Vjekoslav Dulić
- Molecular Biology top 1%
- Oncology top 0.5%
- Cell Biology top 0.5%
- Physiology top 2%
- Cancer Research top 2%
- Co-authors
- Steven I. ReedEmma LeesGretchen H. SteinDaniel J. LewShawna C. O. ReedLinda F. DrullingerAlexandre SoulardVéronique Gire
- Topics
- Cancer-related Molecular Pathways (23 papers)Telomeres, Telomerase, and Senescence (10 papers)DNA Repair Mechanisms (9 papers)
- Cited by
- OncologyAgingCell Biology
- Partner nations
- FranceUnited StatesSwitzerland
In The Last Decade
Vjekoslav Dulić
41 papers receiving 7.6k citations
Hit Papers
Peers
Comparison fields: 5 of 127
- Molecular Biology 5.5k
- Oncology 4.2k
- Cell Biology 1.4k
- Physiology 1.1k
- Cancer Research 886
Countries citing papers authored by Vjekoslav Dulić
This map shows the geographic impact of Vjekoslav Dulić's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vjekoslav Dulić with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vjekoslav Dulić more than expected).
Fields of papers citing papers by Vjekoslav Dulić
This network shows the impact of papers produced by Vjekoslav Dulić. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vjekoslav Dulić. The network helps show where Vjekoslav Dulić may publish in the future.
Co-authorship network of co-authors of Vjekoslav Dulić
This figure shows the co-authorship network connecting the top 25 collaborators of Vjekoslav Dulić. A scholar is included among the top collaborators of Vjekoslav Dulić based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Vjekoslav Dulić. Vjekoslav Dulić is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 265 | |
| 2 | 63 | |
| 3 | 146 | |
| 4 | 9 | |
| 5 | 32 | |
| 6 | 62 | |
| 7 | 27 | |
| 8 | 26 | |
| 9 | 2 | |
| 10 | 12 | |
| 11 | 143 | |
| 12 | 1 | |
| 13 | 87 | |
| 14 | 319 | |
| 15 | 32 | |
| 16 | 116 | |
| 17 | p53-dependent inhibition of cyclin-dependent kinase activities in human fibroblasts during radiation-induced G1 arrestbreakdown → | 1282 |
| 18 | 188 | |
| 19 | Isolation of three novel human cyclins by rescue of G1 cyclin (cln) function in yeastbreakdown → | 741 |
| 20 | 17 |
About Vjekoslav Dulić
Vjekoslav Dulić is a scholar working on Oncology, Cell Biology and Molecular Biology, having authored 41 papers that have together received 7.7k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (23 papers), Telomeres, Telomerase, and Senescence (10 papers) and DNA Repair Mechanisms (9 papers). The work is most often cited by research in Oncology (4.2k citations), Aging (214 citations) and Cell Biology (1.4k citations). Vjekoslav Dulić has collaborated with scholars based in France, United States and Switzerland. Frequent co-authors include Steven I. Reed, Emma Lees, Gretchen H. Stein, Daniel J. Lew, Shawna C. O. Reed, Linda F. Drullinger, Alexandre Soulard, Véronique Gire, J. Wade Harper and Stephen J. Elledge. Their work appears in journals such as Science, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.