Yingyi Wu

445 total citations
19 papers, 282 citations indexed

About

Yingyi Wu is a scholar working on Molecular Biology, Immunology and Aquatic Science. According to data from OpenAlex, Yingyi Wu has authored 19 papers receiving a total of 282 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 7 papers in Immunology and 2 papers in Aquatic Science. Recurrent topics in Yingyi Wu's work include Extracellular vesicles in disease (3 papers), Immunotherapy and Immune Responses (3 papers) and Epigenetics and DNA Methylation (2 papers). Yingyi Wu is often cited by papers focused on Extracellular vesicles in disease (3 papers), Immunotherapy and Immune Responses (3 papers) and Epigenetics and DNA Methylation (2 papers). Yingyi Wu collaborates with scholars based in China, Finland and Germany. Yingyi Wu's co-authors include Zhanxue Xu, Hsiang‐i Tsai, Hongbo Chen, Dandan Su, Yanping Wu, Sepideh Parvanian, Bo Zhang, Changying Xing, Hui Wang and Haitao Zhu and has published in prestigious journals such as ACS Nano, Free Radical Biology and Medicine and International Journal of Molecular Sciences.

In The Last Decade

Yingyi Wu

18 papers receiving 281 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yingyi Wu China 8 151 64 42 40 32 19 282
Yaxing Li China 9 72 0.5× 28 0.4× 38 0.9× 35 0.9× 34 1.1× 44 257
Shenming Wang China 8 222 1.5× 79 1.2× 57 1.4× 11 0.3× 27 0.8× 16 357
Haiying Dai China 8 95 0.6× 26 0.4× 31 0.7× 16 0.4× 63 2.0× 26 247
Yuhan Yang China 7 148 1.0× 122 1.9× 55 1.3× 25 0.6× 43 1.3× 24 375
Lijun Hao China 11 124 0.8× 20 0.3× 24 0.6× 25 0.6× 19 0.6× 38 382
Haige Zhao China 10 164 1.1× 44 0.7× 22 0.5× 11 0.3× 58 1.8× 35 370
Jimin Han China 10 121 0.8× 23 0.4× 26 0.6× 20 0.5× 72 2.3× 16 291
Siqi Zhou China 9 110 0.7× 32 0.5× 37 0.9× 28 0.7× 14 0.4× 25 257
Rui Geng China 11 108 0.7× 38 0.6× 48 1.1× 8 0.2× 22 0.7× 16 334

Countries citing papers authored by Yingyi Wu

Since Specialization
Citations

This map shows the geographic impact of Yingyi Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yingyi Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yingyi Wu more than expected).

Fields of papers citing papers by Yingyi Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yingyi Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yingyi Wu. The network helps show where Yingyi Wu may publish in the future.

Co-authorship network of co-authors of Yingyi Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Yingyi Wu. A scholar is included among the top collaborators of Yingyi Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yingyi Wu. Yingyi Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Zhang, Yongqiang, et al.. (2025). NETs promote invasive behavior of fibroblast-like synoviocytes through GPIbα in rheumatoid arthritis. Frontiers in Immunology. 16. 1667319–1667319. 1 indexed citations
2.
Zhang, Haiwei, Yingyi Wu, Min Xu, et al.. (2025). Mesenchymal stem cells improve ovarian function by suppressing fibrosis through CTGF/FAK signalling in systemic lupus erythematosus. Lupus Science & Medicine. 12(2). e001468–e001468. 1 indexed citations
3.
Wu, Yingyi, Hsiang‐i Tsai, Huiming Zhu, et al.. (2024). CX-5461 ameliorates disease in lupus-prone mice by triggering B-cell ferroptosis via p53-SLC7A11-ALOX12 pathway. Free Radical Biology and Medicine. 223. 325–340. 6 indexed citations
4.
5.
Xiao, Yang, et al.. (2024). The Transcriptional Landscape of Immune-Response 3′-UTR Alternative Polyadenylation in Melanoma. International Journal of Molecular Sciences. 25(5). 3041–3041. 3 indexed citations
6.
Wu, Yingyi, et al.. (2023). Isorhynchophylline attenuates proliferation and migration of synovial fibroblasts via the FOXC1/β-catenin axis. Autoimmunity. 56(1). 2289868–2289868. 4 indexed citations
7.
Chen, Hang, et al.. (2023). InP/ZnS quantum dots cause liver damage in rare minnow (Gobiocypris rarus) larvae. Comparative Biochemistry and Physiology Part C Toxicology & Pharmacology. 266. 109546–109546. 2 indexed citations
8.
Chen, Hang, Juan Chen, Yingyi Wu, Weiwei Xie, & Li Jin. (2023). A study on the mechanism of Indium phosphide/zinc sulfide core/shell quantum dots influencing embryo incubation of rare minnow (Gobiocypris rarus). Aquatic Toxicology. 261. 106593–106593. 4 indexed citations
9.
Chen, Juan, et al.. (2022). Reproductive toxicity of InP/ZnS QDs in male rare minnow (Gobiocypris rarus). Comparative Biochemistry and Physiology Part C Toxicology & Pharmacology. 259. 109392–109392. 4 indexed citations
10.
Yang, Yang, et al.. (2021). Toxic effects of ZnSe/ZnS quantum dots on the reproduction and genotoxiticy of rare minnow (Gobiocypris rarus). Comparative Biochemistry and Physiology Part C Toxicology & Pharmacology. 247. 109065–109065. 17 indexed citations
11.
Tsai, Hsiang‐i, Yanping Wu, Rui Huang, et al.. (2021). PHF6 functions as a tumor suppressor by recruiting methyltransferase SUV39H1 to nucleolar region and offers a novel therapeutic target for PHF6-muntant leukemia. Acta Pharmaceutica Sinica B. 12(4). 1913–1927. 10 indexed citations
12.
Tsai, Hsiang‐i, Xiaobin Zeng, Longshan Liu, et al.. (2021). NF45/NF90‐mediated rDNA transcription provides a novel target for immunosuppressant development. EMBO Molecular Medicine. 13(3). e12834–e12834. 7 indexed citations
13.
Wu, Yingyi, et al.. (2021). Engineering Cell Membrane-Based Nanovesicles for Melanoma Tumor Treatment. Journal of Biomedical Nanotechnology. 17(5). 838–845. 3 indexed citations
14.
Xu, Zhanxue, Hsiang‐i Tsai, Yingyi Wu, et al.. (2020). Engineering Programmed Death Ligand-1/Cytotoxic T-Lymphocyte-Associated Antigen-4 Dual-Targeting Nanovesicles for Immunosuppressive Therapy in Transplantation. ACS Nano. 14(7). 7959–7969. 45 indexed citations
15.
Yang, Min, Zhanxue Xu, Hsiang‐i Tsai, et al.. (2020). PD-L1 cellular nanovesicles carrying rapamycin inhibit alloimmune responses in transplantation. Biomaterials Science. 9(4). 1246–1255. 16 indexed citations
16.
Huang, Yang, et al.. (2020). Bufotalin ameliorates experimental Sjögren’s syndrome development by inhibiting Th17 generation. Naunyn-Schmiedeberg s Archives of Pharmacology. 393(10). 1977–1985. 8 indexed citations
17.
Basu, Jayasri, et al.. (2020). Regional Variations in Placental Protein Expression of Matrix Metalloproteinase-9 in Term Uncomplicated Pregnancy. 9(9). 26–33.
18.
Su, Dandan, Hsiang‐i Tsai, Zhanxue Xu, et al.. (2019). Exosomal PD‐L1 functions as an immunosuppressant to promote wound healing. Journal of Extracellular Vesicles. 9(1). 1709262–1709262. 100 indexed citations
19.
Yuan, Yanggang, Hui Wang, Yingyi Wu, et al.. (2015). P53 Contributes to Cisplatin Induced Renal Oxidative Damage via Regulating P66shc and MnSOD. Cellular Physiology and Biochemistry. 37(4). 1240–1256. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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