Wenman Wu

907 total citations
69 papers, 539 citations indexed

About

Wenman Wu is a scholar working on Hematology, Molecular Biology and Genetics. According to data from OpenAlex, Wenman Wu has authored 69 papers receiving a total of 539 indexed citations (citations by other indexed papers that have themselves been cited), including 48 papers in Hematology, 18 papers in Molecular Biology and 15 papers in Genetics. Recurrent topics in Wenman Wu's work include Blood Coagulation and Thrombosis Mechanisms (28 papers), Hemophilia Treatment and Research (26 papers) and Platelet Disorders and Treatments (17 papers). Wenman Wu is often cited by papers focused on Blood Coagulation and Thrombosis Mechanisms (28 papers), Hemophilia Treatment and Research (26 papers) and Platelet Disorders and Treatments (17 papers). Wenman Wu collaborates with scholars based in China, United States and Hong Kong. Wenman Wu's co-authors include Qiulan Ding, Hongli Wang, Peter N. Walsh, Dmitri V. Kravtsov, David Gailani, Morey A. Blinder, Thao P. Dang, Xuefeng Wang, Mao-fu Sun and Joost C.M. Meijers and has published in prestigious journals such as Journal of Biological Chemistry, Blood and PLoS ONE.

In The Last Decade

Wenman Wu

67 papers receiving 533 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Wenman Wu China	12	307	169	155	89	56	69	539
Ranjeet Sinha United States	11	267 0.9×	111 0.7×	132 0.9×	30 0.3×	28 0.5×	25	481
Sandra Schiffman United States	13	457 1.5×	267 1.6×	82 0.5×	86 1.0×	127 2.3×	21	656
John E. Lund United States	9	168 0.5×	43 0.3×	83 0.5×	84 0.9×	63 1.1×	14	430
Julia Driesen Germany	11	189 0.6×	44 0.3×	103 0.7×	46 0.5×	12 0.2×	16	578
AZ Budzynski United States	10	213 0.7×	34 0.2×	63 0.4×	79 0.9×	169 3.0×	21	442
Rénaté Bonier France	4	85 0.3×	18 0.1×	102 0.7×	24 0.3×	45 0.8×	5	320
Ellie Karampini Netherlands	9	128 0.4×	31 0.2×	71 0.5×	22 0.2×	31 0.6×	12	306
Kazuta Yasui Japan	13	207 0.7×	66 0.4×	120 0.8×	98 1.1×	15 0.3×	43	476
Y. Terry Lee United States	16	467 1.5×	599 3.5×	475 3.1×	50 0.6×	25 0.4×	37	1.0k
H Jouault France	14	324 1.1×	205 1.2×	234 1.5×	31 0.3×	43 0.8×	28	705

Countries citing papers authored by Wenman Wu

Since Specialization

Citations

This map shows the geographic impact of Wenman Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wenman Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wenman Wu more than expected).

Fields of papers citing papers by Wenman Wu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wenman Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wenman Wu. The network helps show where Wenman Wu may publish in the future.

Co-authorship network of co-authors of Wenman Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Wenman Wu. A scholar is included among the top collaborators of Wenman Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wenman Wu. Wenman Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Li, Jiaming, Fang Li, Wenman Wu, et al.. (2024). Mutation Ter462GlnextTer17 introduces a tail to C-terminus of protein C and causes venous thrombosis. Thrombosis Research. 240. 109044–109044. 1 indexed citations

Chen, Weizhi, Zhili Zhang, Yu Liu, et al.. (2023). Structural and functional characterization of novel F7 mutations identified in Chinese factor VII‐deficient patients. British Journal of Haematology. 202(3). 623–635. 3 indexed citations

Liang, Qian, et al.. (2023). Fibrinogen BOE II: dysfibrinogenemia with bleeding and defective thrombin binding. Research and Practice in Thrombosis and Haemostasis. 7(5). 102145–102145. 1 indexed citations

Wu, Wenman, Qin Xu, Shifeng Jiang, et al.. (2023). Thrombosis Caused By a Novel Coagulation Factor IX Mutation (FIX Shanghai II). Blood. 142(Supplement 1). 1233–1233. 1 indexed citations

Li, Lei, Jian Li, Xi Wu, et al.. (2023). Evaluation of prothrombotic risk of two PROC hotspot mutations (Arg189Trp and Lys193del) in Chinese population: a retrospective study. Thrombosis Journal. 21(1). 103–103. 3 indexed citations

Li, Lei, Xi Wu, Wenman Wu, Qiulan Ding, & Xuefeng Wang. (2023). A case-report of the unprovoked thrombotic event in a patient with thymoma and severe FVII deficiency. Thrombosis Journal. 21(1). 52–52. 2 indexed citations

Liu, Guoqing, et al.. (2022). F9 mutations causing deletions beyond the serine protease domain confer higher risk for inhibitor development in hemophilia B. Blood. 141(6). 677–680. 2 indexed citations

Wu, Wenman, Zhengwen Jiang, Dazhi Zhang, et al.. (2022). Noninvasive fetal genotyping of single nucleotide variants and linkage analysis for prenatal diagnosis of monogenic disorders. Human Genomics. 16(1). 28–28. 7 indexed citations

Firrman, Jenni, Wenman Wu, Biao Dong, et al.. (2020). Identification of Key Coagulation Activity Determining Elements in Canine Factor VIII. Molecular Therapy — Methods & Clinical Development. 17. 328–336. 3 indexed citations

10.

Wu, Xi, Jing Dai, Fang Li, et al.. (2019). Prothrombin Arg541Trp Mutation Leads to Defective PC (Protein C) Pathway Activation and Constitutes a Novel Genetic Risk Factor for Venous Thrombosis. Arteriosclerosis Thrombosis and Vascular Biology. 40(2). 483–494. 10 indexed citations

11.

Chen, Changming, Qian Liang, Xi Wu, et al.. (2019). Characterization of two large duplications of F9 associated with mild and severe haemophilia B, respectively. Haemophilia. 25(3). 475–483. 5 indexed citations

12.

Ma, Siyu, Changming Chen, Qian Liang, et al.. (2019). Phenotype and genotype of FXIII deficiency in two unrelated probands: identification of a novel F13A1 large deletion mediated by complex rearrangement. Orphanet Journal of Rare Diseases. 14(1). 182–182. 9 indexed citations

13.

Zhang, Wei, Yan Shen, Guowei Zhang, et al.. (2018). Evaluation of the activity levels of rat FVIII and human FVIII delivered by adeno-associated viral vectors both in vitro and in vivo. Blood Cells Molecules and Diseases. 73. 47–54. 5 indexed citations

14.

Wu, Xi, et al.. (2018). Screening and functional exploration of prothrombin Arg596 related mutations in Chinese venous thromboembolism patients. Journal of Clinical Pathology. 71(7). 614–619. 8 indexed citations

15.

Wu, Xi, Qiulan Ding, Xuefeng Wang, Jing Dai, & Wenman Wu. (2018). The prevalence of heterozygous F12 mutations in Chinese population and its relevance to incidents of thrombosis. BMC Medical Genetics. 19(1). 50–50. 3 indexed citations

16.

Chen, Changming, Xiaoling Xie, Xi Wu, et al.. (2017). Complex recombination with deletion in the F8 and duplication in the TMLHE mediated by int22h copies during early embryogenesis. Thrombosis and Haemostasis. 117(8). 1478–1485. 8 indexed citations

17.

Xiao, Shuzhen, Su Wang, Wenman Wu, et al.. (2017). The Resistance Phenotype and Molecular Epidemiology of Klebsiella pneumoniae in Bloodstream Infections in Shanghai, China, 2012–2015. Frontiers in Microbiology. 8. 250–250. 21 indexed citations

18.

Dong, Biao, Xunbao Duan, Hoi Yee Chow, et al.. (2014). Proteomics Analysis of Co-Purifying Cellular Proteins Associated with rAAV Vectors. PLoS ONE. 9(2). e86453–e86453. 29 indexed citations

19.

Wang, Xuefeng, et al.. (2005). Novel aberrant splicings caused by a splice site mutation (IVS1a+5g>a) in F7 gene. Thrombosis and Haemostasis. 93(6). 1077–1081. 4 indexed citations

20.

Fu, Qin, et al.. (2003). Type I coagulation factor V deficiency caused by compound heterozygous mutation of F5 gene. Haemophilia. 9(5). 646–649. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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