Yannick P. Kok

585 total citations
12 papers, 263 citations indexed

About

Yannick P. Kok is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Yannick P. Kok has authored 12 papers receiving a total of 263 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 7 papers in Oncology and 3 papers in Cell Biology. Recurrent topics in Yannick P. Kok's work include DNA Repair Mechanisms (8 papers), Cancer-related Molecular Pathways (4 papers) and CRISPR and Genetic Engineering (3 papers). Yannick P. Kok is often cited by papers focused on DNA Repair Mechanisms (8 papers), Cancer-related Molecular Pathways (4 papers) and CRISPR and Genetic Engineering (3 papers). Yannick P. Kok collaborates with scholars based in Netherlands, United Kingdom and United States. Yannick P. Kok's co-authors include Marcel A.T.M. van Vugt, Nathalie van den Tempel, Pepijn M. Schoonen, Diana C.J. Spierings, Floris Foijer, Rudolf S.N. Fehrmann, Marieke Everts, Anne Margriet Heijink, Björn Bakker and Sergi Guerrero Llobet and has published in prestigious journals such as Nucleic Acids Research, Nature Communications and Oncogene.

In The Last Decade

Yannick P. Kok

11 papers receiving 260 citations

Peers

Yannick P. Kok
Michalis Petropoulos Greece
Huy Nguyen United States
Rozario Thomas United States
Amy E. Schade United States
Angela Incassati United States
Meng‐Lay Lin United Kingdom
Mihaela Robu Canada
Larissa A. Sambel United States
Stephanie Hampp Germany
Connor E. Dunn United States
Michalis Petropoulos Greece
Yannick P. Kok
Citations per year, relative to Yannick P. Kok Yannick P. Kok (= 1×) peers Michalis Petropoulos

Countries citing papers authored by Yannick P. Kok

Since Specialization
Citations

This map shows the geographic impact of Yannick P. Kok's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yannick P. Kok with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yannick P. Kok more than expected).

Fields of papers citing papers by Yannick P. Kok

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yannick P. Kok. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yannick P. Kok. The network helps show where Yannick P. Kok may publish in the future.

Co-authorship network of co-authors of Yannick P. Kok

This figure shows the co-authorship network connecting the top 25 collaborators of Yannick P. Kok. A scholar is included among the top collaborators of Yannick P. Kok based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yannick P. Kok. Yannick P. Kok is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Benedetti, Stefano De, et al.. (2025). Double Filtration Plasmapheresis for Environmental Toxin Removal: A Case Series of Patients With Hyperlipoproteinemia(a). Journal of Clinical Apheresis. 40(5). e70060–e70060.
2.
Jarzebska, Natalia, Roman N. Rodionov, Richard Straube, et al.. (2025). Neutrophil Extracellular Traps (NETs) as a Potential Target for Anti-Aging: Role of Therapeutic Apheresis. Hormone and Metabolic Research. 57(11). 632–638. 2 indexed citations
3.
Steenblock, Charlotte, Nicole Toepfner, Yannick P. Kok, et al.. (2024). A multimodal approach for treating post-acute infectious syndrome. 1(1). 35–41. 2 indexed citations
4.
Kok, Yannick P., Shibo Yu, Bert van de Kooij, et al.. (2024). RAD52-dependent mitotic DNA synthesis is required for genome stability in Cyclin E1-overexpressing cells. Cell Reports. 43(4). 114116–114116. 5 indexed citations
5.
Talens, Francien, Yannick P. Kok, Mengting Chen, et al.. (2024). RAD51 recruitment but not replication fork stability associates with PARP inhibitor response in ovarian cancer patient-derived xenograft models. NAR Cancer. 6(4). zcae044–zcae044. 1 indexed citations
6.
Kok, Yannick P., Shibo Yu, Jana Dobrovolná, et al.. (2023). PPM1D activity promotes the replication stress caused by cyclin E1 overexpression. Molecular Oncology. 18(1). 6–20. 2 indexed citations
7.
Heijink, Anne Margriet, David Porubský, Jurrian K. de Kanter, et al.. (2022). Sister chromatid exchanges induced by perturbed replication can form independently of BRCA1, BRCA2 and RAD51. Nature Communications. 13(1). 6722–6722. 27 indexed citations
8.
Kok, Yannick P., et al.. (2021). Shaping the BRCAness mutational landscape by alternative double-strand break repair, replication stress and mitotic aberrancies. Nucleic Acids Research. 49(8). 4239–4257. 41 indexed citations
9.
Kok, Yannick P., Sergi Guerrero Llobet, Pepijn M. Schoonen, et al.. (2020). Overexpression of Cyclin E1 or Cdc25A leads to replication stress, mitotic aberrancies, and increased sensitivity to replication checkpoint inhibitors. Oncogenesis. 9(10). 88–88. 51 indexed citations
10.
Heijink, Anne Margriet, Marieke Everts, Yannick P. Kok, et al.. (2019). Modeling of Cisplatin-Induced Signaling Dynamics in Triple-Negative Breast Cancer Cells Reveals Mediators of Sensitivity. Cell Reports. 28(9). 2345–2357.e5. 24 indexed citations
11.
Schoonen, Pepijn M., Yannick P. Kok, Björn Bakker, et al.. (2019). Premature mitotic entry induced by ATR inhibition potentiates olaparib inhibition‐mediated genomic instability, inflammatory signaling, and cytotoxicity in BRCA2‐deficient cancer cells. Molecular Oncology. 13(11). 2422–2440. 66 indexed citations
12.
Tempel, Nathalie van den, Yannick P. Kok, Anne Margriet Heijink, et al.. (2018). TPX2/Aurora kinase A signaling as a potential therapeutic target in genomically unstable cancer cells. Oncogene. 38(6). 852–867. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026