WY Almawi

619 total citations
12 papers, 473 citations indexed

About

WY Almawi is a scholar working on Immunology, Oncology and Genetics. According to data from OpenAlex, WY Almawi has authored 12 papers receiving a total of 473 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 3 papers in Oncology and 3 papers in Genetics. Recurrent topics in WY Almawi's work include Estrogen and related hormone effects (3 papers), T-cell and B-cell Immunology (3 papers) and Immune Response and Inflammation (2 papers). WY Almawi is often cited by papers focused on Estrogen and related hormone effects (3 papers), T-cell and B-cell Immunology (3 papers) and Immune Response and Inflammation (2 papers). WY Almawi collaborates with scholars based in Tunisia, Bahrain and Lebanon. WY Almawi's co-authors include Mark L. Lipman, T B Strom, A. Christopher Stevens, B Zanker, Ikram Sghaier, Étienne Brochot, Sabrina Zidi, Christian Barth, Abdelhafidh Hajjej and Houda Kâabi and has published in prestigious journals such as The Journal of Immunology, BJOG An International Journal of Obstetrics & Gynaecology and American Journal of Infection Control.

In The Last Decade

WY Almawi

12 papers receiving 455 citations

Peers

WY Almawi

IMMUN

EPIDE

EDM

Vladislava Paharkova-Vatchkova United States

Z Amraoui Belgium

Agnieszka Paradowska‐Dogan Germany

Kenneth Lau United States

Troy Auffenberg United States

Dmitry J. Liepinsh United States

Letizia Galleri Italy

L.J. Abraham Australia

Fabrizia Ferracin Switzerland

Vladislava Paharkova-Vatchkova United States

WY Almawi

400 ×1.8

IMMUN

122 ×1.3

85 ×1.4

EPIDE

51 ×0.9

61 ×1.2

EDM

Citations per year, relative to WY Almawi WY Almawi (= 1×) peers Vladislava Paharkova-Vatchkova

Countries citing papers authored by WY Almawi

Since Specialization

Citations

This map shows the geographic impact of WY Almawi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by WY Almawi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites WY Almawi more than expected).

Fields of papers citing papers by WY Almawi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by WY Almawi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by WY Almawi. The network helps show where WY Almawi may publish in the future.

Co-authorship network of co-authors of WY Almawi

This figure shows the co-authorship network connecting the top 25 collaborators of WY Almawi. A scholar is included among the top collaborators of WY Almawi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with WY Almawi. WY Almawi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

Almawi, WY, et al.. (2018). FAS A‐670G and Fas ligand IVS2nt A 124G polymorphisms are significantly increased in women with pre‐eclampsia and may contribute to HELLP syndrome: a case‐controlled study. BJOG An International Journal of Obstetrics & Gynaecology. 125(13). 1758–1764. 11 indexed citations

Sghaier, Ikram, et al.. (2018). TLR3 and TLR4 SNP variants in the liver disease resulting from hepatitis B virus and hepatitis C virus infection. British Journal of Biomedical Science. 76(1). 35–41. 35 indexed citations

Almawi, WY, et al.. (2017). Genetic variation in the progesterone receptor gene and susceptibility to recurrent pregnancy loss: a case–control study. BJOG An International Journal of Obstetrics & Gynaecology. 125(6). 729–735. 20 indexed citations

Almawi, WY, et al.. (2016). HLA DRB1/DQB1 alleles and DRB1‐DQB1 haplotypes and the risk of rheumatoid arthritis in Tunisians: a population‐based case–control study. HLA. 88(3). 100–109. 12 indexed citations

Hajjej, Abdelhafidh, et al.. (2011). HLA class I and class II polymorphism in a population from south-eastern Tunisia (Gabes Area). International Journal of Immunogenetics. 38(3). 191–199. 13 indexed citations

Hajjej, Abdelhafidh, Houda Kâabi, Amel Ben Ammar Elgaaïed, et al.. (2010). HLA class I and class II polymorphisms in Tunisian Berbers. Annals of Human Biology. 38(2). 156–164. 14 indexed citations

Fawaz, Naglaa, Hani Tamim, & WY Almawi. (2005). Low prevalence of antibodies to human T-lymphotropic virus-I/II among blood donors in Eastern Saudi Arabia. American Journal of Infection Control. 33(3). 189–191. 10 indexed citations

Almawi, WY, et al.. (2004). Distribution of HLA class II (DRB1/DQB1) alleles and haplotypes among Bahraini and Lebanese Arabs. Transplantation Proceedings. 36(6). 1844–1846. 4 indexed citations

Almawi, WY, et al.. (2002). Negative regulation of nuclear factor-kappaB activation and function by glucocorticoids. Journal of Molecular Endocrinology. 28(2). 69–78. 193 indexed citations

10.

Almawi, WY, et al.. (1997). Association of Altered T Cell Immunity with IDDM. More than a cause and effect. 5. 126–141. 1 indexed citations

11.

Almawi, WY, et al.. (1996). Partial mediation of glucocorticoid antiproliferative effects by lipocortins. The Journal of Immunology. 157(12). 5231–5239. 26 indexed citations

12.

Almawi, WY, et al.. (1991). Abrogation of glucocorticoid-mediated inhibition of T cell proliferation by the synergistic action of IL-1, IL-6, and IFN-gamma. The Journal of Immunology. 146(10). 3523–3527. 134 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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