Uddin Md. Nazim

631 total citations
25 papers, 520 citations indexed

About

Uddin Md. Nazim is a scholar working on Molecular Biology, Epidemiology and Cancer Research. According to data from OpenAlex, Uddin Md. Nazim has authored 25 papers receiving a total of 520 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 12 papers in Epidemiology and 4 papers in Cancer Research. Recurrent topics in Uddin Md. Nazim's work include Autophagy in Disease and Therapy (12 papers), Cell death mechanisms and regulation (8 papers) and RNA Interference and Gene Delivery (5 papers). Uddin Md. Nazim is often cited by papers focused on Autophagy in Disease and Therapy (12 papers), Cell death mechanisms and regulation (8 papers) and RNA Interference and Gene Delivery (5 papers). Uddin Md. Nazim collaborates with scholars based in South Korea, United Kingdom and Egypt. Uddin Md. Nazim's co-authors include Sang‐Youel Park, Sang‐Youel Park, Ji‐Hong Moon, Jae‐Won Seol, Ju‐Hee Lee, You-Jin Lee, Seong Kug Eo, John-Hwa Lee, Jae‐Kyo Jeong and Sung-Wook Kim and has published in prestigious journals such as Journal of Pharmaceutical Sciences, The International Journal of Biochemistry & Cell Biology and Oncotarget.

In The Last Decade

Uddin Md. Nazim

25 papers receiving 515 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Uddin Md. Nazim South Korea 15 293 167 94 62 59 25 520
Sarbari Saha India 7 326 1.1× 278 1.7× 78 0.8× 42 0.7× 42 0.7× 11 616
Jung Yoon Jang South Korea 15 263 0.9× 87 0.5× 64 0.7× 86 1.4× 139 2.4× 22 571
Danqing Xu China 13 305 1.0× 174 1.0× 39 0.4× 46 0.7× 73 1.2× 16 515
Jianyong Xiao China 15 370 1.3× 98 0.6× 114 1.2× 91 1.5× 41 0.7× 26 560
Kyuhwa Seo South Korea 12 283 1.0× 87 0.5× 55 0.6× 29 0.5× 30 0.5× 13 544
Tiejian Zhao China 8 251 0.9× 192 1.1× 76 0.8× 49 0.8× 26 0.4× 17 511
Maribel Escoll Spain 10 519 1.8× 135 0.8× 84 0.9× 93 1.5× 29 0.5× 15 775
Meng-Wei Lin Taiwan 12 374 1.3× 123 0.7× 77 0.8× 66 1.1× 103 1.7× 12 650
Jingchun Gao China 13 267 0.9× 81 0.5× 52 0.6× 60 1.0× 53 0.9× 29 549
Seungho Choi South Korea 16 325 1.1× 92 0.6× 95 1.0× 65 1.0× 26 0.4× 26 600

Countries citing papers authored by Uddin Md. Nazim

Since Specialization
Citations

This map shows the geographic impact of Uddin Md. Nazim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Uddin Md. Nazim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Uddin Md. Nazim more than expected).

Fields of papers citing papers by Uddin Md. Nazim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Uddin Md. Nazim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Uddin Md. Nazim. The network helps show where Uddin Md. Nazim may publish in the future.

Co-authorship network of co-authors of Uddin Md. Nazim

This figure shows the co-authorship network connecting the top 25 collaborators of Uddin Md. Nazim. A scholar is included among the top collaborators of Uddin Md. Nazim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Uddin Md. Nazim. Uddin Md. Nazim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bishayee, Kausik, Uddin Md. Nazim, Vijay Kumar, et al.. (2022). Reversing the HDAC-inhibitor mediated metabolic escape in MYCN-amplified neuroblastoma. Biomedicine & Pharmacotherapy. 150. 113032–113032. 12 indexed citations
2.
Bishayee, Kausik, et al.. (2022). RNA binding protein HuD promotes autophagy and tumor stress survival by suppressing mTORC1 activity and augmenting ARL6IP1 levels. Journal of Experimental & Clinical Cancer Research. 41(1). 18–18. 12 indexed citations
3.
Nazim, Uddin Md., et al.. (2022). mTORC1-Inhibition Potentiating Metabolic Block by Tyrosine Kinase Inhibitor Ponatinib in Multiple Myeloma. Cancers. 14(11). 2766–2766. 5 indexed citations
4.
Nazim, Uddin Md., et al.. (2021). Selective Targeting of the Novel CK-10 Nanoparticles to the MDA-MB-231 Breast Cancer Cells. Journal of Pharmaceutical Sciences. 111(4). 1197–1207. 6 indexed citations
5.
Nazim, Uddin Md., et al.. (2021). Physicochemical characterization of Novel Particulate Delivery Systems for Antitumor/metastatic Therapeutics. Research Journal of Pharmacy and Technology. 4837–4844. 1 indexed citations
6.
Nazim, Uddin Md., et al.. (2021). Optimization of PLGA nanoparticles for delivery of Novel anticancer CK-10 peptide. Research Journal of Pharmacy and Technology. 5371–5379. 2 indexed citations
7.
Nazim, Uddin Md., et al.. (2020). Downregulation of c‑FLIP and upregulation of DR‑5 by cantharidin sensitizes TRAIL‑mediated apoptosis in prostate cancer cells via autophagy flux. International Journal of Molecular Medicine. 46(1). 280–288. 25 indexed citations
8.
Moon, Ji‐Hong, et al.. (2018). Telmisartan generates ROS-dependent upregulation of death receptor 5 to sensitize TRAIL in lung cancer via inhibition of autophagy flux. The International Journal of Biochemistry & Cell Biology. 102. 20–30. 30 indexed citations
9.
Nazim, Uddin Md. & Sang‐Youel Park. (2018). Luteolin sensitizes human liver cancer cells to TRAIL‑induced apoptosis via autophagy and JNK‑mediated death receptor 5 upregulation. International Journal of Oncology. 54(2). 665–672. 46 indexed citations
10.
Nazim, Uddin Md. & Sang‐Youel Park. (2018). Attenuation of autophagy flux by 6-shogaol sensitizes human liver cancer cells to TRAIL-induced apoptosis via p53 and ROS. International Journal of Molecular Medicine. 43(2). 701–708. 27 indexed citations
11.
Nazim, Uddin Md., et al.. (2017). Enhancement of TRAIL-induced apoptosis by 5-fluorouracil requires activating Bax and p53 pathways in TRAIL-resistant lung cancers. Oncotarget. 8(11). 18095–18105. 26 indexed citations
12.
Nazim, Uddin Md., Ji‐Hong Moon, You-Jin Lee, Jae‐Won Seol, & Sang‐Youel Park. (2017). PPARγ activation by troglitazone enhances human lung cancer cells to TRAIL-induced apoptosis via autophagy flux. Oncotarget. 8(16). 26819–26831. 24 indexed citations
13.
Nazim, Uddin Md., et al.. (2017). Glipizide sensitizes lung cancer cells to TRAIL-induced apoptosis via Akt/mTOR/autophagy pathways. Oncotarget. 8(59). 100021–100033. 16 indexed citations
14.
Lee, Ju‐Hee, Ji‐Hong Moon, Uddin Md. Nazim, et al.. (2016). Melatonin protects skin keratinocyte from hydrogen peroxide-mediated cell deathviathe SIRT1 pathway. Oncotarget. 7(11). 12075–12088. 28 indexed citations
15.
Nazim, Uddin Md., Ji‐Hong Moon, Ju‐Hee Lee, et al.. (2016). Activation of autophagy flux by metformin downregulates cellular FLICE-like inhibitory protein and enhances TRAIL- induced apoptosis. Oncotarget. 7(17). 23468–23481. 53 indexed citations
16.
Moon, Ji‐Hong, Ju‐Hee Lee, Uddin Md. Nazim, et al.. (2016). Human prion protein-induced autophagy flux governs neuron cell damage in primary neuron cells. Oncotarget. 7(21). 29989–30002. 18 indexed citations
17.
18.
Nazim, Uddin Md., Jae‐Kyo Jeong, Jae‐Won Seol, et al.. (2015). Inhibition of the autophagy flux by gingerol enhances TRAIL-induced tumor cell death. Oncology Reports. 33(5). 2331–2336. 25 indexed citations
19.
Nazim, Uddin Md. & Sang‐Youel Park. (2015). Genistein enhances TRAIL-induced cancer cell death via inactivation of autophagic flux. Oncology Reports. 34(5). 2692–2698. 19 indexed citations
20.
Nazim, Uddin Md., et al.. (2011). Photo- and thermal degradation of piroxicam in aqueous solution.. PubMed. 73(4). 387–91. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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