William T. Stott

1.8k total citations
61 papers, 1.3k citations indexed

About

William T. Stott is a scholar working on Molecular Biology, Cancer Research and Plant Science. According to data from OpenAlex, William T. Stott has authored 61 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 20 papers in Cancer Research and 11 papers in Plant Science. Recurrent topics in William T. Stott's work include Carcinogens and Genotoxicity Assessment (20 papers), DNA Repair Mechanisms (6 papers) and Pharmacogenetics and Drug Metabolism (6 papers). William T. Stott is often cited by papers focused on Carcinogens and Genotoxicity Assessment (20 papers), DNA Repair Mechanisms (6 papers) and Pharmacogenetics and Drug Metabolism (6 papers). William T. Stott collaborates with scholars based in United States, India and Belgium. William T. Stott's co-authors include P.G. Watanabe, Lloyd B. Bullerman, J.F. Quast, B. Bhaskar Gollapudi, Hon‐Wing Leung, A.M. Schumann, James B. Knaak, Patricia S. Mason, Ted Fox and F. Peter Guengerich and has published in prestigious journals such as Journal of Agricultural and Food Chemistry, Annals of the New York Academy of Sciences and The Journal of Pediatrics.

In The Last Decade

William T. Stott

58 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
William T. Stott United States 21 438 351 340 257 144 61 1.3k
Luigi Robbiano Italy 22 598 1.4× 390 1.1× 326 1.0× 193 0.8× 77 0.5× 70 1.3k
G. Bronzetti Italy 19 470 1.1× 487 1.4× 331 1.0× 286 1.1× 137 1.0× 101 1.4k
Matthew S. Bogdanffy United States 26 742 1.7× 492 1.4× 560 1.6× 186 0.7× 124 0.9× 71 1.9k
Sandro Grilli Italy 20 601 1.4× 399 1.1× 326 1.0× 202 0.8× 99 0.7× 124 1.2k
Trevor Green United Kingdom 21 350 0.8× 478 1.4× 224 0.7× 129 0.5× 172 1.2× 31 1.1k
Larry S. Andrews United States 20 670 1.5× 280 0.8× 446 1.3× 125 0.5× 150 1.0× 40 1.2k
George Cruzan United States 19 459 1.0× 180 0.5× 347 1.0× 120 0.5× 128 0.9× 32 1000
K. Norpoth Germany 23 879 2.0× 406 1.2× 636 1.9× 150 0.6× 94 0.7× 136 1.7k
Maurizio Taningher Italy 17 539 1.2× 473 1.3× 242 0.7× 163 0.6× 44 0.3× 59 1.0k
V. J. Feil United States 20 158 0.4× 218 0.6× 290 0.9× 105 0.4× 195 1.4× 87 1.1k

Countries citing papers authored by William T. Stott

Since Specialization
Citations

This map shows the geographic impact of William T. Stott's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William T. Stott with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William T. Stott more than expected).

Fields of papers citing papers by William T. Stott

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William T. Stott. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William T. Stott. The network helps show where William T. Stott may publish in the future.

Co-authorship network of co-authors of William T. Stott

This figure shows the co-authorship network connecting the top 25 collaborators of William T. Stott. A scholar is included among the top collaborators of William T. Stott based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William T. Stott. William T. Stott is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Johnson, Keith A., et al.. (2007). Repeated dose toxicity and developmental toxicity of diisopropanolamine to rats. Food and Chemical Toxicology. 45(10). 1838–1845. 5 indexed citations
2.
Stott, William T.. (2005). Philip G. Watanabe. Toxicological Sciences. 84(2). 211–211. 1 indexed citations
3.
Leung, Hon‐Wing, Lisa M. Kamendulis, & William T. Stott. (2005). Review of the carcinogenic activity of diethanolamine and evidence of choline deficiency as a plausible mode of action. Regulatory Toxicology and Pharmacology. 43(3). 260–271. 21 indexed citations
4.
Saghir, Shakil A., et al.. (2005). Investigation of the formation of N-nitrosodiethanolamine in B6C3F1 mice following topical administration of triethanolamine. Regulatory Toxicology and Pharmacology. 43(1). 10–18. 2 indexed citations
5.
Stott, William T.. (2004). Evaluation of the Potential of Triethanolamine to Alter Hepatic Choline Levels in Female B6C3F1 Mice. Toxicological Sciences. 79(2). 242–247. 11 indexed citations
6.
Stott, William T., et al.. (2004). Effect of strain and diet upon constitutive and chemically induced activities of several xenobiotic-metabolizing enzymes in rats. Regulatory Toxicology and Pharmacology. 39(3). 325–333. 13 indexed citations
7.
Seidel, Shawn D., et al.. (2003). Identification of transcriptome profiles for the DNA‐damaging agents bleomycin and hydrogen peroxide in L5178Y mouse lymphoma cells. Environmental and Molecular Mutagenesis. 42(1). 19–25. 20 indexed citations
8.
Stott, William T., B. Bhaskar Gollapudi, & K. S. Rao. (2001). Mammalian Toxicity of 1,3-Dichloropropene. Reviews of Environmental Contamination and Toxicology. 168. 1–42. 15 indexed citations
9.
Stebbins, Kenneth E., et al.. (2000). Chronic Toxicity and Oncogenicity Studies of Ingested 1,3-Dichloropropene in Rats and Mice. Regulatory Toxicology and Pharmacology. 32(1). 1–13. 9 indexed citations
10.
Stott, William T., John M. Waechter, David L. Rick, & Alan L. Mendrala. (2000). Absorption, distribution, metabolism and excretion of intravenously and dermally administered triethanolamine in mice. Food and Chemical Toxicology. 38(11). 1043–1051. 13 indexed citations
11.
Stott, William T., et al.. (2000). Potential mechanisms of tumorigenic action of diethanolamine in mice. Toxicology Letters. 114(1-3). 67–75. 25 indexed citations
12.
Stott, William T.. (1998). Bioavailability and Pharmacokinetics of Microencapsulated 1,3-Dichloropropene in Rats. Toxicological Sciences. 41(1). 21–28. 6 indexed citations
13.
Gollapudi, B. Bhaskar, et al.. (1998). Mode of action considerations in the use of transgenic animals for mutagenicity and carcinogenicity evaluations. Toxicology Letters. 102-103. 479–484. 10 indexed citations
14.
Stott, William T., et al.. (1997). Evaluation of a novel assay of potential toxicity/neurotoxicity of carpet emissions (VOCs) in mice. Food and Chemical Toxicology. 35(2). 241–254. 2 indexed citations
15.
Knaak, James B., et al.. (1997). Toxicology of Mono-, Di-, and Triethanolamine. Reviews of Environmental Contamination and Toxicology. 149. 1–86. 84 indexed citations
16.
Stott, William T.. (1996). Regulatory Implications of Peroxisome Proliferation: An Industrial Perspective. Annals of the New York Academy of Sciences. 804(1). 641–648. 1 indexed citations
17.
Klein, Julia, Dale M. Stack, Apostolos Papageorgiou, et al.. (1994). Maternal cocaine use without evidence of fetal exposure. The Journal of Pediatrics. 125(4). 652–654. 20 indexed citations
18.
Stott, William T., et al.. (1990). Chronic toxicity and oncogenicity of picloram in Fischer 344 rats. Journal of Toxicology and Environmental Health. 30(2). 91–104. 2 indexed citations
19.
Stott, William T.. (1988). Subchronic toxicity of inhaled technical grade 1,3-dichloropropene in rats and mice. Fundamental and Applied Toxicology. 11(2). 207–220. 24 indexed citations
20.
Stott, William T.. (1988). Chemically induced proliferation of peroxisomes: Implications for risk assessment. Regulatory Toxicology and Pharmacology. 8(2). 125–159. 85 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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