Tomoki Bo

428 total citations
18 papers, 323 citations indexed

About

Tomoki Bo is a scholar working on Molecular Biology, Cell Biology and Oncology. According to data from OpenAlex, Tomoki Bo has authored 18 papers receiving a total of 323 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 5 papers in Cell Biology and 4 papers in Oncology. Recurrent topics in Tomoki Bo's work include Mitochondrial Function and Pathology (8 papers), DNA Repair Mechanisms (6 papers) and ATP Synthase and ATPases Research (4 papers). Tomoki Bo is often cited by papers focused on Mitochondrial Function and Pathology (8 papers), DNA Repair Mechanisms (6 papers) and ATP Synthase and ATPases Research (4 papers). Tomoki Bo collaborates with scholars based in Japan. Tomoki Bo's co-authors include Osamu Inanami, Tohru Yamamori, Kumiko Yamamoto, Motofumi Suzuki, Yuri Sakai, Hironobu Yasui, Junichi Fujii, Tsukasa Osaki, Masaki Nagane and Tetsuro Ago and has published in prestigious journals such as Biochemical and Biophysical Research Communications, Molecules and Molecular Biology of the Cell.

In The Last Decade

Tomoki Bo

18 papers receiving 322 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tomoki Bo Japan 11 213 50 39 33 30 18 323
Chun‐Yue I. Lee United States 8 274 1.3× 43 0.9× 26 0.7× 32 1.0× 15 0.5× 9 344
Xielan Kuang China 13 227 1.1× 60 1.2× 29 0.7× 42 1.3× 9 0.3× 27 358
Jee Hyun Rho South Korea 12 189 0.9× 41 0.8× 21 0.5× 36 1.1× 15 0.5× 23 364
Xiaoting Xi China 10 263 1.2× 95 1.9× 25 0.6× 17 0.5× 16 0.5× 15 398
Won Suk Yang South Korea 10 299 1.4× 54 1.1× 10 0.3× 42 1.3× 20 0.7× 20 426
Renata Colla Italy 5 234 1.1× 67 1.3× 11 0.3× 38 1.2× 22 0.7× 6 366
Tsuyoshi Yamanaka Japan 14 133 0.6× 62 1.2× 47 1.2× 48 1.5× 34 1.1× 34 427
Q. Chen United States 3 348 1.6× 52 1.0× 23 0.6× 78 2.4× 22 0.7× 4 461
Luguo Sun China 12 228 1.1× 43 0.9× 13 0.3× 42 1.3× 25 0.8× 16 372

Countries citing papers authored by Tomoki Bo

Since Specialization
Citations

This map shows the geographic impact of Tomoki Bo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomoki Bo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomoki Bo more than expected).

Fields of papers citing papers by Tomoki Bo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomoki Bo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomoki Bo. The network helps show where Tomoki Bo may publish in the future.

Co-authorship network of co-authors of Tomoki Bo

This figure shows the co-authorship network connecting the top 25 collaborators of Tomoki Bo. A scholar is included among the top collaborators of Tomoki Bo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomoki Bo. Tomoki Bo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Bo, Tomoki, Tsukasa Osaki, & Junichi Fujii. (2024). Dephosphorylation of branched-chain α-keto acid dehydrogenase E1α (BCKDHA) promotes branched-chain amino acid catabolism and renders cancer cells resistant to X-rays by mitigating DNA damage. Biochemical and Biophysical Research Communications. 742. 151154–151154. 1 indexed citations
2.
3.
Bo, Tomoki & Junichi Fujii. (2024). Primary Roles of Branched Chain Amino Acids (BCAAs) and Their Metabolism in Physiology and Metabolic Disorders. Molecules. 30(1). 56–56. 10 indexed citations
4.
Bo, Tomoki, et al.. (2023). Heme Biosynthesis is Crucial for Cell Survival and Mitochondrial OXPHOS after X Irradiation. Radiation Research. 201(1). 48–54. 2 indexed citations
5.
Fujii, Junichi, Tsukasa Osaki, & Tomoki Bo. (2022). Ascorbate Is a Primary Antioxidant in Mammals. Molecules. 27(19). 6187–6187. 23 indexed citations
6.
Deguchi, Tatsuya, Kenji Hosoya, Yusuke Murase, et al.. (2021). Metformin preferentially enhances the radio-sensitivity of cancer stem-like cells with highly mitochondrial respiration ability in HMPOS. Molecular Therapy — Oncolytics. 22. 143–151. 13 indexed citations
7.
Bo, Tomoki, Hironobu Yasui, Tohru Shiga, et al.. (2021). Eribulin improves tumor oxygenation demonstrated by 18F-DiFA hypoxia imaging, leading to radio-sensitization in human cancer xenograft models. European Journal of Nuclear Medicine and Molecular Imaging. 49(3). 821–833. 2 indexed citations
8.
Bo, Tomoki, Sho Kobayashi, Osamu Inanami, et al.. (2021). LAT1 inhibitor JPH203 sensitizes cancer cells to radiation by enhancing radiation-induced cellular senescence. Translational Oncology. 14(11). 101212–101212. 11 indexed citations
9.
Fujimoto, Masaki, Tomoki Bo, Kumiko Yamamoto, et al.. (2020). Radiation-induced abnormal centrosome amplification and mitotic catastrophe in human cervical tumor HeLa cells and murine mammary tumor EMT6 cells. Journal of Clinical Biochemistry and Nutrition. 67(3). 240–247. 5 indexed citations
10.
Bo, Tomoki, Tohru Yamamori, Kumiko Yamamoto, et al.. (2019). Mitochondrial fission promotes radiation-induced increase in intracellular Ca2+ level leading to mitotic catastrophe in mouse breast cancer EMT6 cells. Biochemical and Biophysical Research Communications. 522(1). 144–150. 16 indexed citations
11.
Yamamoto, Kumiko, Hironobu Yasui, Tomoki Bo, et al.. (2018). Genotoxic Responses of Mitochondrial Oxygen Consumption Rate and Mitochondrial Semiquinone Radicals in Tumor Cells. Applied Magnetic Resonance. 49(8). 837–851. 8 indexed citations
12.
Yamamoto, Kumiko, Yoshinori Ikenaka, Takahiro Ichise, et al.. (2018). Evaluation of mitochondrial redox status and energy metabolism of X-irradiated HeLa cells by LC/UV, LC/MS/MS and ESR. Free Radical Research. 52(6). 648–660. 11 indexed citations
13.
Yasui, Hironobu, Kumiko Yamamoto, Motofumi Suzuki, et al.. (2017). Lipophilic triphenylphosphonium derivatives enhance radiation-induced cell killing via inhibition of mitochondrial energy metabolism in tumor cells. Cancer Letters. 390. 160–167. 26 indexed citations
14.
Bo, Tomoki, Tohru Yamamori, Motofumi Suzuki, et al.. (2017). Calmodulin-dependent protein kinase II (CaMKII) mediates radiation-induced mitochondrial fission by regulating the phosphorylation of dynamin-related protein 1 (Drp1) at serine 616. Biochemical and Biophysical Research Communications. 495(2). 1601–1607. 63 indexed citations
15.
Suzuki, Motofumi, Tohru Yamamori, Tomoki Bo, Yuri Sakai, & Osamu Inanami. (2017). MK-8776, a novel Chk1 inhibitor, exhibits an improved radiosensitizing effect compared to UCN-01 by exacerbating radiation-induced aberrant mitosis. Translational Oncology. 10(4). 491–500. 27 indexed citations
16.
Sakai, Yuri, Tohru Yamamori, Tomoki Bo, et al.. (2017). NADPH oxidase 4 mediates ROS production in radiation-induced senescent cells and promotes migration of inflammatory cells. Free Radical Research. 52(1). 92–102. 40 indexed citations
17.
Yamamori, Tohru, Osamu Ichii, Tomoki Bo, et al.. (2016). Analysis of the mechanism of radiation-induced upregulation of mitochondrial abundance in mouse fibroblasts. Journal of Radiation Research. 58(3). 292–301. 23 indexed citations
18.
Yamamori, Tohru, Tomoki Bo, Yuri Sakai, et al.. (2015). Inhibition of the mitochondrial fission protein dynamin-related protein 1 (Drp1) impairs mitochondrial fission and mitotic catastrophe after x-irradiation. Molecular Biology of the Cell. 26(25). 4607–4617. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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