Yugen Shi

688 total citations
28 papers, 552 citations indexed

About

Yugen Shi is a scholar working on Cardiology and Cardiovascular Medicine, Surgery and Cellular and Molecular Neuroscience. According to data from OpenAlex, Yugen Shi has authored 28 papers receiving a total of 552 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Cardiology and Cardiovascular Medicine, 8 papers in Surgery and 8 papers in Cellular and Molecular Neuroscience. Recurrent topics in Yugen Shi's work include Nerve injury and regeneration (5 papers), Apelin-related biomedical research (5 papers) and Neuroscience of respiration and sleep (5 papers). Yugen Shi is often cited by papers focused on Nerve injury and regeneration (5 papers), Apelin-related biomedical research (5 papers) and Neuroscience of respiration and sleep (5 papers). Yugen Shi collaborates with scholars based in China. Yugen Shi's co-authors include Hesheng Hu, Wenjuan Cheng, Jie Yin, Mei Xue, Suhua Yan, Xinran Li, Yu Wang, Suhua Yan, Na Yang and Xiaolu Li and has published in prestigious journals such as Free Radical Biology and Medicine, American Journal of Physiology-Cell Physiology and Composites Part B Engineering.

In The Last Decade

Yugen Shi

25 papers receiving 543 citations

Peers

Yugen Shi
Yunhui Du China
Robin Looft‐Wilson United States
Lauren A. Biwer United States
Hajime Funakoshi United States
Yugen Shi
Citations per year, relative to Yugen Shi Yugen Shi (= 1×) peers Hesheng Hu

Countries citing papers authored by Yugen Shi

Since Specialization
Citations

This map shows the geographic impact of Yugen Shi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yugen Shi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yugen Shi more than expected).

Fields of papers citing papers by Yugen Shi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yugen Shi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yugen Shi. The network helps show where Yugen Shi may publish in the future.

Co-authorship network of co-authors of Yugen Shi

This figure shows the co-authorship network connecting the top 25 collaborators of Yugen Shi. A scholar is included among the top collaborators of Yugen Shi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yugen Shi. Yugen Shi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Shi, Yugen, Hesheng Hu, Yu Wang, et al.. (2025). Inhibition of neuron and cardiac remodeling by microenvironment-responsive injectable hydrogels with sympatho-immune regulation properties for myocardial infarction therapy. Composites Part B Engineering. 297. 112300–112300. 5 indexed citations
2.
Jing, Yanyan, Lei Qi, Xueli Zhang, et al.. (2024). miR-let-7a inhibits sympathetic nerve remodeling after myocardial infarction by downregulating the expression of nerve growth factor. Open Medicine. 19(1). 20240975–20240975. 1 indexed citations
3.
4.
Xia, Huitang, Suhua Yan, Yugen Shi, et al.. (2024). Miro2 sulfhydration by CBS/H2S promotes human trophoblast invasion and migration via regulating mitochondria dynamics. Cell Death and Disease. 15(10). 776–776. 3 indexed citations
5.
Li, Yan, Jie Yin, Qian Liu, et al.. (2023). Protective effect of human epicardial adipose‐derived stem cells on myocardial injury driven by poly‐lactic acid nanopillar array. Biotechnology and Applied Biochemistry. 71(1). 110–122. 1 indexed citations
6.
Wang, Kang, Jie Yin, Lei Qi, et al.. (2023). lncRNA LOC100911717-targeting GAP43-mediated sympathetic remodeling after myocardial infarction in rats. Frontiers in Cardiovascular Medicine. 9. 1019435–1019435. 4 indexed citations
7.
Wang, Yu, Yanyan Jing, Hesheng Hu, et al.. (2023). m6A methyltransferase METTL3 contributes to sympathetic hyperactivity post-MI via promoting TRAF6-dependent mitochondrial ROS production. Free Radical Biology and Medicine. 209(Pt 2). 342–354. 11 indexed citations
8.
Liu, Qian, Yan Li, Yugen Shi, et al.. (2022). The protective effect of gamma aminobutyric acid B receptor activation on sympathetic nerve remodeling via the regulation of M2 macrophage polarization after myocardial infarction. Revista Portuguesa de Cardiologia. 42(2). 125–135. 6 indexed citations
9.
Qi, Lei, Ye Wang, Hui Hu, et al.. (2022). m6A methyltransferase METTL3 participated in sympathetic neural remodeling post-MI via the TRAF6/NF-κB pathway and ROS production. Journal of Molecular and Cellular Cardiology. 170. 87–99. 24 indexed citations
10.
Wang, Fan, et al.. (2021). Overexpressing microRNA-203 alleviates myocardial infarction via interacting with long non-coding RNA MIAT and mitochondrial coupling factor 6. Archives of Pharmacal Research. 44(5). 525–535. 8 indexed citations
11.
Wang, Ye, Lei Qi, Mei Xue, et al.. (2021). EphrinB2-RhoA upregulation attenuates sympathetic hyperinnervation and decreases the incidence of ventricular arrhythmia after myocardial infarction. Journal of Cardiology. 79(3). 423–431. 6 indexed citations
12.
Shi, Yugen, Yan Li, Jie Yin, et al.. (2019). A novel sympathetic neuronal GABAergic signalling system regulates NE release to prevent ventricular arrhythmias after acute myocardial infarction. Acta Physiologica. 227(2). e13315–e13315. 24 indexed citations
13.
Wang, Ye, Jie Yin, Hesheng Hu, et al.. (2019). Targeting blockade of nuclear factor-κB in the hypothalamus paraventricular nucleus to prevent cardiac sympathetic hyperinnervation post myocardial infarction. Neuroscience Letters. 707. 134319–134319. 11 indexed citations
14.
Wang, Yu, Hesheng Hu, Jie Yin, et al.. (2019). TLR4 participates in sympathetic hyperactivity Post-MI in the PVN by regulating NF-κB pathway and ROS production. Redox Biology. 24. 101186–101186. 100 indexed citations
15.
Shi, Yugen, Jie Yin, Hesheng Hu, et al.. (2018). Targeted regulation of sympathetic activity in paraventricular nucleus reduces inducible ventricular arrhythmias in rats after myocardial infarction. Journal of Cardiology. 73(1). 81–88. 9 indexed citations
16.
Pang, Li, Jie Yin, Yugen Shi, Yu Wang, & Suhua Yan. (2018). High mobility group box-1 in hypothalamic paraventricular nuclei attenuates sympathetic tone in rats at post-myocardial infarction. Cardiology Journal. 26(5). 555–563. 4 indexed citations
17.
Wang, Fan, Jie Yin, Yugen Shi, et al.. (2018). Expression of oxytocin receptor in the rat superior cervical ganglion after myocardial infarction.. PubMed. 11(2). 739–747. 4 indexed citations
18.
Yin, Jie, Li Chen, Chengdong Zhang, et al.. (2017). Role of P2X7R in the development and progression of pulmonary hypertension. Respiratory Research. 18(1). 127–127. 36 indexed citations
19.
Hu, Hesheng, Yongli Xuan, Mei Xue, et al.. (2016). Semaphorin 3A attenuates cardiac autonomic disorders and reduces inducible ventricular arrhythmias in rats with experimental myocardial infarction. BMC Cardiovascular Disorders. 16(1). 16–16. 33 indexed citations
20.
Yang, Na, Wenjuan Cheng, Hesheng Hu, et al.. (2016). Atorvastatin attenuates sympathetic hyperinnervation together with the augmentation of M2 macrophages in rats postmyocardial infarction. Cardiovascular Therapeutics. 34(4). 234–244. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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