Wei Huang

6.4k total citations · 2 hit papers
151 papers, 4.9k citations indexed

About

Wei Huang is a scholar working on Molecular Biology, Surgery and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Wei Huang has authored 151 papers receiving a total of 4.9k indexed citations (citations by other indexed papers that have themselves been cited), including 71 papers in Molecular Biology, 39 papers in Surgery and 23 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Wei Huang's work include Tissue Engineering and Regenerative Medicine (27 papers), MicroRNA in disease regulation (13 papers) and Extracellular vesicles in disease (11 papers). Wei Huang is often cited by papers focused on Tissue Engineering and Regenerative Medicine (27 papers), MicroRNA in disease regulation (13 papers) and Extracellular vesicles in disease (11 papers). Wei Huang collaborates with scholars based in China, United States and Canada. Wei Huang's co-authors include Yigang Wang, Guo‐Chang Fan, Tianqing Peng, Xiaohong Wang, Muhammad Ashraf, Yuliang Feng, Xi‐Yong Yu, Kobina Essandoh, Wenfeng Cai and Liwang Yang and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Nature Communications.

In The Last Decade

Wei Huang

144 papers receiving 4.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction

2015 355 citations Xiaohong Wang, Wei Huang et al. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease profile →
Effects of oxygen vacancies on hydrogenation efficiency by spillover in catalysts

2025 25 citations Wei Huang et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Wei Huang China	37	3.3k	1.3k	885	674	536	151	4.9k
Andrea Caporali United Kingdom	39	2.9k 0.9×	1.5k 1.1×	560 0.6×	539 0.8×	283 0.5×	93	5.2k
Yangxin Li China	37	3.2k 1.0×	1.3k 1.0×	685 0.8×	549 0.8×	430 0.8×	89	5.0k
Gaia Spinetti Italy	37	2.0k 0.6×	990 0.7×	741 0.8×	663 1.0×	464 0.9×	96	4.3k
Manfred Boehm United States	34	3.5k 1.1×	1.1k 0.9×	884 1.0×	432 0.6×	274 0.5×	80	6.2k
Carlo Gaetano Italy	50	4.1k 1.3×	1.6k 1.2×	615 0.7×	458 0.7×	344 0.6×	149	6.1k
Tzafra Cohen Israel	25	4.4k 1.4×	1.4k 1.1×	603 0.7×	205 0.3×	283 0.5×	40	6.8k
Mattia Albiero Italy	34	2.8k 0.9×	526 0.4×	773 0.9×	643 1.0×	526 1.0×	85	5.7k
Stefan Liebner Germany	41	3.5k 1.1×	687 0.5×	274 0.3×	272 0.4×	466 0.9×	70	6.8k
Yong‐Jian Geng United States	40	2.4k 0.7×	985 0.7×	1.6k 1.8×	846 1.3×	673 1.3×	104	5.9k
Gangjian Qin United States	46	4.9k 1.5×	1.6k 1.2×	1.7k 1.9×	1.4k 2.1×	1.2k 2.2×	144	7.7k

Countries citing papers authored by Wei Huang

Since Specialization

Citations

This map shows the geographic impact of Wei Huang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Huang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Huang more than expected).

Fields of papers citing papers by Wei Huang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Huang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Huang. The network helps show where Wei Huang may publish in the future.

Co-authorship network of co-authors of Wei Huang

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Huang. A scholar is included among the top collaborators of Wei Huang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Huang. Wei Huang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Zhou, Xuedong, Ying Yang, Daojiang Yu, et al.. (2025). Naringenin boosts Parkin-mediated mitophagy via estrogen receptor alpha to maintain mitochondrial quality control and heal diabetic foot ulcer. Journal of Pharmaceutical Analysis. 15(12). 101333–101333. 1 indexed citations

Liu, Xu, Wei Huang, Jing Chen, et al.. (2024). CIRP increases Foxp3+ regulatory T cells and inhibits development of Th17 cells by enhancing TLR4-IL-2 signaling in the late phase of sepsis. International Immunopharmacology. 132. 111924–111924. 2 indexed citations

He, Xingyu, Lin Chenglu, Karthickeyan Chella Krishnan, et al.. (2024). Current Advances and Future Directions of Pluripotent Stem Cells-Derived Engineered Heart Tissue for Treatment of Cardiovascular Diseases. Cells. 13(24). 2098–2098. 2 indexed citations

Yang, Mingming, et al.. (2023). DEPDC1 and KIF4A synergistically inhibit the malignant biological behavior of osteosarcoma cells through Hippo signaling pathway. Journal of Orthopaedic Surgery and Research. 18(1). 145–145. 8 indexed citations

Huang, Wei, Xiaohua Gao, Sungho Lee, et al.. (2023). Cardiomyocyte Alpha-1A Adrenergic Receptors Mitigate Postinfarct Remodeling and Mortality by Constraining Necroptosis. JACC Basic to Translational Science. 9(1). 78–96. 6 indexed citations

Jiang, Lin, Jialiang Liang, Wei Huang, et al.. (2021). CRISPR activation of endogenous genes reprograms fibroblasts into cardiovascular progenitor cells for myocardial infarction therapy. Molecular Therapy. 30(1). 54–74. 32 indexed citations

Jiang, Lin, Jialiang Liang, Wei Huang, et al.. (2020). Strategies and Challenges to Improve Cellular Programming-Based Approaches for Heart Regeneration Therapy. International Journal of Molecular Sciences. 21(20). 7662–7662. 3 indexed citations

Wang, Cui, et al.. (2020). Association Between Doppler Snuffbox Resistive Index and Tissue Perfusion in Septic Patients. Shock. 54(6). 723–730. 5 indexed citations

Liang, Jialiang, Wei Huang, Lin Jiang, et al.. (2019). Concise Review: Reduction of Adverse Cardiac Scarring Facilitates Pluripotent Stem Cell-Based Therapy for Myocardial Infarction. Stem Cells. 37(7). 844–854. 19 indexed citations

10.

Wang, Changjun, Yidong Zhou, Hanjiang Zhu, et al.. (2018). Would 1.0 cm be a more suitable cutoff to subdivide pT1 tumors in hormone receptor‐negative and HER2‐positive breast cancer?. Cancer Medicine. 7(11). 5420–5430. 1 indexed citations

11.

Ma, Ruilian, Jialiang Liang, Wei Huang, et al.. (2016). Electrical Stimulation Enhances Cardiac Differentiation of Human Induced Pluripotent Stem Cells for Myocardial Infarction Therapy. Antioxidants and Redox Signaling. 28(5). 371–384. 72 indexed citations

12.

Cai, Wenfeng, Kai Kang, Wei Huang, et al.. (2015). CXCR4 attenuates cardiomyocytes mitochondrial dysfunction to resist ischaemia‐reperfusion injury. Journal of Cellular and Molecular Medicine. 19(8). 1825–1835. 16 indexed citations

13.

Huang, Wei, Yuliang Feng, Wenfeng Cai, et al.. (2014). MicroRNA-377 Regulates Mesenchymal Stem Cell-Induced Angiogenesis in Ischemic Hearts by Targeting VEGF. PLoS ONE. 9(9). e104666–e104666. 69 indexed citations

14.

Wang, Yuhua, Wei Huang, Zhili Wen, et al.. (2014). Suicide Gene-Mediated Sequencing Ablation Revealed the Potential Therapeutic Mechanism of Induced Pluripotent Stem Cell-Derived Cardiovascular Cell Patch Post-Myocardial Infarction. Antioxidants and Redox Signaling. 21(16). 2177–2191. 7 indexed citations

15.

Wang, Xiaohong, Wei Huang, Yang� Yang, et al.. (2014). Loss of duplexmiR-223 (5p and 3p) aggravates myocardial depression and mortality in polymicrobial sepsis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1842(5). 701–711. 75 indexed citations

16.

Huang, Wei, Yigang Wang, Zhi‐Xin Shan, et al.. (2013). Fn14 Promotes Differentiation of Human Mesenchymal Stem Cells into Heart Valvular Interstitial Cells by Phenotypic Characterization. Journal of Cellular Physiology. 229(5). 580–587. 17 indexed citations

17.

Gong, Yong, et al.. (2012). Determination of Thiamethoxam Residues in Tomato by High Performance Liquid Chromatography with UV Detection. Research Center for Eco-Environmental Sciences OpenIR (Chinese Academy of Sciences). 2 indexed citations

18.

Ryskamp, Daniel A., Paul Witkovsky, Péter Barabás, et al.. (2011). The Polymodal Ion Channel Transient Receptor Potential Vanilloid 4 Modulates Calcium Flux, Spiking Rate, and Apoptosis of Mouse Retinal Ganglion Cells. Journal of Neuroscience. 31(19). 7089–7101. 180 indexed citations

19.

Monaghan-Benson, Elizabeth, Aleksandr E. Vendrov, Grace Byfield, et al.. (2010). The Role of Vascular Endothelial Growth Factor-Induced Activation of NADPH Oxidase in Choroidal Endothelial Cells and Choroidal Neovascularization. American Journal Of Pathology. 177(4). 2091–2102. 46 indexed citations

20.

Li, Xiao, Yizhi Liu, Ying Tao, et al.. (2003). [Relationship between macrophages and apoptosis in patients with myelodysplastic syndromes].. PubMed. 32(3). 226–9.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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