U Weller

697 total citations
10 papers, 556 citations indexed

About

U Weller is a scholar working on Infectious Diseases, Molecular Biology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, U Weller has authored 10 papers receiving a total of 556 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Infectious Diseases, 4 papers in Molecular Biology and 4 papers in Public Health, Environmental and Occupational Health. Recurrent topics in U Weller's work include Streptococcal Infections and Treatments (4 papers), Lipid Membrane Structure and Behavior (3 papers) and Antimicrobial Resistance in Staphylococcus (3 papers). U Weller is often cited by papers focused on Streptococcal Infections and Treatments (4 papers), Lipid Membrane Structure and Behavior (3 papers) and Antimicrobial Resistance in Staphylococcus (3 papers). U Weller collaborates with scholars based in Germany, United Kingdom and United States. U Weller's co-authors include Angela Valeva, Iwan Walev, Sucharit Bhakdi, Michael Kehoe, B. Walker, Hagan Bayley, Michãel Palmer, S Bhakdi, Ivan Walev and Timothy J. Foster and has published in prestigious journals such as Journal of Biological Chemistry, Neuroscience and Antimicrobial Agents and Chemotherapy.

In The Last Decade

U Weller

10 papers receiving 537 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
U Weller Germany 10 256 153 108 75 70 10 556
Wiesia Bobkiewicz Germany 10 317 1.2× 129 0.8× 94 0.9× 85 1.1× 56 0.8× 10 538
H. J. Wellensiek Germany 9 285 1.1× 237 1.5× 223 2.1× 64 0.9× 73 1.0× 21 751
Lucien Cabanié France 11 332 1.3× 122 0.8× 242 2.2× 54 0.7× 122 1.7× 14 738
Holger Repp Germany 16 386 1.5× 62 0.4× 53 0.5× 54 0.7× 88 1.3× 29 633
Anke Osterloh Germany 17 330 1.3× 107 0.7× 245 2.3× 101 1.3× 24 0.3× 27 784
R. Rott Germany 16 389 1.5× 147 1.0× 82 0.8× 47 0.6× 114 1.6× 32 802
Varnesh Tiku Germany 10 506 2.0× 62 0.4× 115 1.1× 31 0.4× 39 0.6× 12 802
Alessia Landi Belgium 9 242 0.9× 110 0.7× 154 1.4× 34 0.5× 39 0.6× 13 525
Jens Eriksson Sweden 15 258 1.0× 70 0.5× 78 0.7× 42 0.6× 63 0.9× 36 531
R Fatzer Switzerland 23 613 2.4× 116 0.8× 56 0.5× 41 0.5× 186 2.7× 83 1.4k

Countries citing papers authored by U Weller

Since Specialization
Citations

This map shows the geographic impact of U Weller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by U Weller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites U Weller more than expected).

Fields of papers citing papers by U Weller

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by U Weller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by U Weller. The network helps show where U Weller may publish in the future.

Co-authorship network of co-authors of U Weller

This figure shows the co-authorship network connecting the top 25 collaborators of U Weller. A scholar is included among the top collaborators of U Weller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with U Weller. U Weller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Bhakdi, Sucharit, Hagan Bayley, Angela Valeva, et al.. (1996). Staphylococcal alpha-toxin, streptolysin-O, and Escherichia coli hemolysin: prototypes of pore-forming bacterial cytolysins. Archives of Microbiology. 165(2). 73–79. 243 indexed citations
2.
Walev, Ivan, et al.. (1996). Selective killing of human monocytes and cytokine release provoked by sphingomyelinase (beta-toxin) of Staphylococcus aureus. Infection and Immunity. 64(8). 2974–2979. 71 indexed citations
3.
Bhakdi, Sucharit, Iwan Walev, Daniel E Jonas, et al.. (1996). Pathogenesis of Sepsis Syndrome: Possible Relevance of Pore-Forming Bacterial Toxins. Current topics in microbiology and immunology. 216. 101–118. 16 indexed citations
4.
Ashton, Anthony C., Yan Li, Frédéric Doussau, et al.. (1995). Tetanus Toxin Inhibits Neuroexocytosis Even When Its Zn2+-dependent Protease Activity Is Removed. Journal of Biological Chemistry. 270(52). 31386–31390. 34 indexed citations
5.
Walev, Iwan, et al.. (1995). Binding, oligomerization, and pore formation by streptolysin O in erythrocytes and fibroblast membranes: detection of nonlytic polymers. Infection and Immunity. 63(4). 1188–1194. 32 indexed citations
6.
7.
Dayanithi, Govindan, Brigitte Stecher, Shinji Yamasaki, et al.. (1994). Exploring the functional domain and the target of the tetanus toxin light chain in neurohypophysial terminals. Neuroscience. 58(2). 423–431. 32 indexed citations
8.
Martín, E, et al.. (1994). Novel aspect of amphotericin B action: accumulation in human monocytes potentiates killing of phagocytosed Candida albicans. Antimicrobial Agents and Chemotherapy. 38(1). 13–22. 28 indexed citations
9.
10.
Ahnert‐Hilger, Gudrun, et al.. (1990). Chains and fragments of tetanus toxin, and their contribution to toxicity.. PubMed. 84(3). 229–36. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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