Tomas Fanutza

813 total citations
10 papers, 419 citations indexed

About

Tomas Fanutza is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Tomas Fanutza has authored 10 papers receiving a total of 419 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Cellular and Molecular Neuroscience and 3 papers in Neurology. Recurrent topics in Tomas Fanutza's work include Neuroscience and Neuropharmacology Research (3 papers), Alzheimer's disease research and treatments (3 papers) and Neuroinflammation and Neurodegeneration Mechanisms (3 papers). Tomas Fanutza is often cited by papers focused on Neuroscience and Neuropharmacology Research (3 papers), Alzheimer's disease research and treatments (3 papers) and Neuroinflammation and Neurodegeneration Mechanisms (3 papers). Tomas Fanutza collaborates with scholars based in United States, Canada and Germany. Tomas Fanutza's co-authors include Michelle E. Ehrlich, Robert D. Blitzer, Ben Readhead, Sam Gandy, Mickaël Audrain, Jean‐Vianney Haure‐Mirande, Joel T. Dudley, Soong Ho Kim, Eric E. Schadt and Minghui Wang and has published in prestigious journals such as Cellular and Molecular Life Sciences, PLoS Genetics and Molecular Psychiatry.

In The Last Decade

Tomas Fanutza

9 papers receiving 416 citations

Peers

Tomas Fanutza

PHYSI

NEURO

CMN

NEURO

Galina Marsh United States

Sergey Larionov Germany

Kenneth J. Colodner United States

Monika Plescher Germany

Fabrizio Biundo United States

Nobutaka Doe Japan

Kert Mätlik Finland

Joaquim Duran‐Vilaregut Spain

Wenjie Mao United States

Kyoko Iinuma Japan

Galina Marsh United States

Tomas Fanutza

148 ×0.8

PHYSI

152 ×1.0

100 ×0.7

NEURO

95 ×0.8

CMN

121 ×2.0

NEURO

Citations per year, relative to Tomas Fanutza Tomas Fanutza (= 1×) peers Galina Marsh

Countries citing papers authored by Tomas Fanutza

Since Specialization

Citations

This map shows the geographic impact of Tomas Fanutza's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomas Fanutza with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomas Fanutza more than expected).

Fields of papers citing papers by Tomas Fanutza

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomas Fanutza. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomas Fanutza. The network helps show where Tomas Fanutza may publish in the future.

Co-authorship network of co-authors of Tomas Fanutza

This figure shows the co-authorship network connecting the top 25 collaborators of Tomas Fanutza. A scholar is included among the top collaborators of Tomas Fanutza based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomas Fanutza. Tomas Fanutza is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

Bär, Julia, Tomas Fanutza, Lisa Seipold, et al.. (2024). Non-canonical function of ADAM10 in presynaptic plasticity. Cellular and Molecular Life Sciences. 81(1). 342–342.

Readhead, Ben, Jean‐Vianney Haure‐Mirande, Diego Mastroeni, et al.. (2020). miR155 regulation of behavior, neuropathology, and cortical transcriptomics in Alzheimer's disease. Acta Neuropathologica. 140(3). 295–315. 28 indexed citations

Fanutza, Tomas, Swati Naphade, Kizito‐Tshitoko Tshilenge, et al.. (2019). Nuclear Receptor Nr4a1 Regulates Striatal Striosome Development and Dopamine D₁Receptor Signaling. eNeuro. 6(5). ENEURO.0305–19.2019. 16 indexed citations

Bucher, Michael, Tomas Fanutza, & Marina Mikhaylova. (2019). Cytoskeletal makeup of the synapse: Shaft versus spine. Cytoskeleton. 77(3-4). 55–64. 35 indexed citations

Zakirova, Zuchra, Tomas Fanutza, Ben Readhead, et al.. (2018). Mutations in THAP1/DYT6 reveal that diverse dystonia genes disrupt similar neuronal pathways and functions. PLoS Genetics. 14(1). e1007169–e1007169. 57 indexed citations

Haure‐Mirande, Jean‐Vianney, Minghui Wang, Mickaël Audrain, et al.. (2018). Integrative approach to sporadic Alzheimer’s disease: deficiency of TYROBP in cerebral Aβ amyloidosis mouse normalizes clinical phenotype and complement subnetwork molecular pathology without reducing Aβ burden. Molecular Psychiatry. 24(3). 431–446. 58 indexed citations

Audrain, Mickaël, Jean‐Vianney Haure‐Mirande, Minghui Wang, et al.. (2018). Integrative approach to sporadic Alzheimer’s disease: deficiency of TYROBP in a tauopathy mouse model reduces C1q and normalizes clinical phenotype while increasing spread and state of phosphorylation of tau. Molecular Psychiatry. 24(9). 1383–1397. 52 indexed citations

Haure‐Mirande, Jean‐Vianney, Mickaël Audrain, Tomas Fanutza, et al.. (2017). Deficiency of TYROBP, an adapter protein for TREM2 and CR3 receptors, is neuroprotective in a mouse model of early Alzheimer’s pathology. Acta Neuropathologica. 134(5). 769–788. 88 indexed citations

Fanutza, Tomas, Dolores Del Prete, Michael J. Ford, Pablo E. Castillo, & Luciano D'adamio. (2015). APP and APLP2 interact with the synaptic release machinery and facilitate transmitter release at hippocampal synapses. eLife. 4. e09743–e09743. 64 indexed citations

10.

Ahboucha, Samir, Giuseppe Talani, Tomas Fanutza, et al.. (2012). Reduced brain levels of DHEAS in hepatic coma patients: Significance for increased GABAergic tone in hepatic encephalopathy. Neurochemistry International. 61(1). 48–53. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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