Xu Lin

862 total citations
31 papers, 548 citations indexed

About

Xu Lin is a scholar working on Molecular Biology, Oncology and Epidemiology. According to data from OpenAlex, Xu Lin has authored 31 papers receiving a total of 548 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 6 papers in Oncology and 5 papers in Epidemiology. Recurrent topics in Xu Lin's work include Cell death mechanisms and regulation (4 papers), Venomous Animal Envenomation and Studies (4 papers) and Cancer-related gene regulation (4 papers). Xu Lin is often cited by papers focused on Cell death mechanisms and regulation (4 papers), Venomous Animal Envenomation and Studies (4 papers) and Cancer-related gene regulation (4 papers). Xu Lin collaborates with scholars based in China, United States and United Kingdom. Xu Lin's co-authors include Xinjian Lin, Wannan Chen, Wei Liu, Jianyin Lin, Qun Xie, Jie Chen, Longpo Zheng, Xiaoqian Wang, Xianting Ding and Jian Yang and has published in prestigious journals such as Nature Communications, FEBS Letters and American Journal Of Pathology.

In The Last Decade

Xu Lin

30 papers receiving 545 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Xu Lin China 17 315 127 97 69 61 31 548
Junfeng Shi China 16 391 1.2× 168 1.3× 51 0.5× 151 2.2× 44 0.7× 54 645
Chao He China 13 280 0.9× 170 1.3× 101 1.0× 148 2.1× 27 0.4× 42 578
Yani Zhao China 10 226 0.7× 63 0.5× 201 2.1× 75 1.1× 48 0.8× 21 604
Weiqun Wang China 16 222 0.7× 106 0.8× 135 1.4× 137 2.0× 24 0.4× 37 596
Dario Priem Belgium 10 512 1.6× 194 1.5× 104 1.1× 95 1.4× 28 0.5× 15 712
Mei Yieng Chin Canada 14 367 1.2× 152 1.2× 42 0.4× 64 0.9× 27 0.4× 19 516
Nupur Mukherjee India 17 415 1.3× 154 1.2× 89 0.9× 202 2.9× 46 0.8× 33 689
Travis Lear United States 17 476 1.5× 64 0.5× 103 1.1× 81 1.2× 34 0.6× 29 722
Martin Majer United States 11 292 0.9× 45 0.4× 41 0.4× 64 0.9× 47 0.8× 20 666

Countries citing papers authored by Xu Lin

Since Specialization
Citations

This map shows the geographic impact of Xu Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xu Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xu Lin more than expected).

Fields of papers citing papers by Xu Lin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xu Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xu Lin. The network helps show where Xu Lin may publish in the future.

Co-authorship network of co-authors of Xu Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Xu Lin. A scholar is included among the top collaborators of Xu Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xu Lin. Xu Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Deng, Dehui, et al.. (2025). HIIT versus MICT in MASLD: mechanisms mediated by gut-liver axis crosstalk, mitochondrial dynamics remodeling, and adipokine signaling attenuation. Lipids in Health and Disease. 24(1). 144–144. 3 indexed citations
2.
Chen, Yuzhong, Xuming Song, Yingkuan Liang, et al.. (2024). Ferritinophagy mediates adaptive resistance to EGFR tyrosine kinase inhibitors in non-small cell lung cancer. Nature Communications. 15(1). 4195–4195. 12 indexed citations
3.
Xu, Xiaoxin, et al.. (2024). m6A modification of VEGFA mRNA by RBM15/YTHDF2/IGF2BP3 contributes to angiogenesis of hepatocellular carcinoma. Molecular Carcinogenesis. 63(11). 2174–2189. 11 indexed citations
4.
Wang, Xiang, Xinxin Ren, Xu Lin, et al.. (2024). Recent progress of ferroptosis in cancers and drug discovery. Asian Journal of Pharmaceutical Sciences. 19(4). 100939–100939. 16 indexed citations
5.
Lin, Hanbin, et al.. (2024). OTUD1 enhances gastric cancer aggressiveness by deubiquitinating EBV-encoded protein BALF1 to stabilize the apoptosis inhibitor Bcl-2. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1870(5). 167132–167132. 4 indexed citations
6.
Jin, Gui‐Lin, Ying Xu, Bing Chen, et al.. (2021). Identification of Koumine as a Translocator Protein 18 kDa Positive Allosteric Modulator for the Treatment of Inflammatory and Neuropathic Pain. Frontiers in Pharmacology. 12. 692917–692917. 13 indexed citations
7.
Wang, Deping, et al.. (2020). Apolipoprotein L1 is transcriptionally regulated by SP1, IRF1 and IRF2 in hepatoma cells. FEBS Letters. 594(19). 3108–3121. 6 indexed citations
8.
Dai, Xiaoman, Xiaohan Lin, Xiaoxing Huang, et al.. (2020). SIK2 represses AKT/GSK3β/β‐catenin signaling and suppresses gastric cancer by inhibiting autophagic degradation of protein phosphatases. Molecular Oncology. 15(1). 228–245. 28 indexed citations
10.
Wu, Yunli, et al.. (2019). Microcystin-LR promotes necroptosis in primary mouse hepatocytes by overproducing reactive oxygen species. Toxicology and Applied Pharmacology. 377. 114626–114626. 20 indexed citations
11.
Liu, Wei, et al.. (2019). AKT activator SC79 protects hepatocytes from TNF-α-mediated apoptosis and alleviates d-Gal/LPS-induced liver injury. American Journal of Physiology-Gastrointestinal and Liver Physiology. 316(3). G387–G396. 74 indexed citations
12.
Liu, Wei, et al.. (2019). PI3K/AKT inhibitors aggravate death receptor-mediated hepatocyte apoptosis and liver injury. Toxicology and Applied Pharmacology. 381. 114729–114729. 30 indexed citations
13.
Liu, Wei, et al.. (2018). A Novel AKT Activator, SC79, Prevents Acute Hepatic Failure Induced by Fas-Mediated Apoptosis of Hepatocytes. American Journal Of Pathology. 188(5). 1171–1182. 27 indexed citations
14.
Ye, Jianxin, Jinsheng Huang, Jie Xu, et al.. (2017). ERp29 controls invasion and metastasis of gastric carcinoma by inhibition of epithelial-mesenchymal transition via PI3K/Aktsignaling pathway. BMC Cancer. 17(1). 626–626. 21 indexed citations
15.
Ding, Xianting, et al.. (2015). Optimized combinations of bortezomib, camptothecin, and doxorubicin show increased efficacy and reduced toxicity in treating oral cancer. Anti-Cancer Drugs. 26(5). 547–554. 29 indexed citations
16.
Qu, Jing, Min Song, Jian Xie, et al.. (2015). Notch2 signaling contributes to cell growth, invasion, and migration in salivary adenoid cystic carcinoma. Molecular and Cellular Biochemistry. 411(1-2). 135–141. 18 indexed citations
17.
Xie, Qun, et al.. (2013). Recombinant adenovirus snake venom cystatin inhibits the growth, invasion, and metastasis of B16F10 cells in vitro and in vivo. Melanoma Research. 23(6). 444–451. 11 indexed citations
19.
Tang, Nanhong, Qun Xie, Xiaoqian Wang, et al.. (2011). Inhibition of invasion and metastasis of MHCC97H cells by expression of snake venom cystatin through reduction of proteinases activity and Epithelial-Mesenchymal Transition. Archives of Pharmacal Research. 34(5). 781–789. 26 indexed citations
20.
She, Feifei, Xu Lin, & Hao Chen. (2009). Helicobacter pylori oipA gene switch status and its association with gastic diseases. Zhonghua xiaohua zazhi. 29(2). 122–125. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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