Wei‐Wei Luo

1.3k total citations
33 papers, 1.0k citations indexed

About

Wei‐Wei Luo is a scholar working on Immunology, Molecular Biology and Cancer Research. According to data from OpenAlex, Wei‐Wei Luo has authored 33 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Immunology, 10 papers in Molecular Biology and 9 papers in Cancer Research. Recurrent topics in Wei‐Wei Luo's work include interferon and immune responses (18 papers), Viral Infections and Vectors (6 papers) and Immune Response and Inflammation (6 papers). Wei‐Wei Luo is often cited by papers focused on interferon and immune responses (18 papers), Viral Infections and Vectors (6 papers) and Immune Response and Inflammation (6 papers). Wei‐Wei Luo collaborates with scholars based in China, United States and India. Wei‐Wei Luo's co-authors include Hong‐Bing Shu, Qing Yang, Shu Li, Huan Lian, Chen Li, Bo Zhong, Yan‐Yi Wang, Ming‐Ming Hu, Jin Wei and Suyun Wang and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Immunity.

In The Last Decade

Wei‐Wei Luo

27 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei‐Wei Luo China 17 724 494 283 167 123 33 1.0k
Guiwen Yang China 17 562 0.8× 461 0.9× 101 0.4× 84 0.5× 175 1.4× 52 1.1k
Long-Feng Lu China 19 785 1.1× 289 0.6× 272 1.0× 62 0.4× 240 2.0× 58 1.0k
Chunrong Yang China 26 1.2k 1.7× 453 0.9× 213 0.8× 40 0.2× 174 1.4× 66 1.7k
Hao Feng China 22 1.0k 1.4× 363 0.7× 220 0.8× 90 0.5× 325 2.6× 96 1.5k
Bo Ma China 10 313 0.4× 207 0.4× 194 0.7× 186 1.1× 64 0.5× 47 719
Yong Ran China 21 994 1.4× 725 1.5× 333 1.2× 179 1.1× 252 2.0× 30 1.5k
Rodney P. Kincaid United States 15 305 0.4× 629 1.3× 141 0.5× 152 0.9× 456 3.7× 23 1.1k
Xavier Romero Spain 20 1.0k 1.4× 204 0.4× 63 0.2× 162 1.0× 33 0.3× 38 1.4k
Sean M. Gregory United States 15 491 0.7× 370 0.7× 143 0.5× 373 2.2× 37 0.3× 24 1.1k
Seyedtaghi Takyar United States 8 288 0.4× 925 1.9× 59 0.2× 82 0.5× 68 0.6× 9 1.3k

Countries citing papers authored by Wei‐Wei Luo

Since Specialization
Citations

This map shows the geographic impact of Wei‐Wei Luo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei‐Wei Luo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei‐Wei Luo more than expected).

Fields of papers citing papers by Wei‐Wei Luo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei‐Wei Luo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei‐Wei Luo. The network helps show where Wei‐Wei Luo may publish in the future.

Co-authorship network of co-authors of Wei‐Wei Luo

This figure shows the co-authorship network connecting the top 25 collaborators of Wei‐Wei Luo. A scholar is included among the top collaborators of Wei‐Wei Luo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei‐Wei Luo. Wei‐Wei Luo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Luo, Wei‐Wei, et al.. (2025). Application of neurobiofeedback therapy technology on social skills and emotion regulation in children with autism spectrum disorder. World Journal of Psychiatry. 15(12). 111522–111522.
3.
4.
Xu, Zhisheng, Suyun Wang, Yining Yang, et al.. (2024). LDLR is an entry receptor for Crimean-Congo hemorrhagic fever virus. Cell Research. 34(2). 140–150. 21 indexed citations
5.
Tong, Zhen, et al.. (2024). Activation of the cGAS‐STING‐IRF3 Axis by Type I and II Interferons Contributes to Host Defense. Advanced Science. 11(35). e2308890–e2308890. 16 indexed citations
6.
Zhang, Tong, et al.. (2024). Wearable EEG Neurofeedback Based-on Machine Learning Algorithms for Children with Autism: A Randomized, Placebo-controlled Study. Current Medical Science. 44(6). 1141–1147. 1 indexed citations
7.
Chen, Geng, Ying Zhou, Xiaomu Yu, et al.. (2022). Genome-Wide Association Study Reveals SNPs and Candidate Genes Related to Growth and Body Shape in Bighead Carp (Hypophthalmichthys nobilis). Marine Biotechnology. 24(6). 1138–1147. 6 indexed citations
8.
Luo, Wei‐Wei, Junru Wang, Ying Zhou, et al.. (2022). Dynamic mRNA and miRNA expression of the head during early development in bighead carp (Hypophthalmichthys nobilis). BMC Genomics. 23(1). 168–168. 6 indexed citations
9.
Luo, Wei‐Wei, Zhen Tong, Pan Cao, et al.. (2022). Transcription-independent regulation of STING activation and innate immune responses by IRF8 in monocytes. Nature Communications. 13(1). 4822–4822. 30 indexed citations
10.
11.
Zhang, Hongyan, Zhi-Sheng Xu, Yong Ran, et al.. (2020). USP44 positively regulates innate immune response to DNA viruses through deubiquitinating MITA. PLoS Pathogens. 16(1). e1008178–e1008178. 28 indexed citations
12.
Nie, Ying, Yan Yang, Yong Ran, et al.. (2020). Ubiquitination of TLR3 by TRIM3 signals its ESCRT-mediated trafficking to the endolysosomes for innate antiviral response. Proceedings of the National Academy of Sciences. 117(38). 23707–23716. 30 indexed citations
13.
Ran, Yong, et al.. (2019). YIPF5 Is Essential for Innate Immunity to DNA Virus and Facilitates COPII-Dependent STING Trafficking. The Journal of Immunology. 203(6). 1560–1570. 49 indexed citations
14.
Cao, Pan, Wei‐Wei Luo, Chen Li, et al.. (2019). The heterogeneous nuclear ribonucleoprotein hnRNPM inhibits RNA virus-triggered innate immunity by antagonizing RNA sensing of RIG-I-like receptors. PLoS Pathogens. 15(8). e1007983–e1007983. 21 indexed citations
15.
Wei, Jin, Huan Lian, Wei Guo, et al.. (2018). SNX8 modulates innate immune response to DNA virus by mediating trafficking and activation of MITA. PLoS Pathogens. 14(10). e1007336–e1007336. 34 indexed citations
16.
Lian, Huan, Jin Wei, Ru Zang, et al.. (2018). ZCCHC3 is a co-sensor of cGAS for dsDNA recognition in innate immune response. Nature Communications. 9(1). 3349–3349. 115 indexed citations
17.
Lian, Huan, Ru Zang, Jin Wei, et al.. (2018). The Zinc-Finger Protein ZCCHC3 Binds RNA and Facilitates Viral RNA Sensing and Activation of the RIG-I-like Receptors. Immunity. 49(3). 438–448.e5. 110 indexed citations
18.
Zhou, Ying, Wei‐Wei Luo, Xiaomu Yu, Qingshan Liu, & Jingou Tong. (2018). Brain and intestine transcriptome analyses and identification of genes involved in feed conversion efficiency of Yellow River carp (Cyprinus carpio haematopterus). Comparative Biochemistry and Physiology Part D Genomics and Proteomics. 29. 221–227. 17 indexed citations
19.
Yang, Qing, Tiantian Liu, Heng Lin, et al.. (2017). TRIM32-TAX1BP1-dependent selective autophagic degradation of TRIF negatively regulates TLR3/4-mediated innate immune responses. PLoS Pathogens. 13(9). e1006600–e1006600. 96 indexed citations
20.
Luo, Wei‐Wei, Shu Li, Chen Li, et al.. (2016). iRhom2 is essential for innate immunity to DNA viruses by mediating trafficking and stability of the adaptor STING. Nature Immunology. 17(9). 1057–1066. 209 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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