Trung H.M. Pham

3.0k total citations · 1 hit paper
24 papers, 2.3k citations indexed

About

Trung H.M. Pham is a scholar working on Immunology, Molecular Biology and Infectious Diseases. According to data from OpenAlex, Trung H.M. Pham has authored 24 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 8 papers in Molecular Biology and 7 papers in Infectious Diseases. Recurrent topics in Trung H.M. Pham's work include T-cell and B-cell Immunology (7 papers), Sphingolipid Metabolism and Signaling (6 papers) and Salmonella and Campylobacter epidemiology (5 papers). Trung H.M. Pham is often cited by papers focused on T-cell and B-cell Immunology (7 papers), Sphingolipid Metabolism and Signaling (6 papers) and Salmonella and Campylobacter epidemiology (5 papers). Trung H.M. Pham collaborates with scholars based in United States, United Kingdom and Japan. Trung H.M. Pham's co-authors include Jason G. Cyster, Takaharu Okada, Charles G. Lo, Shaun R. Coughlin, Mehrdad Matloubian, Rajita Pappu, Susan R. Schwab, Irina Grigorova, Ying Xu and Denise M. Monack and has published in prestigious journals such as Science, Journal of Clinical Investigation and Nature Genetics.

In The Last Decade

Trung H.M. Pham

23 papers receiving 2.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A Gut Commensal-Produced Metabolite Mediates Colonization Resistance to Salmonella Infection

2018 354 citations Amanda Jacobson, Lilian H. Lam et al. Cell Host & Microbe profile →

Peers

Trung H.M. Pham

IMMUN

ONCOL

Michael L. Pendrak United States

Michael E. Hobert United States

Neil Warner United States

Eiji Umemoto Japan

Zbigniew Mikulski United States

S P Colgan United States

Hans‐Jürgen Mägert Germany

Nesrin Özören Türkiye

Garabet Yeretssian United States

Yi‐Nan Gong United States

Michael L. Pendrak United States

Trung H.M. Pham

905 ×0.8

386 ×0.4

IMMUN

127 ×0.4

ONCOL

114 ×0.4

199 ×0.9

Citations per year, relative to Trung H.M. Pham Trung H.M. Pham (= 1×) peers Michael L. Pendrak

Countries citing papers authored by Trung H.M. Pham

Since Specialization

Citations

This map shows the geographic impact of Trung H.M. Pham's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Trung H.M. Pham with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Trung H.M. Pham more than expected).

Fields of papers citing papers by Trung H.M. Pham

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Trung H.M. Pham. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Trung H.M. Pham. The network helps show where Trung H.M. Pham may publish in the future.

Co-authorship network of co-authors of Trung H.M. Pham

This figure shows the co-authorship network connecting the top 25 collaborators of Trung H.M. Pham. A scholar is included among the top collaborators of Trung H.M. Pham based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Trung H.M. Pham. Trung H.M. Pham is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Luccia, Blanda Di, Liliana M. Massis, Daniel Butler, et al.. (2025). Salmonella-superspreader hosts require gut regulatory T cells to maintain a disease-tolerant state. The Journal of Experimental Medicine. 222(11).

Butler, Daniel, Blanda Di Luccia, Vida Shokoohi, et al.. (2024). Transcriptional profiling links unique human macrophage phenotypes to the growth of intracellular Salmonella enterica serovar Typhi. Scientific Reports. 14(1). 12811–12811. 2 indexed citations

Pham, Trung H.M., Yuan Xue, Susan Brewer, et al.. (2023). Single-cell profiling identifies ACE ⁺ granuloma macrophages as a nonpermissive niche for intracellular bacteria during persistent Salmonella infection. Science Advances. 9(1). eadd4333–eadd4333. 17 indexed citations

Pham, Trung H.M. & Denise M. Monack. (2023). Turning foes into permissive hosts: manipulation of macrophage polarization by intracellular bacteria. Current Opinion in Immunology. 84. 102367–102367. 8 indexed citations

Brewer, Susan, Jens Kortmann, Sky W. Brubaker, et al.. (2021). A Salmonella Typhi RNA thermosensor regulates virulence factors and innate immune evasion in response to host temperature. PLoS Pathogens. 17(3). e1009345–e1009345. 20 indexed citations

Jennings, E. R., Jingkun Zeng, Regina A. Günster, et al.. (2019). Salmonella Effector SteE Converts the Mammalian Serine/Threonine Kinase GSK3 into a Tyrosine Kinase to Direct Macrophage Polarization. Cell Host & Microbe. 27(1). 41–53.e6. 86 indexed citations

Pham, Trung H.M., Susan Brewer, Teresa L. M. Thurston, et al.. (2019). Salmonella-Driven Polarization of Granuloma Macrophages Antagonizes TNF-Mediated Pathogen Restriction during Persistent Infection. Cell Host & Microbe. 27(1). 54–67.e5. 91 indexed citations

Jacobson, Amanda, Lilian H. Lam, Manohary Rajendram, et al.. (2018). A Gut Commensal-Produced Metabolite Mediates Colonization Resistance to Salmonella Infection. Cell Host & Microbe. 24(2). 296–307.e7. 354 indexed citations breakdown →

Stewart, Sarah, et al.. (2018). Well-Appearing Newborn With a Vesiculobullous Rash at Birth. PEDIATRICS. 141(3). 1 indexed citations

10.

Carden, Sarah, G. Walker, Jared Honeycutt, et al.. (2017). Pseudogenization of the Secreted Effector Gene sseI Confers Rapid Systemic Dissemination of S. Typhimurium ST313 within Migratory Dendritic Cells. Cell Host & Microbe. 21(2). 182–194. 65 indexed citations

11.

Ravindranath, Mepur H., et al.. (2014). Suppression of allo-human leucocyte antigen (HLA) antibodies secreted by B memory cellsin vitro: intravenous immunoglobulin (IVIg) versusa monoclonal anti-HLA-E IgG that mimics HLA-I reactivities of IVIg. Clinical & Experimental Immunology. 177(2). 464–477. 15 indexed citations

12.

Lambe, Teresa, Greg Crawford, Andrew L. Johnson, et al.. (2011). DOCK8 is essential for T‐cell survival and the maintenance of CD8⁺ T‐cell memory. European Journal of Immunology. 41(12). 3423–3435. 90 indexed citations

13.

Arnon, Tal I., Ying Xu, Charles G. Lo, et al.. (2011). GRK2-Dependent S1PR1 Desensitization Is Required for Lymphocytes to Overcome Their Attraction to Blood. Science. 333(6051). 1898–1903. 164 indexed citations

14.

Camerer, Eric, Jean B. Regard, Ivo Cornelissen, et al.. (2009). Sphingosine-1-phosphate in the plasma compartment regulates basal and inflammation-induced vascular leak in mice. Journal of Clinical Investigation. 119(7). 1871–9. 297 indexed citations

15.

Pham, Trung H.M., Peter Bałuk, Ying Xu, et al.. (2009). Lymphatic endothelial cell sphingosine kinase activity is required for lymphocyte egress and lymphatic patterning. The Journal of Cell Biology. 187(7). i15–i15. 7 indexed citations

16.

Pham, Trung H.M., Peter Bałuk, Ying Xu, et al.. (2009). Lymphatic endothelial cell sphingosine kinase activity is required for lymphocyte egress and lymphatic patterning. The Journal of Experimental Medicine. 207(1). 17–27. 372 indexed citations

17.

Pham, Trung H.M., Takaharu Okada, Mehrdad Matloubian, Charles G. Lo, & Jason G. Cyster. (2008). S1P1 Receptor Signaling Overrides Retention Mediated by Gαi-Coupled Receptors to Promote T Cell Egress. Immunity. 28(1). 122–133. 358 indexed citations

18.

Grigorova, Irina, Susan R. Schwab, Tri Giang Phan, et al.. (2008). Cortical sinus probing, S1P1-dependent entry and flow-based capture of egressing T cells. Nature Immunology. 10(1). 58–65. 157 indexed citations

19.

Cai, Junchao, Paul I. Terasaki, Qiuxia Mao, et al.. (2006). Development of Nondonor-Specific HLA-DR Antibodies in Allograft Recipients Is Associated with Shared Epitopes with Mismatched Donor DR Antigens. American Journal of Transplantation. 6(12). 2947–2954. 66 indexed citations

20.

Desrosiers, Marie‐Pierre, Agnieszka Kielczewska, J. Concepción Loredo‐Osti, et al.. (2005). Epistasis between mouse Klra and major histocompatibility complex class I loci is associated with a new mechanism of natural killer cell–mediated innate resistance to cytomegalovirus infection. Nature Genetics. 37(6). 593–599. 123 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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