Youki Watanabe

584 total citations
22 papers, 454 citations indexed

About

Youki Watanabe is a scholar working on Reproductive Medicine, Molecular Biology and Endocrine and Autonomic Systems. According to data from OpenAlex, Youki Watanabe has authored 22 papers receiving a total of 454 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Reproductive Medicine, 11 papers in Molecular Biology and 4 papers in Endocrine and Autonomic Systems. Recurrent topics in Youki Watanabe's work include Hypothalamic control of reproductive hormones (17 papers), Ovarian function and disorders (10 papers) and Plant Reproductive Biology (10 papers). Youki Watanabe is often cited by papers focused on Hypothalamic control of reproductive hormones (17 papers), Ovarian function and disorders (10 papers) and Plant Reproductive Biology (10 papers). Youki Watanabe collaborates with scholars based in Japan, Australia and Cambodia. Youki Watanabe's co-authors include Yoshihisa Uenoyama, Hiroko Tsukamura, Naoko Inoue, Sho Nakamura, Kana Ikegami, Kei‐ichiro Maeda, Shiori Minabe, Nahoko Ieda, Teppei Goto and Masumi Hirabayashi and has published in prestigious journals such as PLoS ONE, Endocrinology and Frontiers in Neuroendocrinology.

In The Last Decade

Youki Watanabe

21 papers receiving 450 citations

Peers

Youki Watanabe
Youki Watanabe
Citations per year, relative to Youki Watanabe Youki Watanabe (= 1×) peers Toru Ichimaru

Countries citing papers authored by Youki Watanabe

Since Specialization
Citations

This map shows the geographic impact of Youki Watanabe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Youki Watanabe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Youki Watanabe more than expected).

Fields of papers citing papers by Youki Watanabe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Youki Watanabe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Youki Watanabe. The network helps show where Youki Watanabe may publish in the future.

Co-authorship network of co-authors of Youki Watanabe

This figure shows the co-authorship network connecting the top 25 collaborators of Youki Watanabe. A scholar is included among the top collaborators of Youki Watanabe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Youki Watanabe. Youki Watanabe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Watanabe, Youki, Kinuyo Iwata, Shiori Minabe, et al.. (2023). Central injection of neuropeptide B induces luteinizing hormone release in male and female rats. Peptides. 168. 171064–171064.
2.
Minabe, Shiori, Kinuyo Iwata, Youki Watanabe, Hirotaka Ishii, & Hitoshi Ozawa. (2022). Long-term effects of prenatal undernutrition on female rat hypothalamic KNDy neurons. Endocrine Connections. 12(1). 2 indexed citations
3.
Nakamura, Sho, Youki Watanabe, Teppei Goto, et al.. (2021). Kisspeptin neurons as a key player bridging the endocrine system and sexual behavior in mammals. Frontiers in Neuroendocrinology. 64. 100952–100952. 15 indexed citations
4.
Ikegami, Kana, Youki Watanabe, Sho Nakamura, et al.. (2021). Cellular and molecular mechanisms regulating the KNDy neuronal activities to generate and modulate GnRH pulse in mammals. Frontiers in Neuroendocrinology. 64. 100968–100968. 19 indexed citations
5.
Sato, Marimo, Shiori Minabe, Fumie Magata, et al.. (2021). Morphological Analysis of the Hindbrain Glucose Sensor-Hypothalamic Neural Pathway Activated by Hindbrain Glucoprivation. Endocrinology. 162(9). 5 indexed citations
6.
Watanabe, Youki, Kana Ikegami, Sho Nakamura, et al.. (2020). Mating-induced increase in <i>Kiss1</i> mRNA expression in the anteroventral periventricular nucleus prior to an increase in LH and testosterone release in male rats. Journal of Reproduction and Development. 66(6). 579–586. 14 indexed citations
7.
Ikegami, Kana, Teppei Goto, Sho Nakamura, et al.. (2020). Conditional kisspeptin neuron-specific <i>Kiss1</i> knockout with newly generated <i>Kiss1</i>-floxed and <i>Kiss1</i>-Cre mice replicates a hypogonadal phenotype of global <i>Kiss1</i> knockout mice. Journal of Reproduction and Development. 66(4). 359–367. 22 indexed citations
8.
Ieda, Nahoko, Shiori Minabe, Kana Ikegami, et al.. (2020). GnRH(1-5), a metabolite of gonadotropin-releasing hormone, enhances luteinizing hormone release <i>via</i> activation of kisspeptin neurons in female rats. Endocrine Journal. 67(4). 409–418. 20 indexed citations
9.
Minabe, Shiori, Marimo Sato, Naoko Inoue, et al.. (2019). Neonatal Estrogen Causes Irreversible Male Infertility via Specific Suppressive Action on Hypothalamic Kiss1 Neurons. Endocrinology. 160(5). 1223–1233. 15 indexed citations
11.
Watanabe, Youki, Yoshihisa Uenoyama, Naoko Inoue, et al.. (2017). Evaluation of heat stress response in crossbred dairy cows under tropical climate by analysis of heart rate variability. Journal of Veterinary Medical Science. 80(1). 181–185. 22 indexed citations
12.
Minabe, Shiori, Nahoko Ieda, Youki Watanabe, et al.. (2017). Long-Term Neonatal Estrogen Exposure Causes Irreversible Inhibition of LH Pulses by Suppressing Arcuate Kisspeptin Expression via Estrogen Receptors α and β in Female Rodents. Endocrinology. 158(9). 2918–2929. 16 indexed citations
13.
Watanabe, Youki, Kana Ikegami, Nahoko Ieda, et al.. (2017). Enhancement of the luteinising hormone surge by male olfactory signals is associated with anteroventral periventricular Kiss1 cell activation in female rats. Journal of Neuroendocrinology. 29(8). 23 indexed citations
14.
Uenoyama, Yoshihisa, Junko Tomikawa, Naoko Inoue, et al.. (2016). Molecular and Epigenetic Mechanism Regulating Hypothalamic <b><i>Kiss1</i></b> Gene Expression in Mammals. Neuroendocrinology. 103(6). 640–649. 21 indexed citations
16.
Uenoyama, Yoshihisa, Sho Nakamura, Y. Hayakawa, et al.. (2015). Lack of Pulse and Surge Modes and Glutamatergic Stimulation of Luteinising Hormone Release in Kiss1 Knockout Rats. Journal of Neuroendocrinology. 27(3). 187–197. 100 indexed citations
17.
Ieda, Nahoko, Yoshihisa Uenoyama, Yoko Tajima, et al.. (2014). <i>KISS1</i> Gene Expression in the Developing Brain of Female Pigs in Pre- and Peripubertal Periods. Journal of Reproduction and Development. 60(4). 312–316. 21 indexed citations
18.
Uenoyama, Yoshihisa, Shiori Minabe, Youki Watanabe, et al.. (2013). Microarray Analysis of Perinatal-Estrogen-Induced Changes in Gene Expression Related to Brain Sexual Differentiation in Mice. PLoS ONE. 8(11). e79437–e79437. 17 indexed citations
19.
Nakahara, Tatsuo, Yoshihisa Uenoyama, Akira Iwase, et al.. (2013). Chronic Peripheral Administration of Kappa-Opioid Receptor Antagonist Advances Puberty Onset Associated with Acceleration of Pulsatile Luteinizing Hormone Secretion in Female Rats. Journal of Reproduction and Development. 59(5). 479–484. 45 indexed citations
20.
Ishiguro, Akio, Youki Watanabe, & Y. Uchikawa. (2002). Fault diagnosis of plant systems using immune networks. 34–42. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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