Wei Bao

1.4k total citations
35 papers, 1.1k citations indexed

About

Wei Bao is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Wei Bao has authored 35 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 12 papers in Cancer Research and 5 papers in Oncology. Recurrent topics in Wei Bao's work include Cancer-related molecular mechanisms research (8 papers), Circular RNAs in diseases (6 papers) and MicroRNA in disease regulation (5 papers). Wei Bao is often cited by papers focused on Cancer-related molecular mechanisms research (8 papers), Circular RNAs in diseases (6 papers) and MicroRNA in disease regulation (5 papers). Wei Bao collaborates with scholars based in China, United Kingdom and South Korea. Wei Bao's co-authors include Xiaoping Wan, Ye Yang, Ping Zhu, Haifeng Qiu, Huihui Wang, Xin Luo, Yanan Wang, Tingting Yang, Sufang Wu and Zixiu Du and has published in prestigious journals such as PLoS ONE, Biomaterials and Small.

In The Last Decade

Wei Bao

34 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei Bao China 18 651 393 197 117 109 35 1.1k
Amy E. Birsner United States 21 644 1.0× 187 0.5× 176 0.9× 177 1.5× 149 1.4× 34 1.2k
Mélanie Mestdagt Belgium 11 568 0.9× 211 0.5× 382 1.9× 159 1.4× 85 0.8× 13 1.0k
Justin Bottsford-Miller United States 16 559 0.9× 324 0.8× 591 3.0× 258 2.2× 93 0.9× 42 1.4k
Tzu‐Hao Wang Taiwan 13 502 0.8× 134 0.3× 145 0.7× 58 0.5× 60 0.6× 16 861
Guangzhi Wang China 18 490 0.8× 314 0.8× 160 0.8× 155 1.3× 32 0.3× 49 909
N. Skuli United States 3 493 0.8× 389 1.0× 136 0.7× 92 0.8× 27 0.2× 6 969
Angela Bentivegna Italy 21 724 1.1× 275 0.7× 243 1.2× 70 0.6× 17 0.2× 57 1.4k
Zhengsheng Wu China 22 970 1.5× 667 1.7× 376 1.9× 174 1.5× 18 0.2× 43 1.5k
Ping Long China 10 586 0.9× 243 0.6× 146 0.7× 82 0.7× 25 0.2× 28 897

Countries citing papers authored by Wei Bao

Since Specialization
Citations

This map shows the geographic impact of Wei Bao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Bao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Bao more than expected).

Fields of papers citing papers by Wei Bao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Bao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Bao. The network helps show where Wei Bao may publish in the future.

Co-authorship network of co-authors of Wei Bao

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Bao. A scholar is included among the top collaborators of Wei Bao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Bao. Wei Bao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wei, Muyun, Luan Chen, Jie Tan, et al.. (2025). Integrative analysis based on CRISPR screen identifies apilimod as a potential therapeutic agent for cisplatin-induced acute kidney injury treatment. Science China Life Sciences. 68(6). 1770–1785. 1 indexed citations
2.
Li, Haosheng, Xiaonan Shen, Kuai Liu, et al.. (2024). A pH-responsive cetuximab-conjugated DMAKO-20 nano-delivery system for overcoming K-ras mutations and drug resistance in colorectal carcinoma. Acta Biomaterialia. 177. 456–471. 7 indexed citations
4.
Wang, Hao, et al.. (2023). Cisplatin-induced PANDAR-Chemo-EVs contribute to a more aggressive and chemoresistant ovarian cancer phenotype through the SRSF9-SIRT4/SIRT6 axis. Journal of Gynecologic Oncology. 35(2). e13–e13. 8 indexed citations
5.
Huang, Li, Shi Zhan, Yu Pan, Wei Bao, & Ye Yang. (2023). The role of Vps4 in cancer development. Frontiers in Oncology. 13. 1203359–1203359. 8 indexed citations
6.
Chen, Juan, Kuai Liu, Wei Wei, et al.. (2023). Precision Therapy of Recurrent Breast Cancer through Targeting Different Malignant Tumor Cells with a HER2/CD44‐Targeted Hydrogel Nanobot. Small. 19(37). e2301043–e2301043. 14 indexed citations
7.
8.
Yang, Ye, et al.. (2020). Hydrogen inhibits endometrial cancer growth via a ROS/NLRP3/caspase-1/GSDMD-mediated pyroptotic pathway. BMC Cancer. 20(1). 28–28. 154 indexed citations
9.
Peng, Jiahui, Juan Chen, Fang Xie, et al.. (2019). Herceptin-conjugated paclitaxel loaded PCL-PEG worm-like nanocrystal micelles for the combinatorial treatment of HER2-positive breast cancer. Biomaterials. 222. 119420–119420. 96 indexed citations
10.
Zhuang, Yan, Jiangdong Xiang, Wei Bao, et al.. (2017). MDH2 Stimulated by Estrogen-GPR30 Pathway Down-Regulated PTEN Expression Promoting the Proliferation and Invasion of Cells in Endometrial Cancer. Translational Oncology. 10(2). 203–210. 15 indexed citations
11.
Wang, Yanan, Shiyu Wang, Yuan Liu, et al.. (2017). LSD1 sustains estrogen-driven endometrial carcinoma cell proliferation through the PI3K/AKT pathway via di-demethylating H3K9 of cyclin D1. International Journal of Oncology. 50(3). 942–952. 24 indexed citations
12.
Zhang, Yongli, et al.. (2016). SOX17 increases the cisplatin sensitivity of an endometrial cancer cell line. Cancer Cell International. 16(1). 29–29. 15 indexed citations
13.
Bao, Wei, Jingyun Wang, Tingting Yang, et al.. (2014). FOXA1 promotes tumor cell proliferation through AR involving the Notch pathway in endometrial cancer. BMC Cancer. 14(1). 78–78. 61 indexed citations
14.
Liu, Binya, Qi Che, Haifeng Qiu, et al.. (2014). Elevated MiR-222-3p Promotes Proliferation and Invasion of Endometrial Carcinoma via Targeting ERα. PLoS ONE. 9(1). e87563–e87563. 46 indexed citations
15.
Bao, Wei, et al.. (2014). Downregulation of p16ink4a inhibits cell proliferation and induces G1 cell cycle arrest in cervical cancer cells. International Journal of Molecular Medicine. 33(6). 1577–1585. 18 indexed citations
16.
Wang, Jingyun, Wei Bao, Yun Liao, et al.. (2014). Forkhead-box A1 suppresses the progression of endometrial cancer via crosstalk with estrogen receptor α. Oncology Reports. 31(3). 1225–1234. 22 indexed citations
17.
Yang, Tingting, Haifeng Qiu, Wei Bao, et al.. (2013). Epigenetic Inactivation of EFEMP1 Is Associated with Tumor Suppressive Function in Endometrial Carcinoma. PLoS ONE. 8(6). e67458–e67458. 33 indexed citations
18.
Bao, Wei, et al.. (2013). A TrkB–STAT3–miR-204-5p regulatory circuitry controls proliferation and invasion of endometrial carcinoma cells. Molecular Cancer. 12(1). 155–155. 115 indexed citations
19.
Bao, Wei, Haifeng Qiu, Tingting Yang, et al.. (2013). Upregulation of TrkB Promotes Epithelial-Mesenchymal Transition and Anoikis Resistance in Endometrial Carcinoma. PLoS ONE. 8(7). e70616–e70616. 67 indexed citations
20.
Zhou, Long, et al.. (2011). Crosstalk Between Estrogen Receptor and Mitogen-Activated Protein Kinase Signaling in the Development and Progression of Endometrial Cancer. International Journal of Gynecological Cancer. 21(8). 1357–1365. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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