Weihua Gan

448 total citations
19 papers, 319 citations indexed

About

Weihua Gan is a scholar working on Molecular Biology, Cancer Research and Nephrology. According to data from OpenAlex, Weihua Gan has authored 19 papers receiving a total of 319 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Cancer Research and 3 papers in Nephrology. Recurrent topics in Weihua Gan's work include RNA modifications and cancer (5 papers), Cancer-related molecular mechanisms research (5 papers) and RNA Research and Splicing (5 papers). Weihua Gan is often cited by papers focused on RNA modifications and cancer (5 papers), Cancer-related molecular mechanisms research (5 papers) and RNA Research and Splicing (5 papers). Weihua Gan collaborates with scholars based in China and United States. Weihua Gan's co-authors include Aiqing Zhang, Yunwen Yang, Jiayu Song, Bin Wang, Shanwen Li, Bi‐Cheng Liu, Zuo‐Lin Li, Tao‐Tao Tang, Huimin Shi and Lin‐Li Lv and has published in prestigious journals such as PLoS ONE, Biochemical and Biophysical Research Communications and British Journal of Pharmacology.

In The Last Decade

Weihua Gan

18 papers receiving 318 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Weihua Gan China 9 210 111 51 48 39 19 319
WenXing Fan China 11 194 0.9× 130 1.2× 42 0.8× 46 1.0× 24 0.6× 25 313
Mingyue Rao China 11 223 1.1× 85 0.8× 51 1.0× 42 0.9× 31 0.8× 18 400
Zongwei Lin China 10 204 1.0× 90 0.8× 36 0.7× 20 0.4× 69 1.8× 19 388
Wenfang Peng China 11 275 1.3× 257 2.3× 19 0.4× 33 0.7× 50 1.3× 23 435
Xiaoyan Yu China 11 164 0.8× 97 0.9× 16 0.3× 25 0.5× 74 1.9× 25 324
Yujiao Sun China 9 109 0.5× 42 0.4× 22 0.4× 26 0.5× 21 0.5× 18 226
Yawei Huang China 7 241 1.1× 188 1.7× 39 0.8× 31 0.6× 9 0.2× 19 369
Beilei Zhao China 11 197 0.9× 40 0.4× 77 1.5× 106 2.2× 29 0.7× 20 430
Chihiro Horimai Japan 7 155 0.7× 47 0.4× 38 0.7× 51 1.1× 61 1.6× 9 378

Countries citing papers authored by Weihua Gan

Since Specialization
Citations

This map shows the geographic impact of Weihua Gan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Weihua Gan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Weihua Gan more than expected).

Fields of papers citing papers by Weihua Gan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Weihua Gan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Weihua Gan. The network helps show where Weihua Gan may publish in the future.

Co-authorship network of co-authors of Weihua Gan

This figure shows the co-authorship network connecting the top 25 collaborators of Weihua Gan. A scholar is included among the top collaborators of Weihua Gan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Weihua Gan. Weihua Gan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Zhang, Zhenxing, et al.. (2024). The potential role of differentially expressed tRNA-derived fragments in high glucose-induced podocytes. Renal Failure. 46(1). 2318413–2318413. 4 indexed citations
3.
Li, Shanwen, et al.. (2023). tRF-003634 alleviates adriamycin-induced podocyte injury by reducing the stability of TLR4 mRNA. PLoS ONE. 18(10). e0293043–e0293043. 1 indexed citations
4.
Shi, Huimin, Hui Zheng, Emily Wang, et al.. (2023). Satellite cell-derived exosome-mediated delivery of microRNA-23a/27a/26a cluster ameliorates the renal tubulointerstitial fibrosis in mouse diabetic nephropathy. Acta Pharmacologica Sinica. 44(12). 2455–2468. 20 indexed citations
5.
Huang, Chan, Ling Ding, Huimin Shi, et al.. (2023). Expression profiles and potential roles of serum tRNA‑derived fragments in diabetic nephropathy. Experimental and Therapeutic Medicine. 26(1). 311–311. 6 indexed citations
6.
Song, Jiayu, et al.. (2023). Vitexin attenuates chronic kidney disease by inhibiting renal tubular epithelial cell ferroptosis via NRF2 activation. Molecular Medicine. 29(1). 147–147. 46 indexed citations
7.
Li, Xian, et al.. (2023). Mitochondrial quality control in acute kidney disease. Journal of Nephrology. 36(5). 1283–1291. 3 indexed citations
8.
Song, Jiayu, et al.. (2022). Mitochondrial Targeted Antioxidant SKQ1 Ameliorates Acute Kidney Injury by Inhibiting Ferroptosis. Oxidative Medicine and Cellular Longevity. 2022(1). 2223957–2223957. 25 indexed citations
9.
Yin, Qing, Yajie Zhao, Tao‐Tao Tang, et al.. (2022). MiR-155 deficiency protects renal tubular epithelial cells from telomeric and genomic DNA damage in cisplatin-induced acute kidney injury. Theranostics. 12(10). 4753–4766. 26 indexed citations
10.
Song, Jiayu, Jianxia Zhang, Aiqing Zhang, et al.. (2022). Case Report: A Novel Non-Canonical Splice Site Variant (c.1638+7T>C) in TRPM6 Cause Primary Homagnesemia With Secondary Hocalcemia. Frontiers in Pediatrics. 10. 834241–834241.
11.
Zheng, Hui, et al.. (2022). Expression profiles of tRNA‑derived fragments in high glucose‑treated tubular epithelial cells. Experimental and Therapeutic Medicine. 25(1). 26–26. 6 indexed citations
12.
Wang, Bin, Ze‐Mu Wang, Weihua Gan, et al.. (2020). Macrophage-Derived Exosomal Mir-155 Regulating Cardiomyocyte Pyroptosis and Hypertrophy in Uremic Cardiomyopathy. JACC Basic to Translational Science. 5(2). 148–166. 75 indexed citations
13.
Miao, Hongjun, Han Li, Mingfu Wu, et al.. (2020). Update on recommendations for the diagnosis and treatment of SARS-CoV-2 infection in children. European Journal of Clinical Microbiology & Infectious Diseases. 39(12). 2211–2223. 11 indexed citations
14.
Li, Shanwen, Yiwen Liu, Xiaowei He, et al.. (2020). tRNA‐Derived Fragments in Podocytes with Adriamycin‐Induced Injury Reveal the Potential Mechanism of Idiopathic Nephrotic Syndrome. BioMed Research International. 2020(1). 7826763–7826763. 8 indexed citations
15.
Shi, Huimin, Yue Wu, Yuepeng Cao, et al.. (2019). tRNA-derived fragments (tRFs) contribute to podocyte differentiation. Biochemical and Biophysical Research Communications. 521(1). 1–8. 19 indexed citations
16.
Guan, Zheng, Shanwen Li, Huimin Shi, et al.. (2019). Gene expression profiling analysis reveals that the long non‑coding RNA uc.412 is involved in mesangial cell proliferation. Molecular Medicine Reports. 20(6). 5297–5303. 4 indexed citations
17.
Zhang, Aiqing, Ying Han, Bin Wang, Shanwen Li, & Weihua Gan. (2015). Beyond Gap Junction Channel Function: the Expression of Cx43 Contributes to Aldosterone-Induced Mesangial Cell Proliferation via the ERK1/2 and PKC Pathways. Cellular Physiology and Biochemistry. 36(3). 1210–1222. 14 indexed citations
18.
Wang, Shixia, et al.. (2012). Expression and immunogenicity of novel subunit enterovirus 71 VP1 antigens. Biochemical and Biophysical Research Communications. 420(4). 755–761. 7 indexed citations
19.
Gan, Weihua, et al.. (2010). Over-expression of NYGGF4 (PID1) inhibits glucose transport in skeletal myotubes by blocking the IRS1/PI3K/AKT insulin pathway. Molecular Genetics and Metabolism. 102(3). 374–377. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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