Wei Shuai

1.5k total citations
69 papers, 1.1k citations indexed

About

Wei Shuai is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Oncology. According to data from OpenAlex, Wei Shuai has authored 69 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Cardiology and Cardiovascular Medicine, 21 papers in Molecular Biology and 11 papers in Oncology. Recurrent topics in Wei Shuai's work include Cardiac electrophysiology and arrhythmias (14 papers), Cardiovascular Function and Risk Factors (12 papers) and Atrial Fibrillation Management and Outcomes (11 papers). Wei Shuai is often cited by papers focused on Cardiac electrophysiology and arrhythmias (14 papers), Cardiovascular Function and Risk Factors (12 papers) and Atrial Fibrillation Management and Outcomes (11 papers). Wei Shuai collaborates with scholars based in China, United States and Israel. Wei Shuai's co-authors include He Huang, Bin Kong, Zheng Xiao, Chang Dai, Jin Fang, Bin Kong, Caijie Shen, Hongjie Yang, Xiaobo Jiang and Hui Fu and has published in prestigious journals such as Journal of the American Chemical Society, SHILAP Revista de lepidopterología and Molecular and Cellular Biology.

In The Last Decade

Wei Shuai

63 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei Shuai China 19 518 365 220 165 93 69 1.1k
Eduardo Oliver Spain 20 569 1.1× 548 1.5× 328 1.5× 173 1.0× 105 1.1× 55 1.4k
Navid Koleini Canada 13 373 0.7× 397 1.1× 132 0.6× 125 0.8× 76 0.8× 30 1.0k
Urna Kansakar United States 20 440 0.8× 260 0.7× 103 0.5× 148 0.9× 157 1.7× 53 1.1k
Maria Grandoch Germany 26 698 1.3× 380 1.0× 124 0.6× 183 1.1× 165 1.8× 71 1.7k
Bin Kong China 20 368 0.7× 342 0.9× 88 0.4× 78 0.5× 74 0.8× 62 968
Amanda Versteilen Netherlands 16 406 0.8× 198 0.5× 100 0.5× 78 0.5× 84 0.9× 22 1.2k
Carlos Tarín Spain 17 469 0.9× 213 0.6× 190 0.9× 95 0.6× 211 2.3× 17 1.2k
Yaguang Bi China 18 725 1.4× 202 0.6× 226 1.0× 172 1.0× 122 1.3× 23 1.4k
Boris Manoury France 18 636 1.2× 368 1.0× 450 2.0× 82 0.5× 92 1.0× 31 1.4k
Yuan Guo China 22 386 0.7× 121 0.3× 147 0.7× 213 1.3× 149 1.6× 82 1.3k

Countries citing papers authored by Wei Shuai

Since Specialization
Citations

This map shows the geographic impact of Wei Shuai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Shuai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Shuai more than expected).

Fields of papers citing papers by Wei Shuai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Shuai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Shuai. The network helps show where Wei Shuai may publish in the future.

Co-authorship network of co-authors of Wei Shuai

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Shuai. A scholar is included among the top collaborators of Wei Shuai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Shuai. Wei Shuai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tang, Ling, Song Lv, Jianping Li, et al.. (2025). Synergistic effects of air pollutants and CO2 at Chinese thermal power plants based on real-monitored data. Journal of Environmental Management. 388. 125900–125900.
2.
Meng, Hong, et al.. (2025). USP38 stabilizes NOX4 and activates CaMKII to enhance ventricular arrhythmias susceptibility in CKD mice. Free Radical Biology and Medicine. 237. 344–356. 1 indexed citations
3.
4.
Yang, Yuying, Junyao Li, Zhiqiang Niu, et al.. (2025). RNA-seq analysis of shrimp tropomyosin-induced allergic reactions through PI3K/Akt pathway. Frontiers in Nutrition. 12. 1623971–1623971.
5.
Xiao, Zheng, et al.. (2025). Ubiquitin-specific protease 38 exacerbates diabetic cardiomyopathy via post-translational modification of ACAD11. Redox Biology. 84. 103704–103704. 2 indexed citations
6.
Meng, Hong, Liang Guo, Junyong Zhu, et al.. (2024). Anxiety Disorder and Cardiovascular Disease: A Two-Sample Mendelian Randomization Study. ESC Heart Failure. 11(2). 1174–1181. 12 indexed citations
7.
8.
Xiao, Zheng, et al.. (2024). Ubiquitin specific protease 38 aggravates pathological cardiac remodeling by stabilizing phospho-TBK1. International Journal of Biological Sciences. 20(5). 1815–1832. 9 indexed citations
9.
Xiao, Zheng, Hongjie Yang, Hong Meng, et al.. (2024). Ubiquitin-specific protease 38 promotes atrial fibrillation in diabetic mice by stabilizing iron regulatory protein 2. Free Radical Biology and Medicine. 224. 88–102. 6 indexed citations
10.
Shuai, Wei, Chen Chen, Enguang Zuo, et al.. (2023). Rapid diagnosis of rheumatoid arthritis and ankylosing spondylitis based on Fourier transform infrared spectroscopy and deep learning. Photodiagnosis and Photodynamic Therapy. 45. 103885–103885. 10 indexed citations
11.
Gong, Yang, Bin Kong, Wei Shuai, et al.. (2023). USP38 regulates inflammatory cardiac remodeling after myocardial infarction. Clinical Science. 137(21). 1665–1681. 6 indexed citations
12.
Liu, Qi, Junyong Zhu, Bin Kong, Wei Shuai, & He Huang. (2023). Tirzepatide attenuates lipopolysaccharide-induced left ventricular remodeling and dysfunction by inhibiting the TLR4/NF-kB/NLRP3 pathway. International Immunopharmacology. 120. 110311–110311. 38 indexed citations
13.
Shuai, Wei, et al.. (2023). 5-Methoxytryptophan alleviates atrial structural remodeling in ibrutinib-associated atrial fibrillation. Heliyon. 9(9). e19501–e19501. 4 indexed citations
14.
Zhang, Jingjing, Chenyu Li, Wei Shuai, et al.. (2023). maresin2 fine-tunes ULK1 O-GlcNAcylation to improve post myocardial infarction remodeling. European Journal of Pharmacology. 962. 176223–176223. 1 indexed citations
15.
Yang, Jian, Wei Shuai, Jun Yang, et al.. (2020). Deletion of Microfibrillar‐Associated Protein 4 Attenuates Left Ventricular Remodeling and Dysfunction in Heart Failure. Journal of the American Heart Association. 9(17). e015307–e015307. 25 indexed citations
16.
Fu, Hui, Wei Shuai, Bin Kong, Xiaobo Jiang, & He Huang. (2020). MD1 Depletion Predisposes to Ventricular Arrhythmias in the Setting of Myocardial Infarction. Heart Lung and Circulation. 30(6). 869–881. 8 indexed citations
17.
Shuai, Wei, Bin Kong, Hongjie Yang, Hui Fu, & He Huang. (2020). Loss of Myeloid Differentiation Protein 1 Promotes Atrial Fibrillation in Heart Failure with Preserved Ejection Fraction. ESC Heart Failure. 7(2). 626–638. 12 indexed citations
18.
Shuai, Wei, Bin Kong, Hui Fu, Caijie Shen, & He Huang. (2019). Loss of MD1 increases vulnerability to ventricular arrhythmia in diet-induced obesity mice via enhanced activation of the TLR4/MyD88/CaMKII signaling pathway. Nutrition Metabolism and Cardiovascular Diseases. 29(9). 991–998. 25 indexed citations
19.
Xiong, Qiutang, Wei Shuai, Chenliang Zhou, & Weiguo Dong. (2019). Circulating bilirubin level is determined by both erythrocyte amounts and the proportion of aged erythrocytes in ageing and cardiovascular diseases. Biomedicine & Pharmacotherapy. 123. 109744–109744. 6 indexed citations
20.
Shen, Caijie, Bin Kong, Yu Liu, et al.. (2018). YY1-induced upregulation of lncRNA KCNQ1OT1 regulates angiotensin II-induced atrial fibrillation by modulating miR-384b/CACNA1C axis. Biochemical and Biophysical Research Communications. 505(1). 134–140. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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