William F. Schwindinger

2.8k total citations
38 papers, 2.2k citations indexed

About

William F. Schwindinger is a scholar working on Molecular Biology, Genetics and Rheumatology. According to data from OpenAlex, William F. Schwindinger has authored 38 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 8 papers in Genetics and 5 papers in Rheumatology. Recurrent topics in William F. Schwindinger's work include Receptor Mechanisms and Signaling (8 papers), Protein Kinase Regulation and GTPase Signaling (7 papers) and Genetic Syndromes and Imprinting (5 papers). William F. Schwindinger is often cited by papers focused on Receptor Mechanisms and Signaling (8 papers), Protein Kinase Regulation and GTPase Signaling (7 papers) and Genetic Syndromes and Imprinting (5 papers). William F. Schwindinger collaborates with scholars based in United States, Russia and France. William F. Schwindinger's co-authors include Michael A. Levine, Janet D. Robishaw, Clair A. Francomano, Jonathan R. Warner, Matthew D. Ringel, Howard M. Fried, Gary S. Wand, Harvey J. Schugar, Larry S. Zweifel and Maria Jasin and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Nucleic Acids Research.

In The Last Decade

William F. Schwindinger

38 papers receiving 2.1k citations

Peers

William F. Schwindinger
Pieter H. Anborgh Canada
Kunio Kitamura Japan
Marc Glucksman United States
Taiji Kato Japan
Stavit A. Shalev Israel
D. D. Wood Canada
Purva Bali United States
Yaron R. Hadari United States
Lynne A. Jones United States
Adeline Vanderver United States
Pieter H. Anborgh Canada
William F. Schwindinger
Citations per year, relative to William F. Schwindinger William F. Schwindinger (= 1×) peers Pieter H. Anborgh

Countries citing papers authored by William F. Schwindinger

Since Specialization
Citations

This map shows the geographic impact of William F. Schwindinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William F. Schwindinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William F. Schwindinger more than expected).

Fields of papers citing papers by William F. Schwindinger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William F. Schwindinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William F. Schwindinger. The network helps show where William F. Schwindinger may publish in the future.

Co-authorship network of co-authors of William F. Schwindinger

This figure shows the co-authorship network connecting the top 25 collaborators of William F. Schwindinger. A scholar is included among the top collaborators of William F. Schwindinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William F. Schwindinger. William F. Schwindinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schwindinger, William F., et al.. (2018). Prokineticin-2 and ghrelin robustly influence the sexual and ingestive behaviors of female Syrian hamsters. Hormones and Behavior. 106. 135–143. 3 indexed citations
2.
Moon, Anne, et al.. (2014). Disruption of G-Protein γ5 Subtype Causes Embryonic Lethality in Mice. PLoS ONE. 9(3). e90970–e90970. 14 indexed citations
3.
Schwindinger, William F., Uyenlinh L. Mirshahi, Kathleen Sheridan, et al.. (2011). Synergistic Roles for G-protein γ3 and γ7 Subtypes in Seizure Susceptibility as Revealed in Double Knock-out Mice. Journal of Biological Chemistry. 287(10). 7121–7133. 40 indexed citations
4.
Huso, David L., Sarah Edie, Michael A. Levine, et al.. (2011). Heterotopic Ossifications in a Mouse Model of Albright Hereditary Osteodystrophy. PLoS ONE. 6(6). e21755–e21755. 28 indexed citations
5.
Schwindinger, William F., et al.. (2010). Adenosine A2A Receptor Signaling and Golf Assembly Show a Specific Requirement for the γ7 Subtype in the Striatum. Journal of Biological Chemistry. 285(39). 29787–29796. 47 indexed citations
6.
Masker, Kathryn, Alicia Golden, Christian J. Gaffney, et al.. (2007). Transcriptional profile of Rous Sarcoma Virus transformed chicken embryo fibroblasts reveals new signaling targets of viral-src. Virology. 364(1). 10–20. 13 indexed citations
7.
Germain‐Lee, Emily L., William F. Schwindinger, Janet L. Crane, et al.. (2005). A Mouse Model of Albright Hereditary Osteodystrophy Generated by Targeted Disruption of Exon 1 of the Gnas Gene. Endocrinology. 146(11). 4697–4709. 100 indexed citations
8.
Schwindinger, William F., et al.. (2003). Loss of G Protein γ7 Alters Behavior and Reduces Striatal αolf Level and cAMP Production. Journal of Biological Chemistry. 278(8). 6575–6579. 96 indexed citations
9.
Robishaw, Janet D., Qin Wang, & William F. Schwindinger. (2002). Ribozyme-Mediated Suppression of G Protein γ Subunits. Methods in enzymology on CD-ROM/Methods in enzymology. 344. 435–451. 3 indexed citations
10.
Schwindinger, William F., et al.. (2001). Selective Resistance to Parathyroid Hormone Caused by a Novel Uncoupling Mutation in the Carboxyl Terminus of Gαs. Journal of Biological Chemistry. 276(1). 165–171. 38 indexed citations
11.
Schwindinger, William F. & Janet D. Robishaw. (2001). Heterotrimeric G-protein βγ-dimers in growth and differentiation. Oncogene. 20(13). 1653–1660. 167 indexed citations
12.
Wand, Gary S., Michael A. Levine, Larry S. Zweifel, William F. Schwindinger, & Ted Abel. (2001). The cAMP–Protein Kinase A Signal Transduction Pathway Modulates Ethanol Consumption and Sedative Effects of Ethanol. Journal of Neuroscience. 21(14). 5297–5303. 129 indexed citations
13.
Ringel, Matthew D., William F. Schwindinger, Motoyasu Saji, Martha A. Zeiger, & Michael A. Levine. (1998). Determination of Transgene Copy Number and Expression Level Using Denaturing Gradient Gel Electrophoresis. BioTechniques. 24(1). 126–131. 3 indexed citations
14.
Schwindinger, William F., Kimberly J. Reese, Ann M. Lawler, John D. Gearhart, & Michael A. Levine. (1997). Targeted Disruption of Gnas in Embryonic Stem Cells*. Endocrinology. 138(10). 4058–4063. 20 indexed citations
15.
Ringel, Matthew D., William F. Schwindinger, & Michael A. Levine. (1996). Clinical Implications of Genetic Defects in G Proteins: The Molecular Basis of McCune-Albright Syndrome and Albright Hereditary Osteodystrophy. Medicine. 75(4). 171–184. 113 indexed citations
16.
Schwindinger, William F., et al.. (1995). Characterization of Albright hereditary osteodystrophy and related disorders.. PubMed. 36(1). 3–13. 3 indexed citations
17.
Schwindinger, William F., et al.. (1994). Signal-Transducing G Proteins: Basic and Clinical Implications. Progress in nucleic acid research and molecular biology. 47. 81–111. 8 indexed citations
18.
Schwindinger, William F. & Michael A. Levine. (1994). Albright Hereditary Osteodystrophy. The Endocrinologist. 4(1). 17–27. 11 indexed citations
19.
Fried, Howard M., et al.. (1983). Cycloheximide resistance in yeast: the gene and its portein. Nucleic Acids Research. 11(10). 3123–3135. 164 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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