Weining Wu

2.1k total citations
43 papers, 1.5k citations indexed

About

Weining Wu is a scholar working on Molecular Biology, Cancer Research and Infectious Diseases. According to data from OpenAlex, Weining Wu has authored 43 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 16 papers in Cancer Research and 14 papers in Infectious Diseases. Recurrent topics in Weining Wu's work include MicroRNA in disease regulation (14 papers), Viral Infections and Vectors (11 papers) and Circular RNAs in diseases (10 papers). Weining Wu is often cited by papers focused on MicroRNA in disease regulation (14 papers), Viral Infections and Vectors (11 papers) and Circular RNAs in diseases (10 papers). Weining Wu collaborates with scholars based in China, United Kingdom and United States. Weining Wu's co-authors include Tianfu Yu, Yingyi Wang, Yongping You, Julian A. Hiscox, Junxia Zhang, John N. Barr, Er Nie, Tongle Zhi, Youzhi Wu and Xu Zhou and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Oncogene.

In The Last Decade

Weining Wu

42 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Weining Wu China 24 897 669 347 279 134 43 1.5k
Alexandre Akoulitchev United Kingdom 16 1.3k 1.5× 552 0.8× 92 0.3× 146 0.5× 129 1.0× 35 1.6k
Shaowen Wang China 23 817 0.9× 224 0.3× 118 0.3× 109 0.4× 536 4.0× 78 1.5k
Guigao Lin China 17 688 0.8× 320 0.5× 109 0.3× 177 0.6× 61 0.5× 71 1.2k
Walter N. Moss United States 27 1.8k 2.0× 538 0.8× 205 0.6× 380 1.4× 235 1.8× 74 2.2k
Massimo Caputi United States 22 1.7k 1.9× 157 0.2× 179 0.5× 109 0.4× 163 1.2× 38 2.0k
Donna M. D’Agostino Italy 26 938 1.0× 411 0.6× 161 0.5× 189 0.7× 935 7.0× 58 2.0k
Liwei Rong Canada 20 1.5k 1.6× 143 0.2× 478 1.4× 305 1.1× 684 5.1× 30 2.3k
Ying Poi Liu Netherlands 23 2.1k 2.3× 1.1k 1.7× 84 0.2× 81 0.3× 180 1.3× 42 2.7k
Maurizio Ceppi Switzerland 16 782 0.9× 472 0.7× 81 0.2× 97 0.3× 562 4.2× 34 1.5k

Countries citing papers authored by Weining Wu

Since Specialization
Citations

This map shows the geographic impact of Weining Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Weining Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Weining Wu more than expected).

Fields of papers citing papers by Weining Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Weining Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Weining Wu. The network helps show where Weining Wu may publish in the future.

Co-authorship network of co-authors of Weining Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Weining Wu. A scholar is included among the top collaborators of Weining Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Weining Wu. Weining Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Gu, Shen, Weining Wu, Chao Wu, et al.. (2025). RBMS1 promotes the proliferation of glioma cells via regulation of the c-Myc–SSH1 axis. Biochemical and Biophysical Research Communications. 758. 151586–151586.
2.
Wu, Weining, Neil Berry, Sarah Kempster, et al.. (2022). SARS-CoV-2 Virus-like Particles Produced by a Single Recombinant Baculovirus Generate Anti-S Antibody and Protect against Variant Challenge. Viruses. 14(5). 914–914. 15 indexed citations
3.
Xia, Xian, Weining Wu, Yanxiang Cui, Polly Roy, & Z. Hong Zhou. (2021). Bluetongue virus capsid protein VP5 perforates membranes at low endosomal pH during viral entry. Nature Microbiology. 6(11). 1424–1432. 19 indexed citations
4.
Wu, Weining & Polly Roy. (2021). Sialic Acid Binding Sites in VP2 of Bluetongue Virus and Their Use during Virus Entry. Journal of Virology. 96(1). 11 indexed citations
5.
Wu, Weining, Tianfu Yu, Youzhi Wu, et al.. (2019). The miR155HG/miR-185/ANXA2 loop contributes to glioblastoma growth and progression. Journal of Experimental & Clinical Cancer Research. 38(1). 133–133. 95 indexed citations
6.
Zeng, Ailiang, Zhiyun Wei, Wei Yan, et al.. (2018). Exosomal transfer of miR-151a enhances chemosensitivity to temozolomide in drug-resistant glioblastoma. Cancer Letters. 436. 10–21. 161 indexed citations
7.
Nie, Er, Xin Jin, Weining Wu, et al.. (2018). Fstl1/DIP2A/MGMT signaling pathway plays important roles in temozolomide resistance in glioblastoma. Oncogene. 38(15). 2706–2721. 37 indexed citations
8.
Tian, Wei, et al.. (2018). MiRNA-139–3p inhibits the proliferation, invasion, and migration of human glioma cells by targeting MDA-9/syntenin. Biochemical and Biophysical Research Communications. 508(1). 295–301. 26 indexed citations
9.
Hu, Qi, Weining Wu, Ailiang Zeng, et al.. (2017). Polycomb group expression signatures in the malignant progression of gliomas. Oncology Letters. 13(4). 2583–2590. 3 indexed citations
10.
Wang, Yingyi, Tianfu Yu, Er Nie, et al.. (2017). Blocking MIR155HG/miR-155 axis inhibits mesenchymal transition in glioma. Neuro-Oncology. 19(9). 1195–1205. 109 indexed citations
11.
Wu, Weining, Qi Hu, Er Nie, et al.. (2017). Hypoxia induces H19 expression through direct and indirect Hif-1α activity, promoting oncogenic effects in glioblastoma. Scientific Reports. 7(1). 45029–45029. 106 indexed citations
12.
13.
Nie, Er, Xin Jin, Weining Wu, et al.. (2016). BACH1 Promotes Temozolomide Resistance in Glioblastoma through Antagonizing the Function of p53. Scientific Reports. 6(1). 39743–39743. 32 indexed citations
14.
15.
Wang, Yuan, et al.. (2014). Purification and photodynamic bioactivity of phycoerythrin and phycocyanin from Porphyra yezoensis Ueda. Journal of Ocean University of China. 13(3). 479–484. 10 indexed citations
16.
Ariza, A., Cheryl Walter, Kyle C. Dent, et al.. (2013). Nucleocapsid protein structures from orthobunyaviruses reveal insight into ribonucleoprotein architecture and RNA polymerization. Nucleic Acids Research. 41(11). 5912–5926. 65 indexed citations
17.
Wu, Weining, Andrew Macdonald, Julian A. Hiscox, & John N. Barr. (2011). Different NF-κB activation characteristics of human respiratory syncytial virus subgroups A and B. Microbial Pathogenesis. 52(3). 184–191. 8 indexed citations
18.
Wu, Weining, Diane C. Munday, Gareth Howell, et al.. (2011). Characterization of the Interaction between Human Respiratory Syncytial Virus and the Cell Cycle in Continuous Cell Culture and Primary Human Airway Epithelial Cells. Journal of Virology. 85(19). 10300–10309. 28 indexed citations
19.
Munday, Diane C., Edward Emmott, Rebecca Surtees, et al.. (2010). Quantitative Proteomic Analysis of A549 Cells Infected with Human Respiratory Syncytial Virus. Molecular & Cellular Proteomics. 9(11). 2438–2459. 77 indexed citations
20.
He, Peimin, et al.. (2002). STUDIES ON ULTRASTRUCTURE OF PYRENOID FROM SEVERAL ALGAE. Acta Hydrobiologica Sinica. 26(4). 327–334. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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