Wen Pan

748 total citations
21 papers, 541 citations indexed

About

Wen Pan is a scholar working on Infectious Diseases, Molecular Biology and Immunology. According to data from OpenAlex, Wen Pan has authored 21 papers receiving a total of 541 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Infectious Diseases, 9 papers in Molecular Biology and 9 papers in Immunology. Recurrent topics in Wen Pan's work include Mosquito-borne diseases and control (7 papers), Viral Infections and Vectors (7 papers) and interferon and immune responses (6 papers). Wen Pan is often cited by papers focused on Mosquito-borne diseases and control (7 papers), Viral Infections and Vectors (7 papers) and interferon and immune responses (6 papers). Wen Pan collaborates with scholars based in China, United States and Hong Kong. Wen Pan's co-authors include Jianfeng Dai, Tingting Feng, Penghua Wang, Xiǎohóng Shí, Qihan Wu, Kezhen Wang, Guanghao Li, Yongdong Yan, Xiujuan Wang and Zhiqian Chen and has published in prestigious journals such as PLoS ONE, Biochemical and Biophysical Research Communications and Frontiers in Immunology.

In The Last Decade

Wen Pan

21 papers receiving 534 citations

Peers

Wen Pan

IMMUN

PHEOH

EPIDE

María I. Giraldo United States

Michelle D. Audsley Australia

Mónica Mondragón‐Castelán Mexico

Julio García‐Cordero Mexico

Crystal W. Burke United States

Svetlana V. Scherbik United States

Michael Schreiber Germany

R. Travis Taylor United States

Kimberly Schmitt United States

Jack Komisar United States

María I. Giraldo United States

Wen Pan

271 ×1.3

IMMUN

363 ×1.8

176 ×1.1

360 ×2.6

PHEOH

174 ×2.5

EPIDE

Citations per year, relative to Wen Pan Wen Pan (= 1×) peers María I. Giraldo

Countries citing papers authored by Wen Pan

Since Specialization

Citations

This map shows the geographic impact of Wen Pan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wen Pan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wen Pan more than expected).

Fields of papers citing papers by Wen Pan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wen Pan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wen Pan. The network helps show where Wen Pan may publish in the future.

Co-authorship network of co-authors of Wen Pan

This figure shows the co-authorship network connecting the top 25 collaborators of Wen Pan. A scholar is included among the top collaborators of Wen Pan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wen Pan. Wen Pan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pan, Wen, Junshan Zhang, Mei Yang, et al.. (2024). Porphyrin-based porous organic polymers synthesized using the Alder–Longo method: the most traditional synthetic strategy with exceptional capacity. RSC Advances. 14(29). 20837–20855. 6 indexed citations

Chen, Zhiqiang, Jinyu Zhang, Tingting Feng, et al.. (2024). DDX20 positively regulates the interferon pathway to inhibit viral infection. Antiviral Research. 225. 105875–105875. 7 indexed citations

Chen, Zhiqiang, Jinyu Zhang, Jun Wang, et al.. (2024). N6-methyladenosine RNA modification promotes Severe Fever with Thrombocytopenia Syndrome Virus infection. PLoS Pathogens. 20(11). e1012725–e1012725. 3 indexed citations

Jiang, Tao, Wen Pan, Tingting Feng, et al.. (2024). Ubiquitination of NS1 Confers Differential Adaptation of Zika Virus in Mammalian Hosts and Mosquito Vectors. Advanced Science. 11(39). e2408024–e2408024. 5 indexed citations

Pan, Wen, et al.. (2023). Croton tiglium L. seeds ameliorate loperamide-induced constipation via regulating gastrointestinal hormones and gut microbiota before and after processing. Journal of Ethnopharmacology. 319(Pt 3). 117378–117378. 10 indexed citations

Zhang, Qi, Zhiqiang Chen, Tingting Feng, et al.. (2021). Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) Membrane (M) and Spike (S) Proteins Antagonize Host Type I Interferon Response. Frontiers in Cellular and Infection Microbiology. 11. 766922–766922. 28 indexed citations

Zhang, Jinyu, Yanmei Li, Wen Pan, et al.. (2020). Inhibitory effect of cyclocytidine hydrochloride on vesicular stomatitis virus infection. Acta Biochimica et Biophysica Sinica. 52(5). 576–579. 1 indexed citations

Wang, Kezhen, Xiujuan Wang, Tingting Feng, et al.. (2018). Interferon-stimulated TRIM69 interrupts dengue virus replication by ubiquitinating viral nonstructural protein 3. PLoS Pathogens. 14(8). e1007287–e1007287. 68 indexed citations

Feng, Tingting, et al.. (2018). Ubiquitin-conjugating enzyme UBE2J1 negatively modulates interferon pathway and promotes RNA virus infection. Virology Journal. 15(1). 132–132. 29 indexed citations

10.

Pan, Wen, et al.. (2018). An Immunosuppressive Tick Salivary Gland Protein DsCystatin Interferes With Toll-Like Receptor Signaling by Downregulating TRAF6. Frontiers in Immunology. 9. 1245–1245. 32 indexed citations

11.

Tang, Man, Mingqiang Li, Qizhang Li, et al.. (2017). Conditional potency is a hallmark of viral protein-derived toxic peptides. Medical Hypotheses. 100. 2–3. 1 indexed citations

12.

Feng, Tingting, et al.. (2017). DEAD-Box Helicase DDX25 Is a Negative Regulator of Type I Interferon Pathway and Facilitates RNA Virus Infection. Frontiers in Cellular and Infection Microbiology. 7. 356–356. 18 indexed citations

13.

Pan, Wen, et al.. (2017). Functional characterization of two defensins, HlDFS1 and HlDFS2, from the hard tick Haemaphysalis longicornis. Parasites & Vectors. 10(1). 455–455. 11 indexed citations

14.

Wang, Kezhen, Juanjuan Wang, Wen Pan, et al.. (2016). Glycosphingolipid GM3 is Indispensable for Dengue Virus Genome Replication. International Journal of Biological Sciences. 12(7). 872–883. 20 indexed citations

15.

Wang, Juanjuan, et al.. (2015). Molecular characterization of a defensin gene from a hard tick, Dermacentor silvarum. Parasites & Vectors. 8(1). 25–25. 19 indexed citations

16.

Pan, Wen, et al.. (2015). Effects of Different Doses of Nucleocapsid Protein from Hantaan Virus A9 Strain on Regulation of Interferon Signaling. Viral Immunology. 28(8). 448–454. 8 indexed citations

17.

Pan, Wen, et al.. (2014). Matrix Metalloproteinase 3 Promotes Cellular Anti-Dengue Virus Response via Interaction with Transcription Factor NFκB in Cell Nucleus. PLoS ONE. 9(1). e84748–e84748. 32 indexed citations

18.

Li, Guanghao, Tingting Feng, Wen Pan, Xiǎohóng Shí, & Jianfeng Dai. (2014). DEAD-box RNA helicase DDX3X inhibits DENV replication via regulating type one interferon pathway. Biochemical and Biophysical Research Communications. 456(1). 327–332. 54 indexed citations

19.

Pan, Wen, et al.. (2012). Guanylate-binding protein 1 participates in cellular antiviral response to dengue virus. Virology Journal. 9(1). 292–292. 57 indexed citations

20.

Dai, Jianfeng, Wen Pan, & Penghua Wang. (2011). ISG15 facilitates cellular antiviral response to dengue and west nile virus infection in vitro. Virology Journal. 8(1). 468–468. 76 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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