Uwe Thiel

870 total citations
25 papers, 431 citations indexed

About

Uwe Thiel is a scholar working on Oncology, Immunology and Genetics. According to data from OpenAlex, Uwe Thiel has authored 25 papers receiving a total of 431 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Oncology, 15 papers in Immunology and 5 papers in Genetics. Recurrent topics in Uwe Thiel's work include CAR-T cell therapy research (16 papers), Immunotherapy and Immune Responses (11 papers) and Immune Cell Function and Interaction (7 papers). Uwe Thiel is often cited by papers focused on CAR-T cell therapy research (16 papers), Immunotherapy and Immune Responses (11 papers) and Immune Cell Function and Interaction (7 papers). Uwe Thiel collaborates with scholars based in Germany, Austria and Canada. Uwe Thiel's co-authors include Stefan Burdach, Günther Richter, Thomas G. P. Grünewald, Dirk H. Busch, Melanie Thiede, Olivia Prazeres da Costa, Rebeca Alba Rubío, Irene von Luettichau, Sebastian Johannes Schober and A. Kirschner and has published in prestigious journals such as Cancer Research, Clinical Cancer Research and European Heart Journal.

In The Last Decade

Uwe Thiel

24 papers receiving 426 citations

Peers

Uwe Thiel
Alessandro Zaccagna Italy
Johannes Andel Austria
Yuji Hirami Japan
Ulla Vanleeuw Belgium
Matthias Beyens Belgium
Maciej Kaczorowski Poland
Nausicaa Malissen France
B. Le Mevel France
Daniel Pinďák Slovakia
Sasan Ghaffari Iran
Alessandro Zaccagna Italy
Uwe Thiel
Citations per year, relative to Uwe Thiel Uwe Thiel (= 1×) peers Alessandro Zaccagna

Countries citing papers authored by Uwe Thiel

Since Specialization
Citations

This map shows the geographic impact of Uwe Thiel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Uwe Thiel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Uwe Thiel more than expected).

Fields of papers citing papers by Uwe Thiel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Uwe Thiel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Uwe Thiel. The network helps show where Uwe Thiel may publish in the future.

Co-authorship network of co-authors of Uwe Thiel

This figure shows the co-authorship network connecting the top 25 collaborators of Uwe Thiel. A scholar is included among the top collaborators of Uwe Thiel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Uwe Thiel. Uwe Thiel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Thiede, Melanie, Leopold Flohé, Valentina Evdokimova, et al.. (2024). Cytokine screening identifies TNF to potentially enhance immunogenicity of pediatric sarcomas. Frontiers in Immunology. 15. 1347404–1347404. 1 indexed citations
2.
Schober, Sebastian Johannes, Melanie Thiede, Julia Hauer, et al.. (2024). TCR-transgenic T cells and YB-1-based oncolytic virotherapy improve survival in a preclinical Ewing sarcoma xenograft mouse model. Frontiers in Immunology. 15. 1330868–1330868. 3 indexed citations
3.
4.
Schober, Sebastian Johannes, Melanie Thiede, Julia Hauer, et al.. (2023). The Oncolytic Adenovirus XVir-N-31 Joins Forces with CDK4/6 Inhibition Augmenting Innate and Adaptive Antitumor Immunity in Ewing Sarcoma. Clinical Cancer Research. 29(10). 1996–2011. 14 indexed citations
5.
Poorebrahim, Mansour, Melanie Thiede, Kilian Schober, et al.. (2022). T Cells Directed against the Metastatic Driver Chondromodulin-1 in Ewing Sarcoma: Comparative Engineering with CRISPR/Cas9 vs. Retroviral Gene Transfer for Adoptive Transfer. Cancers. 14(22). 5485–5485. 1 indexed citations
6.
Schober, Sebastian Johannes, et al.. (2021). Monocyte Maturation Mediators Upregulate CD83, ICAM-1 and MHC Class 1 Expression on Ewing’s Sarcoma, Enhancing T Cell Cytotoxicity. Cells. 10(11). 3070–3070. 5 indexed citations
7.
Evdokimova, Valentina, Peter Ruzanov, Sebastian Johannes Schober, et al.. (2021). Ewing Sarcoma-Derived Extracellular Vesicles Impair Dendritic Cell Maturation and Function. Cells. 10(8). 2081–2081. 22 indexed citations
8.
Schober, Sebastian Johannes, Melanie Thiede, Dirk Wohlleber, et al.. (2020). MHC Class I-Restricted TCR-Transgenic CD4+ T Cells Against STEAP1 Mediate Local Tumor Control of Ewing Sarcoma In Vivo. Cells. 9(7). 1581–1581. 20 indexed citations
9.
Kessel, Kerstin A., et al.. (2020). Prospective evaluation of multitarget treatment of pediatric patients with helical intensity-modulated radiotherapy. Strahlentherapie und Onkologie. 196(12). 1103–1115. 3 indexed citations
10.
Schober, Sebastian Johannes, Melanie Thiede, Dirk Wohlleber, et al.. (2018). Abstract LB-106: Allorepertoire-derived HLA class I/peptide-specific T cell receptor transgenic CD4+ T cells mediate antitumor responses in Ewing sarcoma mimicking allo-rejection. Cancer Research. 78(13_Supplement). LB–106. 1 indexed citations
11.
Kirschner, A., Melanie Thiede, Thomas G. P. Grünewald, et al.. (2017). Pappalysin-1 T cell receptor transgenic allo-restricted T cells kill Ewing sarcomain vitroandin vivo. OncoImmunology. 6(2). e1273301–e1273301. 28 indexed citations
12.
Grünewald, Thomas G. P., Richard Klar, Dirk Wohlleber, et al.. (2016). Transgenic antigen-specific, HLA-A*02:01-allo-restricted cytotoxic T cells recognize tumor-associated target antigen STEAP1 with high specificity. OncoImmunology. 5(6). e1175795–e1175795. 23 indexed citations
13.
Thiel, Uwe, Franziska Blaeschke, Günther Richter, & Stefan Burdach. (2016). Human HLA-A*02:01/CHM1+ allo-restricted T cell receptor-transgenic CD8+ T cells specifically inhibit Ewing sarcoma growth in vitro and in vivo. European Journal of Cancer. 69. S51–S51. 3 indexed citations
14.
Blaeschke, Franziska, Uwe Thiel, A. Kirschner, et al.. (2016). Human HLA-A*02:01/CHM1+ allo-restricted T cell receptor transgenic CD8+ T Cells specifically inhibit Ewing sarcoma growthin vitroandin vivo. Oncotarget. 7(28). 43267–43280. 21 indexed citations
15.
Raposo, Graça, Ulrich Welsch, Olivia Prazeres da Costa, et al.. (2013). First identification of Ewing's sarcoma‐derived extracellular vesicles and exploration of their biological and potential diagnostic implications. Biology of the Cell. 105(7). 289–303. 52 indexed citations
16.
Grünewald, Thomas G. P., Isabel Diebold, Iréne Esposito, et al.. (2011). STEAP1 Is Associated with the Invasive and Oxidative Stress Phenotype of Ewing Tumors. Molecular Cancer Research. 10(1). 52–65. 94 indexed citations
17.
Thiel, Uwe, Heinke Conrad, Dirk H. Busch, et al.. (2011). Specific recognition and inhibition of Ewing tumour growth by antigen-specific allo-restricted cytotoxic T cells. British Journal of Cancer. 104(6). 948–956. 29 indexed citations
18.
19.
Hanrath, Peter, D Mathey, Uwe Thiel, et al.. (1981). Myocardial Thallium201imaging in hypertrophic obstructive cardiomyopathy. European Heart Journal. 2(3). 177–185. 31 indexed citations
20.
Hanrath, Peter, et al.. (1980). [Correlation between myocardial Thallium-201 kinetics, myocardial lactate metabolism and coronary angiographic findings in hypertrophic cardiomyopathy (author's transl)].. PubMed. 69(5). 353–9. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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