Wei Meng

2.4k total citations
70 papers, 1.8k citations indexed

About

Wei Meng is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Wei Meng has authored 70 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 52 papers in Molecular Biology, 31 papers in Cancer Research and 7 papers in Oncology. Recurrent topics in Wei Meng's work include MicroRNA in disease regulation (24 papers), Cancer-related molecular mechanisms research (18 papers) and Circular RNAs in diseases (13 papers). Wei Meng is often cited by papers focused on MicroRNA in disease regulation (24 papers), Cancer-related molecular mechanisms research (18 papers) and Circular RNAs in diseases (13 papers). Wei Meng collaborates with scholars based in China, United States and Italy. Wei Meng's co-authors include Arnab Chakravarti, Ri Cui, Carlo M. Croce, Jie Ma, Yujie Tang, Tim Lautenschlaeger, Guang Liang, Matteo Fassan, Lei Zhang and Meng Li and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Journal of Clinical Oncology.

In The Last Decade

Wei Meng

64 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei Meng China 22 1.4k 839 239 204 165 70 1.8k
Ingrid Øra Sweden 28 1.5k 1.1× 801 1.0× 175 0.7× 537 2.6× 154 0.9× 81 2.6k
Carmen Doebele Germany 15 1.8k 1.3× 1.4k 1.6× 86 0.4× 172 0.8× 159 1.0× 15 2.3k
Jessica E.S. Shay United States 11 903 0.6× 932 1.1× 107 0.4× 183 0.9× 87 0.5× 22 1.5k
D. Hiller United States 15 853 0.6× 451 0.5× 272 1.1× 379 1.9× 490 3.0× 35 1.8k
David Ferland-McCollough United Kingdom 12 994 0.7× 677 0.8× 69 0.3× 121 0.6× 102 0.6× 12 1.5k
Sina Koch Germany 15 1.3k 0.9× 288 0.3× 298 1.2× 417 2.0× 87 0.5× 22 2.0k
Kevin McDonnell United States 16 961 0.7× 499 0.6× 102 0.4× 497 2.4× 95 0.6× 39 1.5k
Aline Meulle France 9 671 0.5× 607 0.7× 111 0.5× 553 2.7× 87 0.5× 12 1.4k
Philip Kahl Germany 25 1.6k 1.1× 466 0.6× 143 0.6× 284 1.4× 310 1.9× 47 2.2k
Ping Guo China 14 649 0.5× 279 0.3× 122 0.5× 286 1.4× 67 0.4× 33 1.1k

Countries citing papers authored by Wei Meng

Since Specialization
Citations

This map shows the geographic impact of Wei Meng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Meng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Meng more than expected).

Fields of papers citing papers by Wei Meng

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Meng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Meng. The network helps show where Wei Meng may publish in the future.

Co-authorship network of co-authors of Wei Meng

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Meng. A scholar is included among the top collaborators of Wei Meng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Meng. Wei Meng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lv, Ying, Xiuzhu Weng, Xinyi Zhang, et al.. (2025). Quercetin alleviates postmenopausal atherosclerosis by suppressing endothelial cell ferroptosis via regulating the KEAP1/NRF2/GPX4 signalling pathway. British Journal of Pharmacology. 183(3). 620–643.
2.
Zhou, Xiangtian, Chaochao Luo, Wei Meng, et al.. (2025). Deciphering the Oncogenic Landscape of Hepatocytes Through Integrated Single‐Nucleus and Bulk RNA‐Seq of Hepatocellular Carcinoma. Advanced Science. 12(14). e2412944–e2412944. 1 indexed citations
3.
Wang, Rongrong, Danlei Chen, Wei Meng, et al.. (2025). Histone lactylation-induced premature senescence contributes to 1-nitropyrene-Induced chronic obstructive pulmonary disease. Redox Biology. 84. 103703–103703. 2 indexed citations
4.
5.
6.
Meng, Wei, et al.. (2024). Circular RNA hsa_circ_0001610 promotes prostate cancer progression by sponging miR-1324 and upregulating PTK6. Gene. 930. 148818–148818. 1 indexed citations
7.
Mai, Rongyun, Zhihong Tang, Xiaobo Wang, et al.. (2024). Ubiquitination of P53 Regulated by Ubiquitin-Specific Protease 14 Delays the Invasion of Hepatitis B Virus and the Development of Hepatitis. Viral Immunology. 37(9). 432–439.
8.
Meng, Wei, et al.. (2023). Cellular senescence: A potential mode of circular RNAs regulating prostate cancer. SHILAP Revista de lepidopterología. 2(4). 3 indexed citations
9.
Jiang, Zhongmin, et al.. (2023). Detection of CSTF2 by nano fluorescent probe and its correlation with malignant biological characteristics in liver cancer. PubMed Central.
10.
Cui, Xiaohan, et al.. (2023). POU5F1 promotes the proliferation, migration, and invasion of gastric cancer cells by reducing the ubiquitination level of TRAF6. Cell Death and Disease. 14(12). 802–802. 14 indexed citations
11.
Zhou, Lin, et al.. (2021). Ubiquitin-Specific Peptidase 7: A Novel Deubiquitinase That Regulates Protein Homeostasis and Cancers. Frontiers in Oncology. 11. 784672–784672. 17 indexed citations
12.
Meng, Wei, Jiajia Wang, Baocheng Wang, et al.. (2018). CDK7 inhibition is a novel therapeutic strategy against GBM both in vitro and in vivo. Cancer Management and Research. Volume 10. 5747–5758. 31 indexed citations
13.
Meng, Wei, Baocheng Wang, Weiwei Mao, et al.. (2018). Enhanced efficacy of histone deacetylase inhibitor combined with bromodomain inhibitor in glioblastoma. Journal of Experimental & Clinical Cancer Research. 37(1). 241–241. 31 indexed citations
14.
Meng, Wei, Jason A. Efstathiou, Rajbir Singh, et al.. (2018). MicroRNA Biomarkers for Patients With Muscle-Invasive Bladder Cancer Undergoing Selective Bladder-Sparing Trimodality Treatment. International Journal of Radiation Oncology*Biology*Physics. 104(1). 197–206. 17 indexed citations
15.
Huang, Wei, Yuliang Feng, Jialiang Liang, et al.. (2018). Loss of microRNA-128 promotes cardiomyocyte proliferation and heart regeneration. Nature Communications. 9(1). 700–700. 130 indexed citations
16.
Wei, Huijun, Ri Cui, Nicola Zanesi, et al.. (2017). miR-130a Deregulates PTEN and Stimulates Tumor Growth. Cancer Research. 77(22). 6168–6178. 53 indexed citations
17.
Nagaraja, Surya, Nicholas A. Vitanza, Pamelyn J. Woo, et al.. (2017). Transcriptional Dependencies in Diffuse Intrinsic Pontine Glioma. Cancer Cell. 31(5). 635–652.e6. 257 indexed citations
18.
Liu, Jiajing, et al.. (2014). Association study of two inflammation-related polymorphisms with susceptibility to hepatocellular carcinoma: a meta-analysis. BMC Medical Genetics. 15(1). 92–92. 17 indexed citations
19.
Hua, Jing, Zhe Mu, Bright I. Nwaru, et al.. (2014). Child neglect in one-child families from Suzhou City of Mainland China. BMC International Health and Human Rights. 14(1). 8–8. 26 indexed citations
20.
Meng, Wei, Zhenqing Ye, Ri Cui, et al.. (2013). MicroRNA-31 Predicts the Presence of Lymph Node Metastases and Survival in Patients with Lung Adenocarcinoma. Clinical Cancer Research. 19(19). 5423–5433. 93 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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