Yohei Miyamoto

1.8k total citations
64 papers, 1.4k citations indexed

About

Yohei Miyamoto is a scholar working on Molecular Biology, Nephrology and Oncology. According to data from OpenAlex, Yohei Miyamoto has authored 64 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 16 papers in Nephrology and 13 papers in Oncology. Recurrent topics in Yohei Miyamoto's work include Drug Transport and Resistance Mechanisms (9 papers), Pharmacogenetics and Drug Metabolism (8 papers) and Dialysis and Renal Disease Management (6 papers). Yohei Miyamoto is often cited by papers focused on Drug Transport and Resistance Mechanisms (9 papers), Pharmacogenetics and Drug Metabolism (8 papers) and Dialysis and Renal Disease Management (6 papers). Yohei Miyamoto collaborates with scholars based in Japan, United States and Australia. Yohei Miyamoto's co-authors include Masaki Otagiri, Hiroshi Watanabe, Toru Maruyama, Daisuke Kadowaki, Yu Ishima, Yuko Togashi, Shunsuke Kotani, Keiyu Oshida, Masafumi Fukagawa and Tsuyoshi Noguchi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, FEBS Letters and International Journal of Molecular Sciences.

In The Last Decade

Yohei Miyamoto

62 papers receiving 1.4k citations

Peers

Yohei Miyamoto
Hua Miao China
Yoshio Ogura Japan
Xi Dong China
Rui‐Qing Jiao China
Zsuzsa Varga Hungary
Ellappan Babu United States
Tianqi Wang China
Orhan Canbolat Türkiye
Vivian Soetikno Indonesia
Lining Miao China
Hua Miao China
Yohei Miyamoto
Citations per year, relative to Yohei Miyamoto Yohei Miyamoto (= 1×) peers Hua Miao

Countries citing papers authored by Yohei Miyamoto

Since Specialization
Citations

This map shows the geographic impact of Yohei Miyamoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yohei Miyamoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yohei Miyamoto more than expected).

Fields of papers citing papers by Yohei Miyamoto

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yohei Miyamoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yohei Miyamoto. The network helps show where Yohei Miyamoto may publish in the future.

Co-authorship network of co-authors of Yohei Miyamoto

This figure shows the co-authorship network connecting the top 25 collaborators of Yohei Miyamoto. A scholar is included among the top collaborators of Yohei Miyamoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yohei Miyamoto. Yohei Miyamoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Doi, Hiroyuki, et al.. (2023). A Phase I Study of TRK-250, a Novel siRNA-Based Oligonucleotide, in Patients with Idiopathic Pulmonary Fibrosis. Journal of Aerosol Medicine and Pulmonary Drug Delivery. 36(6). 300–308. 13 indexed citations
2.
Kadowaki, Daisuke, Shigeyuki Miyamura, Kenichiro Kitamura, et al.. (2018). Benzbromarone Attenuates Oxidative Stress in Angiotensin II‐ and Salt‐Induced Hypertensive Model Rats. Oxidative Medicine and Cellular Longevity. 2018(1). 7635274–7635274. 21 indexed citations
3.
Maeda, Akihisa, Mayumi Nakajima, Kei Takahashi, et al.. (2017). Gene expression profiles in auricle skin as a possible additional endpoint for determination of sensitizers: A multi-endpoint evaluation of the local lymph node assay. Toxicology Letters. 280. 133–141. 3 indexed citations
4.
Togashi, Yuko, et al.. (2016). Histopathological image analysis of chemical-induced hepatocellular hypertrophy in mice. Experimental and Toxicologic Pathology. 68(4). 233–239. 17 indexed citations
5.
Oshida, Keiyu, et al.. (2016). Identification of Transporters Involved in Beraprost Sodium Transport In Vitro. European Journal of Drug Metabolism and Pharmacokinetics. 42(1). 117–128. 8 indexed citations
6.
Nakao, Kaoru, et al.. (2015). Nalfurafine hydrochloride, a selective κ opioid receptor agonist, has no reinforcing effect on intravenous self-administration in rhesus monkeys. Journal of Pharmacological Sciences. 130(1). 8–14. 18 indexed citations
7.
Watanabe, Hiroshi, Yohei Miyamoto, Daisuke Honda, et al.. (2013). p -Cresyl sulfate causes renal tubular cell damage by inducing oxidative stress by activation of NADPH oxidase. Kidney International. 83(4). 582–592. 306 indexed citations
8.
Watanabe, Hiroshi, Ryota Tanaka, Hisae Tanaka, et al.. (2013). A human serum albumin–thioredoxin fusion protein prevents experimental contrast-induced nephropathy. Kidney International. 83(3). 446–454. 45 indexed citations
9.
Togashi, Yuko, et al.. (2013). Urinary cystatin C as a renal biomarker and its immunohistochemical localization in anti-GBM glomerulonephritis rats. Experimental and Toxicologic Pathology. 65(7-8). 1137–1143. 9 indexed citations
10.
Miyamoto, Yohei, Yasunori Iwao, Katsumi Mera, et al.. (2012). A uremic toxin, 3-carboxy-4-methyl-5-propyl-2-furanpropionate induces cell damage to proximal tubular cells via the generation of a radical intermediate. Biochemical Pharmacology. 84(9). 1207–1214. 34 indexed citations
11.
Oshida, Keiyu, et al.. (2011). Novel gene markers of immunosuppressive chemicals in mouse lymph node assay. Toxicology Letters. 205(1). 79–85. 5 indexed citations
12.
Togashi, Yuko, et al.. (2011). Urinary cystatin C as a biomarker for acute kidney injury and its immunohistochemical localization in kidney in the CDDP-treated rats. Experimental and Toxicologic Pathology. 64(7-8). 797–805. 39 indexed citations
13.
Miyamoto, Yohei, et al.. (2010). Scratching Behavior of ICR-Derived Glomerulonephritis (ICGN) Mice. Journal of Veterinary Medical Science. 72(9). 1243–1245. 2 indexed citations
14.
15.
Fukazawa, Tominaga, et al.. (2010). Reduction of non-specific adsorption of drugs to plastic containers used in bioassays or analyses. Journal of Pharmacological and Toxicological Methods. 61(3). 329–333. 36 indexed citations
16.
Iwano, Shunsuke, Makiko Ichikawa, Satoko Takizawa, Hisashi Hashimoto, & Yohei Miyamoto. (2009). Identification of AhR-regulated genes involved in PAH-induced immunotoxicity using a highly-sensitive DNA chip, 3D-GeneTM Human Immunity and Metabolic Syndrome 9k. Toxicology in Vitro. 24(1). 85–91. 16 indexed citations
17.
Miyamoto, Yohei, et al.. (2005). Effect of Human Erythropoietin (hEPO) Treatment on Anemia in ICR-derived Glomerulonephritis (ICGN) Mice. EXPERIMENTAL ANIMALS. 54(2). 181–184. 3 indexed citations
18.
Miyamoto, Yohei & Mari Miyamoto. (2004). Immunohistochemical localizations of secretin, cholecystokinin, and somatostatin in the rat small intestine after acute cisplatin treatment. Experimental and Molecular Pathology. 77(3). 238–245. 9 indexed citations
19.
Miyamoto, Yohei, et al.. (2000). COLLABORATIVE WORK TO EVALUATE TOXICITY ON MALE REPRODUCTIVE ORGANS BY REPEATED DOSE STUDIES IN RATS : 2)TESTICULAR TOXICITY IN RATS TREATED ORALLY WITH ETHINYLESTRADIOL FOR 2 WEEKS. The Journal of Toxicological Sciences. 25(SpecialIssue). 33–42. 5 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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