Vjekoslav Dulić

9.7k total citations · 5 hit papers
41 papers, 7.7k citations indexed

About

Vjekoslav Dulić is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Vjekoslav Dulić has authored 41 papers receiving a total of 7.7k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Molecular Biology, 24 papers in Oncology and 12 papers in Cell Biology. Recurrent topics in Vjekoslav Dulić's work include Cancer-related Molecular Pathways (23 papers), Telomeres, Telomerase, and Senescence (10 papers) and DNA Repair Mechanisms (9 papers). Vjekoslav Dulić is often cited by papers focused on Cancer-related Molecular Pathways (23 papers), Telomeres, Telomerase, and Senescence (10 papers) and DNA Repair Mechanisms (9 papers). Vjekoslav Dulić collaborates with scholars based in France, United States and Switzerland. Vjekoslav Dulić's co-authors include Steven I. Reed, Emma Lees, Gretchen H. Stein, Daniel J. Lew, Shawna C. O. Reed, Linda F. Drullinger, Véronique Gire, Alexandre Soulard, J. Wade Harper and Stephen J. Elledge and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Vjekoslav Dulić

41 papers receiving 7.6k citations

Hit Papers

p53-dependent inhibition of cyclin-dependent kinase activ... 1991 2026 2002 2014 1994 1992 1992 1991 1999 400 800 1.2k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Vjekoslav Dulić France 28 5.5k 4.2k 1.4k 1.1k 886 41 7.7k
Asha S. Multani United States 40 5.0k 0.9× 3.3k 0.8× 677 0.5× 1.4k 1.2× 1.9k 2.1× 126 8.2k
Hiroaki Kiyokawa United States 47 5.5k 1.0× 3.5k 0.8× 1.2k 0.8× 404 0.4× 958 1.1× 117 7.8k
Sam W. Lee United States 48 5.0k 0.9× 2.3k 0.6× 906 0.6× 671 0.6× 1.1k 1.2× 93 7.1k
Mila E. McCurrach United States 20 7.3k 1.3× 3.6k 0.8× 658 0.5× 2.1k 1.8× 1.3k 1.5× 21 9.6k
William K. Kaufmann United States 44 5.0k 0.9× 2.8k 0.7× 898 0.6× 452 0.4× 1.6k 1.8× 161 7.1k
Katsuyuki Tamai Japan 38 9.9k 1.8× 5.2k 1.2× 2.0k 1.4× 538 0.5× 2.0k 2.3× 96 12.4k
Samuel Benchimol Canada 46 5.7k 1.0× 4.6k 1.1× 447 0.3× 1.3k 1.1× 1.4k 1.5× 94 8.8k
Jason D. Weber United States 39 6.2k 1.1× 3.9k 0.9× 688 0.5× 517 0.5× 1.4k 1.6× 78 8.9k
Stephen N. Jones United States 50 10.6k 1.9× 5.9k 1.4× 1.1k 0.8× 838 0.7× 2.4k 2.7× 114 13.2k
Athena W. Lin United States 15 6.2k 1.1× 2.9k 0.7× 626 0.4× 3.2k 2.8× 1.2k 1.4× 27 9.0k

Countries citing papers authored by Vjekoslav Dulić

Since Specialization
Citations

This map shows the geographic impact of Vjekoslav Dulić's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vjekoslav Dulić with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vjekoslav Dulić more than expected).

Fields of papers citing papers by Vjekoslav Dulić

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Vjekoslav Dulić. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vjekoslav Dulić. The network helps show where Vjekoslav Dulić may publish in the future.

Co-authorship network of co-authors of Vjekoslav Dulić

This figure shows the co-authorship network connecting the top 25 collaborators of Vjekoslav Dulić. A scholar is included among the top collaborators of Vjekoslav Dulić based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Vjekoslav Dulić. Vjekoslav Dulić is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lossaint, Gérald, Véronique Gire, Karim Mrouj, et al.. (2022). Reciprocal regulation of p21 and Chk1 controls the cyclin D1-RB pathway to mediate senescence onset after G2 arrest. Journal of Cell Science. 135(8). 26 indexed citations
2.
Sobecki, Michal, Karim Mrouj, Jacques Colinge, et al.. (2017). Cell-Cycle Regulation Accounts for Variability in Ki-67 Expression Levels. Cancer Research. 77(10). 2722–2734. 265 indexed citations
3.
Wang, Jingkui, Daniel Mauvoisin, Eva Martín, et al.. (2016). Nuclear Proteomics Uncovers Diurnal Regulatory Landscapes in Mouse Liver. Cell Metabolism. 25(1). 102–117. 146 indexed citations
4.
Gire, Véronique & Vjekoslav Dulić. (2015). Senescence from G2 arrest, revisited. Cell Cycle. 14(3). 297–304. 216 indexed citations
5.
Brondello, Jean‐Marc, Alexandre Prieur, Didier Philipot, et al.. (2012). La sénescence cellulaire. médecine/sciences. 28(3). 288–296. 9 indexed citations
6.
Dulić, Vjekoslav. (2012). Senescence Regulation by mTOR. Methods in molecular biology. 965. 15–35. 32 indexed citations
7.
Chassot, Anne‐Amandine, Laurent Turchi, Thierry Virolle, et al.. (2007). Id3 is a novel regulator of p27kip1 mRNA in early G1 phase and is required for cell-cycle progression. Oncogene. 26(39). 5772–5783. 26 indexed citations
8.
Reed, Steven I., Vjekoslav Dulić, Daniel J. Lew, Helena E. Richardson, & Curt Wittenberg. (2007). G1 Control in Yeast and Animal Cells. Novartis Foundation symposium. 170. 7–19. 2 indexed citations
9.
Dupuy, Jérôme, et al.. (2006). Structural Basis for the Modulation of CDK-Dependent/Independent Activity of Cyclin D1. Cell Cycle. 5(23). 2760–2768. 12 indexed citations
10.
Gire, Véronique, Pierre Roux, David Wynford‐Thomas, Jean‐Marc Brondello, & Vjekoslav Dulić. (2004). DNA damage checkpoint kinase Chk2 triggers replicative senescence. The EMBO Journal. 23(13). 2554–2563. 143 indexed citations
11.
Peter, Marion, Laura Magnaghi-Jaulin, Anna Castro, et al.. (2001). Quand la dynamique chromosomique contrôle la division cellulaire. Pathologie Biologie. 49(8). 649–654. 1 indexed citations
12.
Dulić, Vjekoslav, et al.. (2000). Uncoupling between Phenotypic Senescence and Cell Cycle Arrest in Aging p21-Deficient Fibroblasts. Molecular and Cellular Biology. 20(18). 6741–6754. 106 indexed citations
13.
Sevilla, Lídia, Christel Aperlo, Vjekoslav Dulić, et al.. (1999). The Ets2 Transcription Factor Inhibits Apoptosis Induced by Colony-Stimulating Factor 1 Deprivation of Macrophages through a Bcl-x L -Dependent Mechanism. Molecular and Cellular Biology. 19(4). 2624–2634. 87 indexed citations
14.
Dulić, Vjekoslav, Gretchen H. Stein, Dariush Farahi Far, & Steven I. Reed. (1998). Nuclear Accumulation of p21 Cip1 at the Onset of Mitosis: a Role at the G 2 /M-Phase Transition. Molecular and Cellular Biology. 18(1). 546–557. 319 indexed citations
15.
Stein, Gretchen H. & Vjekoslav Dulić. (1995). Origins of G1 arrest in senescent human fibroblasts. BioEssays. 17(6). 537–543. 116 indexed citations
16.
Dulić, Vjekoslav, et al.. (1993). Altered regulation of G1 cyclins in senescent human diploid fibroblasts: accumulation of inactive cyclin E-Cdk2 and cyclin D1-Cdk2 complexes.. Proceedings of the National Academy of Sciences. 90(23). 11034–11038. 254 indexed citations
17.
Lees, Emma, et al.. (1992). Cyclin E/cdk2 and cyclin A/cdk2 kinases associate with p107 and E2F in a temporally distinct manner.. Genes & Development. 6(10). 1874–1885. 396 indexed citations
18.
Lew, Daniel J., Vjekoslav Dulić, & Steven I. Reed. (1991). Isolation of three novel human cyclins by rescue of G1 cyclin (cln) function in yeast. Cell. 66(6). 1197–1206. 741 indexed citations breakdown →
19.
Dulić, Vjekoslav, et al.. (1991). [48] Yeast endocytosis assays. Methods in enzymology on CD-ROM/Methods in enzymology. 194. 697–710. 188 indexed citations
20.
Reed, Steven I., Curt Wittenberg, Daniel J. Lew, Vjekoslav Dulić, & Martha Henze. (1991). G1 Control in Yeast and Animal Cells. Cold Spring Harbor Symposia on Quantitative Biology. 56(0). 61–67. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026