.5× 54 0.6× 60 0.9× 19 693
Takuo Katsumoto Japan 15 721 1.2× 264 1.2× 134 1.3× 171 2.0× 96 1.5× 25 941
Ikue Tai Japan 2 401 0.7× 104 0.5× 166 1.6× 75 0.9× 73 1.1× 2 507
Tetsuharu Shinjyo Japan 10 453 0.8× 131 0.6× 181 1.8× 157 1.8× 88 1.3× 13 703
Silvia Álvarez United States 7 747 1.3× 207 1.0× 254 2.5× 152 1.8× 95 1.4× 8 967
Todd Ashworth United States 12 622 1.1× 175 0.8× 134 1.3× 131 1.5× 60 0.9× 13 851
Maurizio Trubia Italy 16 303 0.5× 155 0.7× 91 0.9× 52 0.6× 107 1.6× 18 615
Ilhem Guernah United States 5 690 1.2× 144 0.7× 175 1.7× 82 1.0× 199 3.0× 6 835
Patricia S. Hähnel Germany 10 297 0.5× 112 0.5× 152 1.5× 69 0.8× 58 0.9× 13 513

Countries citing papers authored by Yukiko Aikawa

Since Specialization
Citations

This map shows the geographic impact of Yukiko Aikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yukiko Aikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yukiko Aikawa more than expected).

Fields of papers citing papers by Yukiko Aikawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yukiko Aikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yukiko Aikawa. The network helps show where Yukiko Aikawa may publish in the future.

Co-authorship network of co-authors of Yukiko Aikawa

This figure shows the co-authorship network connecting the top 25 collaborators of Yukiko Aikawa. A scholar is included among the top collaborators of Yukiko Aikawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yukiko Aikawa. Yukiko Aikawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kuroki, Yoko, et al.. (2025). CTCF Is Essential for the Development and Maintenance of CALM ‐ AF10 ‐Induced Leukemia. Cancer Science. 116(11). 3016–3026.
2.
Yamagata, Kazutsune, Haruka Shinohara, Yutaka Shima, et al.. (2022). Dual inhibition of EZH1/2 induces cell cycle arrest of B cell acute lymphoblastic leukemia cells through upregulation of CDKN1C and TP53INP1. International Journal of Hematology. 117(1). 78–89. 8 indexed citations
3.
Katsumoto, Takuo, Yoko Ogawara, Kazutsune Yamagata, et al.. (2022). MOZ is critical for the development of MOZ/MLL fusion–induced leukemia through regulation of Hoxa9/Meis1 expression. Blood Advances. 6(19). 5527–5537. 7 indexed citations
4.
Yamagata, Kazutsune, et al.. (2021). Tip60 activates Hoxa9 and Meis1 expression through acetylation of H2A.Z, promoting MLL-AF10 and MLL-ENL acute myeloid leukemia. Leukemia. 35(10). 2840–2853. 15 indexed citations
5.
Shima, Haruko, Kazutsune Yamagata, Takuo Katsumoto, et al.. (2018). Ring1A and Ring1B inhibit expression of Glis2 to maintain murine MOZ-TIF2 AML stem cells. Blood. 131(16). 1833–1845. 16 indexed citations
6.
Ogawara, Yoko, Masayuki Yamaguchi, Yukiko Aikawa, et al.. (2016). The mutant IDH1 inhibitor prevents growth of glioblastoma with IDH1 mutation in patient-derived xenograft (PDX) model. 8.
7.
Ogawara, Yoko, Takuo Katsumoto, Yukiko Aikawa, et al.. (2015). IDH2 and NPM1 Mutations Cooperate to Activate Hoxa9/Meis1 and Hypoxia Pathways in Acute Myeloid Leukemia. Cancer Research. 75(10). 2005–2016. 45 indexed citations
8.
Yamagata, Kazutsune, et al.. (2015). Leukemia-Associated MLL-AF10 Fusion Maintains an Active State of Chromatin on Its Target Genes By Recruiting Tip60. Blood. 126(23). 304–304. 1 indexed citations
9.
Suzuki, Mai, Kazutsune Yamagata, Yukiko Aikawa, Toshio Watanabe, & Issay Kitabayashi. (2014). Abstract 1569: The nuclear export signal (NES) within CALM is necessary for CALM-AF10-induced leukemia. Cancer Research. 74(19_Supplement). 1569–1569. 1 indexed citations
10.
Aikawa, Yukiko, Kazutsune Yamagata, Takuo Katsumoto, et al.. (2014). Essential role of PU.1 in maintenance of mixed lineage leukemia‐associated leukemic stem cells. Cancer Science. 106(3). 227–236. 13 indexed citations
11.
Shima, Haruko, Kazutsune Yamagata, Yukiko Aikawa, et al.. (2013). Bromodomain-PHD finger protein 1 is critical for leukemogenesis associated with MOZ–TIF2 fusion. International Journal of Hematology. 99(1). 21–31. 30 indexed citations
12.
Ogawara, Yoko, Takuo Katsumoto, Yukiko Aikawa, et al.. (2013). Critical Roles Of The IDH2 Mutation In Development and Maintenance Of Acute Myeloid Leukemia. Blood. 122(21). 225–225. 1 indexed citations
13.
Aikawa, Yukiko, Takuo Katsumoto, Pu Zhang, et al.. (2010). PU.1-mediated upregulation of CSF1R is crucial for leukemia stem cell potential induced by MOZ-TIF2. Nature Medicine. 16(5). 580–585. 76 indexed citations
14.
Rokudai, Susumu, et al.. (2008). Monocytic Leukemia Zinc Finger (MOZ) Interacts with p53 to Induce p21 Expression and Cell-cycle Arrest. Journal of Biological Chemistry. 284(1). 237–244. 71 indexed citations
15.
Aikawa, Yukiko, Kyoichi Isono, Nobuyuki Takakura, et al.. (2006). Roles of HIPK1 and HIPK2 in AML1‐ and p300‐dependent transcription, hematopoiesis and blood vessel formation. The EMBO Journal. 25(17). 3955–3965. 109 indexed citations
16.
Katsumoto, Takuo, Yukiko Aikawa, Atsushi Iwama, et al.. (2006). MOZ is essential for maintenance of hematopoietic stem cells. Genes & Development. 20(10). 1321–1330. 162 indexed citations
17.
Katsumoto, Takuo, Yukiko Aikawa, Takahiro Ochiya, & Issay Kitabayashi. (2005). Roles of MOZ in Hematopoiesis.. Blood. 106(11). 2269–2269. 1 indexed citations
18.
Pandolfi, Pier Paolo, et al.. (2004). Physical and functional link of the leukemia-associated factors AML1 and PML. Blood. 105(1). 292–300. 36 indexed citations
19.
Arai, Yasuhito, Akira Tanigami, Yoshinori Murakami, et al.. (1999). A 2-Mb Sequence-Ready Contig Map and a Novel Immunoglobulin Superfamily Gene IGSF4 in the LOH Region of Chromosome 11q23.2. Genomics. 62(2). 139–146. 108 indexed citations
20.
Ueno, Yoshio, Yukiko Aikawa, Hiroki Okumura, et al.. (1997). Trichothecenes produced by Fusarium species Fn 2B. JSM Mycotoxins. 1997(45). 25–31. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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