Yuhuan Qiao

686 total citations
29 papers, 573 citations indexed

About

Yuhuan Qiao is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Yuhuan Qiao has authored 29 papers receiving a total of 573 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 12 papers in Oncology and 6 papers in Immunology. Recurrent topics in Yuhuan Qiao's work include PI3K/AKT/mTOR signaling in cancer (5 papers), Peptidase Inhibition and Analysis (5 papers) and Pregnancy and preeclampsia studies (3 papers). Yuhuan Qiao is often cited by papers focused on PI3K/AKT/mTOR signaling in cancer (5 papers), Peptidase Inhibition and Analysis (5 papers) and Pregnancy and preeclampsia studies (3 papers). Yuhuan Qiao collaborates with scholars based in China, Norway and United States. Yuhuan Qiao's co-authors include Ruixia Guo, Zhenhe Suo, Liping Han, Mengzhen Zhang, Shuang Yang, Ruth Holm, Jahn M. Nesland, Gunnar B. Kristensen, Xianlan Zhao and Yan Zhou and has published in prestigious journals such as Cancer Research, Life Sciences and Fertility and Sterility.

In The Last Decade

Yuhuan Qiao

27 papers receiving 559 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yuhuan Qiao China 16 333 181 173 87 63 29 573
Masabumi Shibuya Japan 7 400 1.2× 104 0.6× 123 0.7× 30 0.3× 40 0.6× 9 522
Maurizio Trubia Italy 16 303 0.9× 107 0.6× 91 0.5× 38 0.4× 17 0.3× 18 615
Chunping Qiu China 19 432 1.3× 282 1.6× 119 0.7× 124 1.4× 8 0.1× 31 741
M Morganti Italy 11 209 0.6× 70 0.4× 225 1.3× 60 0.7× 48 0.8× 19 511
Wen Yuan China 10 232 0.7× 137 0.8× 253 1.5× 39 0.4× 20 0.3× 19 507
Cristina Sáiz-Ladera Spain 15 363 1.1× 151 0.8× 219 1.3× 13 0.1× 26 0.4× 28 638
Julia M. Jansen Germany 13 368 1.1× 173 1.0× 296 1.7× 126 1.4× 13 0.2× 21 902
Nathalie Kersual France 7 340 1.0× 179 1.0× 227 1.3× 19 0.2× 20 0.3× 9 570
Joema Felipe Lima United States 10 264 0.8× 214 1.2× 242 1.4× 86 1.0× 4 0.1× 15 535

Countries citing papers authored by Yuhuan Qiao

Since Specialization
Citations

This map shows the geographic impact of Yuhuan Qiao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuhuan Qiao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuhuan Qiao more than expected).

Fields of papers citing papers by Yuhuan Qiao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuhuan Qiao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuhuan Qiao. The network helps show where Yuhuan Qiao may publish in the future.

Co-authorship network of co-authors of Yuhuan Qiao

This figure shows the co-authorship network connecting the top 25 collaborators of Yuhuan Qiao. A scholar is included among the top collaborators of Yuhuan Qiao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yuhuan Qiao. Yuhuan Qiao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Guo, Ruixia, et al.. (2020). LncRNA NNT-AS1 contributes to the cisplatin resistance of cervical cancer through NNT-AS1/miR-186/HMGB1 axis. Cancer Cell International. 20(1). 190–190. 35 indexed citations
2.
Zhou, Yan, Qi An, Ruixia Guo, et al.. (2017). miR424-5p functions as an anti-oncogene in cervical cancer cell growth by targeting KDM5B via the Notch signaling pathway. Life Sciences. 171. 9–15. 61 indexed citations
4.
Wang, Xinyan, et al.. (2013). [Influence of pertussis toxin on GPER-mediated activation of phosphatidylinositol 3-kinase/protein kinase B signaling induced by 17β-estradiol in endometrial carcinoma cells].. PubMed. 48(2). 129–33. 1 indexed citations
5.
Dai, Shanjun, Yuhuan Qiao, Haixia Jin, et al.. (2012). Effect of coincubation time of sperm-oocytes on fertilization, embryonic development, and subsequent pregnancy outcome. Systems Biology in Reproductive Medicine. 58(6). 348–353. 15 indexed citations
6.
Ge, Xin, et al.. (2012). The G Protein-Coupled Receptor GPR30 Mediates the Nontranscriptional Effect of Estrogen on the Activation of PI3K/Akt Pathway in Endometrial Cancer Cells. International Journal of Gynecological Cancer. 23(1). 52–59. 42 indexed citations
7.
Qin, Haixia, et al.. (2012). HDAC6 siRNA Inhibits Proliferation and Induces Apoptosis of HeLa Cells and its Related Molecular Mechanism. Asian Pacific Journal of Cancer Prevention. 13(7). 3367–3371. 10 indexed citations
8.
Wang, Lin, et al.. (2011). Expressions of sFRP1 and β-Catenin in Cervical Cancer. Journal of Medical Biochemistry. 31(1). 6–11. 1 indexed citations
9.
Hu, Fen, Wenwen Shen, Jie Zhang, et al.. (2010). BMP-6 inhibits MMP-9 expression by regulating heme oxygenase-1 in MCF-7 breast cancer cells. Journal of Cancer Research and Clinical Oncology. 137(6). 985–995. 27 indexed citations
10.
Sun, Wei, Wenwen Shen, Shuang Yang, et al.. (2010). Homo-binding character of LMO2 isoforms and their both synergic and antagonistic functions in regulating hematopoietic-related target genes. Journal of Biomedical Science. 17(1). 22–22. 7 indexed citations
11.
Hu, Fen, Chuan Wang, Jun Du, et al.. (2009). δEF1 promotes breast cancer cell proliferation through down-regulating p21 expression. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1802(2). 301–312. 18 indexed citations
12.
Guo, Ruixia, et al.. (2008). Increased staining for phosphorylated AKT and nuclear factor‐κB p65 and their relationship with prognosis in epithelial ovarian cancer. Pathology International. 58(12). 749–756. 57 indexed citations
14.
Zhang, Cuilian, Juanke Xie, Shaodi Zhang, & Yuhuan Qiao. (2008). [Effect of estradiol supplementation during the luteal phase on mouse endometrial expression of leukaemia inhibitory factor and pinopodes in controlled ovarian stimulation cycles].. PubMed. 43(12). 937–41.
15.
Zhou, Wei, et al.. (2007). [Expression and significance of Edg4 and Edg7 in the placentas of patients with hypertensive disorder complicating pregnancy].. PubMed. 42(6). 386–9. 4 indexed citations
16.
Qiao, Yuhuan, Jia Cheng, & Ruixia Guo. (2007). [Expression of phosphorylated protein kinase B and PTEN protein in ovarian epithelial cancer].. PubMed. 42(5). 325–9. 2 indexed citations
17.
Yang, Shuang, Jun Du, Zhao‐Qi Wang, et al.. (2007). BMP-6 promotes E-cadherin expression through repressing δEF1 in breast cancer cells. BMC Cancer. 7(1). 211–211. 63 indexed citations
18.
Wei, Lihui, et al.. (2006). Blockage of PI3K/PKB/P27kip1 signaling pathway can antagonize 17β-estradiol-induced Ishikawa proliferation and cell cycle progression. Chinese Medical Journal. 119(3). 242–245. 3 indexed citations
19.
Han, Liping, Yuhuan Qiao, Gunnar B. Kristensen, et al.. (2005). The clinical significance of EphA2 and Ephrin A-1 in epithelial ovarian carcinomas. Gynecologic Oncology. 99(2). 278–286. 61 indexed citations
20.
Wu, Dan, Yuhuan Qiao, Gunnar B. Kristensen, et al.. (2004). Prognostic significance of dysadherin expression in cervical squamous cell carcinoma. Pathology & Oncology Research. 10(4). 212–218. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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