W. E. C. Bradley

1.8k total citations
54 papers, 1.3k citations indexed

About

W. E. C. Bradley is a scholar working on Molecular Biology, Genetics and Oncology. According to data from OpenAlex, W. E. C. Bradley has authored 54 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Molecular Biology, 18 papers in Genetics and 8 papers in Oncology. Recurrent topics in W. E. C. Bradley's work include CRISPR and Genetic Engineering (11 papers), Biochemical and Molecular Research (8 papers) and Virus-based gene therapy research (7 papers). W. E. C. Bradley is often cited by papers focused on CRISPR and Genetic Engineering (11 papers), Biochemical and Molecular Research (8 papers) and Virus-based gene therapy research (7 papers). W. E. C. Bradley collaborates with scholars based in Canada, United States and Ireland. W. E. C. Bradley's co-authors include Cécile Rochette‐Egly, Karen Messing, Lloyd A. Culp, J Bérard, Benoit Houle, Ana María Seifert, Abdelmajid Belouchi, Stephen J. Usala, J B Menke and S. Massé and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Clinical Oncology.

In The Last Decade

W. E. C. Bradley

53 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
W. E. C. Bradley Canada 21 1.0k 497 210 173 168 54 1.3k
Françoise Capony France 21 588 0.6× 371 0.7× 501 2.4× 128 0.7× 291 1.7× 31 1.4k
Xiangyuan Wang United States 24 1.5k 1.4× 553 1.1× 131 0.6× 75 0.4× 174 1.0× 43 2.0k
Mattia Frontini United Kingdom 24 1.0k 1.0× 228 0.5× 193 0.9× 24 0.1× 176 1.0× 42 1.5k
W. N. Scott United Kingdom 18 431 0.4× 190 0.4× 151 0.7× 142 0.8× 280 1.7× 42 1.1k
Sotokichi Morii Japan 14 289 0.3× 233 0.5× 166 0.8× 147 0.8× 203 1.2× 90 1.1k
Margarethe Litfin Germany 12 910 0.9× 161 0.3× 260 1.2× 33 0.2× 237 1.4× 17 1.3k
Todd W. Sandhoff United States 11 1.1k 1.1× 333 0.7× 77 0.4× 151 0.9× 227 1.4× 11 1.5k
Ludivine Dion France 18 459 0.5× 197 0.4× 54 0.3× 39 0.2× 119 0.7× 55 804
A.M. de Leeuw Netherlands 17 581 0.6× 142 0.3× 54 0.3× 35 0.2× 210 1.3× 23 1.5k
Caroline Berthier France 12 991 1.0× 94 0.2× 70 0.3× 47 0.3× 126 0.8× 19 1.2k

Countries citing papers authored by W. E. C. Bradley

Since Specialization
Citations

This map shows the geographic impact of W. E. C. Bradley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. E. C. Bradley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. E. C. Bradley more than expected).

Fields of papers citing papers by W. E. C. Bradley

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. E. C. Bradley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. E. C. Bradley. The network helps show where W. E. C. Bradley may publish in the future.

Co-authorship network of co-authors of W. E. C. Bradley

This figure shows the co-authorship network connecting the top 25 collaborators of W. E. C. Bradley. A scholar is included among the top collaborators of W. E. C. Bradley based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. E. C. Bradley. W. E. C. Bradley is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bradley, W. E. C., et al.. (2010). More is less: Inactivation and deletion events and the search for tumor suppressor genes. Journal of Cellular Biochemistry. 110(2). 281–287. 2 indexed citations
2.
Bradley, W. E. C., John Raelson, Hélène Fournier, et al.. (2010). Hotspots of Large Rare Deletions in the Human Genome. PLoS ONE. 5(2). e9401–e9401. 43 indexed citations
3.
Murakami, E, Aravind Basavapathruni, W. E. C. Bradley, & Kenneth C. Anderson. (2005). Mechanism of action of a novel viral mutagenic covert nucleotide: molecular interactions with HIV-1 reverse transcriptase and host cell DNA polymerases. Antiviral Research. 67(1). 10–17. 27 indexed citations
4.
Bérard, J, et al.. (1997). Compromised allograft rejection response in transgenic mice expressing antisense sequences to retinoic acid receptor beta2. The Journal of Immunology. 159(2). 623–634. 4 indexed citations
5.
Toulouse, André, et al.. (1996). Structure of the human retinoic acid receptor β1 gene. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1309(1-2). 1–4. 21 indexed citations
6.
Belouchi, Abdelmajid & W. E. C. Bradley. (1992). A mutational hotspot in the aprt gene of Chinese hamster cells. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 266(2). 221–230. 4 indexed citations
7.
Bradley, W. E. C., et al.. (1991). Phenotype reversal in induced mutants of CHO cells: Analysis of the reversed cell lines. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 249(1). 135–145. 1 indexed citations
8.
Belouchi, Abdelmajid & W. E. C. Bradley. (1991). Analysis of second-step mutations of class II and class III CHOaprt heterozygotes: Chromosomal differences in deletion frequencies. Somatic Cell and Molecular Genetics. 17(3). 277–286. 5 indexed citations
9.
Bradley, W. E. C., et al.. (1991). A very large spontaneous deletion ataprt locus in CHO cells: Sequence similarities with smallaprt deletions. Somatic Cell and Molecular Genetics. 17(1). 57–68. 13 indexed citations
10.
Houle, Benoit, et al.. (1991). Implication of RARB in Epidermoid (Squamous) Lung Cancer. Genes Chromosomes and Cancer. 3(5). 358–366. 68 indexed citations
11.
Usala, Stephen J., J B Menke, J Bérard, et al.. (1991). A New Point Mutation in the 3,5,3′-Triiodothyronine-Binding Domain of the c-erbAβThyroid Hormone Receptor Is Tightly Linked to Generalized Thyroid Hormone Resistance. The Journal of Clinical Endocrinology & Metabolism. 72(1). 32–38. 58 indexed citations
12.
Brauch, Hiltrud, Farida Latif, Stephen B. Baylin, et al.. (1990). Mspl and Dral Polymorphisms at the ERBA beta locus on chromosome 3p. Nucleic Acids Research. 18(9). 2833–2833. 1 indexed citations
13.
Bradley, W. E. C., et al.. (1990). Loss of alleles inaprt mutants of CHO cells demonstrated by BclI restriction-fragment-length variation. Somatic Cell and Molecular Genetics. 16(3). 225–230. 5 indexed citations
14.
Bradley, W. E. C., et al.. (1989). High-frequency deletion event ataprt locus of CHO cells: Detection and characterization of endpoints. Somatic Cell and Molecular Genetics. 15(1). 19–28. 24 indexed citations
15.
Dobrovic, Alexander, et al.. (1988). DNA methylation and genetic inactivation at thymidine kinase locus: Two different mechanisms for silencing autosomal genes. Somatic Cell and Molecular Genetics. 14(1). 55–68. 19 indexed citations
16.
Bradley, W. E. C., Abdelmajid Belouchi, & Karen Messing. (1988). The aprt heterozygote/hemizygote system for screening mutagenic agents allows detection of large deletions. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 199(1). 131–138. 34 indexed citations
17.
Messing, Karen & W. E. C. Bradley. (1985). In vivo mutant frequency rises among breast cancer patients after exposure to high doses of γ-radiation. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 152(1). 107–112. 69 indexed citations
18.
Simon, Anne E., Milton W. Taylor, & W. E. C. Bradley. (1983). Mechanism of Mutation at the aprt Locus in Chinese Hamster Ovary Cells: Analysis of Heterozygotes and Hemizygotes. Molecular and Cellular Biology. 3(10). 1703–1710. 1 indexed citations
19.
Bradley, W. E. C.. (1983). Mutation at Autosomal Loci of Chinese Hamster Ovary Cells: Involvement of a High-Frequency Event Silencing Two Linked Alleles. Molecular and Cellular Biology. 3(7). 1172–1181. 6 indexed citations
20.
Bradley, W. E. C., et al.. (1982). Bromodeoxyuridine resistance in CHO cells occurs in three discrete steps. Somatic Cell and Molecular Genetics. 8(2). 207–222. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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