Timothy Schappe

3.3k total citations · 1 hit paper
20 papers, 1.5k citations indexed

About

Timothy Schappe is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Timothy Schappe has authored 20 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Immunology, 7 papers in Oncology and 5 papers in Molecular Biology. Recurrent topics in Timothy Schappe's work include Immune Cell Function and Interaction (15 papers), CAR-T cell therapy research (7 papers) and T-cell and B-cell Immunology (7 papers). Timothy Schappe is often cited by papers focused on Immune Cell Function and Interaction (15 papers), CAR-T cell therapy research (7 papers) and T-cell and B-cell Immunology (7 papers). Timothy Schappe collaborates with scholars based in United States, Australia and Norway. Timothy Schappe's co-authors include Todd A. Fehniger, Melissa M. Berrien-Elliott, Brea A. Jewell, Jeffrey Leong, Sara Abdel-Latif, Maximillian Rosario, Julia A. Wagner, Rizwan Romee, Nada A. Abumrad and Carly C. Neal and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Timothy Schappe

19 papers receiving 1.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Cytokine-induced memory-like natural killer cells exhibit enhanced responses against myeloid leukemia

2016 660 citations Rizwan Romee, Maximillian Rosario et al. Science Translational Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Timothy Schappe United States	13	1.0k	608	399	183	113	20	1.5k
Barry Ripley Japan	16	731 0.7×	378 0.6×	544 1.4×	112 0.6×	106 0.9×	24	1.5k
Sharad Shrestha United States	11	1.3k 1.3×	315 0.5×	725 1.8×	110 0.6×	277 2.5×	13	1.9k
Leila N. Varghese Australia	10	414 0.4×	512 0.8×	404 1.0×	149 0.8×	64 0.6×	14	1.1k
Nigel Sharfe Canada	19	959 1.0×	278 0.5×	570 1.4×	112 0.6×	114 1.0×	33	1.7k
Semir Beyaz United States	15	530 0.5×	214 0.4×	525 1.3×	89 0.5×	132 1.2×	25	1.2k
Fumitaka Terabe Japan	12	463 0.5×	365 0.6×	234 0.6×	111 0.6×	137 1.2×	16	1.1k
Matthew R. Farren United States	19	482 0.5×	625 1.0×	396 1.0×	43 0.2×	74 0.7×	34	1.3k
Marı́a N. Navarro Spain	17	684 0.7×	253 0.4×	434 1.1×	66 0.4×	111 1.0×	23	1.3k
Abhishek Aphale United States	13	880 0.9×	283 0.5×	370 0.9×	48 0.3×	154 1.4×	16	1.3k
Ritsu Suzuki Japan	9	942 0.9×	1.4k 2.3×	599 1.5×	55 0.3×	98 0.9×	9	1.9k

Countries citing papers authored by Timothy Schappe

Since Specialization

Citations

This map shows the geographic impact of Timothy Schappe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Timothy Schappe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Timothy Schappe more than expected).

Fields of papers citing papers by Timothy Schappe

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Timothy Schappe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Timothy Schappe. The network helps show where Timothy Schappe may publish in the future.

Co-authorship network of co-authors of Timothy Schappe

This figure shows the co-authorship network connecting the top 25 collaborators of Timothy Schappe. A scholar is included among the top collaborators of Timothy Schappe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Timothy Schappe. Timothy Schappe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Cubitt, Celia C., Pamela Wong, Jennifer A. Foltz, et al.. (2024). Induced CD8α identifies human NK cells with enhanced proliferative fitness and modulates NK cell activation. Journal of Clinical Investigation. 134(15). 3 indexed citations

Tran, Jennifer, Pamela Wong, Changxu Fan, et al.. (2023). Combined IL-12, IL-15 and IL-18 activation induces epigenetic changes in human NK cells during the memory-like state transition. The Journal of Immunology. 210(Supplement_1). 160.12–160.12. 1 indexed citations

Cubitt, Celia C., Ethan McClain, Michelle Becker‐Hapak, et al.. (2022). A novel fusion protein scaffold 18/12/TxM activates the IL-12, IL-15, and IL-18 receptors to induce human memory-like natural killer cells. Molecular Therapy — Oncolytics. 24. 585–596. 8 indexed citations

Marín, Nancy D., Bradley A. Krasnick, Michelle Becker‐Hapak, et al.. (2021). Memory-like Differentiation Enhances NK Cell Responses to Melanoma. Clinical Cancer Research. 27(17). 4859–4869. 44 indexed citations

Wong, Pamela, Julia A. Wagner, Melissa M. Berrien-Elliott, Timothy Schappe, & Todd A. Fehniger. (2021). Flow cytometry-based ex vivo murine NK cell cytotoxicity assay. STAR Protocols. 2(1). 100262–100262. 14 indexed citations

Wagner, Julia A., Pamela Wong, Timothy Schappe, et al.. (2020). Stage-Specific Requirement for Eomes in Mature NK Cell Homeostasis and Cytotoxicity. Cell Reports. 31(9). 107720–107720. 46 indexed citations

Marín, Nancy D., Michelle Becker‐Hapak, Joachim Koch, et al.. (2019). Abstract 1546: The CD30/CD16A bispecific innate immune cell engager AFM13 elicits heterogeneous single-cell NK cell responses and effectively triggers memory-like (ML) NK cells. Cancer Research. 79(13_Supplement). 1546–1546. 1 indexed citations

Berrien-Elliott, Melissa M., Julia A. Wagner, Rizwan Romee, et al.. (2018). Abstract 5704: Mass cytometry identifies the expansion, persistence, and immune checkpoints of adoptively transferred memory-like NK cells in patients with leukemia. Cancer Research. 78(13_Supplement). 5704–5704. 2 indexed citations

Wagner, Julia, Maximillian Rosario, Rizwan Romee, et al.. (2017). CD56bright NK cells exhibit potent antitumor responses following IL-15 priming. Journal of Clinical Investigation. 127(11). 4042–4058. 216 indexed citations

10.

Wagner, Julia A., Melissa M. Berrien-Elliott, Maximillian Rosario, et al.. (2016). Cytokine-Induced Memory-Like Differentiation Enhances Unlicensed Natural Killer Cell Antileukemia and FcγRIIIa-Triggered Responses. Biology of Blood and Marrow Transplantation. 23(3). 398–404. 43 indexed citations

11.

Wagner, Julia A., Rizwan Romee, Maximillian Rosario, et al.. (2016). Human CD56bright NK Cells Acquire Potent Anti-Leukemia Functionality Following IL-15 Priming. Blood. 128(22). 550–550.

12.

Romee, Rizwan, Maximillian Rosario, Melissa M. Berrien-Elliott, et al.. (2016). Cytokine-induced memory-like natural killer cells exhibit enhanced responses against myeloid leukemia. Science Translational Medicine. 8(357). 357ra123–357ra123. 660 indexed citations breakdown →

13.

Leong, Jeffrey, Stephanie E. Schneider, Ryan P. Sullivan, et al.. (2015). PTEN regulates natural killer cell trafficking in vivo. Proceedings of the National Academy of Sciences. 112(7). E700–9. 26 indexed citations

14.

Romee, Rizwan, Maximillian Rosario, Melissa M. Berrien-Elliott, et al.. (2015). Human Cytokine-Induced Memory-like NK Cells Exhibit in Vivo Anti-Leukemia Activity in Xenografted NSG Mice and in Patients with Acute Myeloid Leukemia (AML). Blood. 126(23). 101–101. 5 indexed citations

15.

Sullivan, Ryan P., Jeffrey Leong, Stephanie E. Schneider, et al.. (2015). MicroRNA-15/16 Antagonizes Myb To Control NK Cell Maturation. The Journal of Immunology. 195(6). 2806–2817. 41 indexed citations

16.

Craft, Clarissa S., Terri Pietka, Timothy Schappe, et al.. (2014). The Extracellular Matrix Protein MAGP1 Supports Thermogenesis and Protects Against Obesity and Diabetes Through Regulation of TGF-β. Diabetes. 63(6). 1920–1932. 53 indexed citations

17.

Pietka, Terri, Timothy Schappe, Caterina Conte, et al.. (2014). Adipose and Muscle Tissue Profile of CD36 Transcripts in Obese Subjects Highlights the Role of CD36 in Fatty Acid Homeostasis and Insulin Resistance. Diabetes Care. 37(7). 1990–1997. 31 indexed citations

18.

Love‐Gregory, Latisha, Richard Sherva, Timothy Schappe, et al.. (2010). Common CD36 SNPs reduce protein expression and may contribute to a protective atherogenic profile. Human Molecular Genetics. 20(1). 193–201. 119 indexed citations

19.

Mancuso, David J., Harold F. Sims, Kui Yang, et al.. (2010). Genetic Ablation of Calcium-independent Phospholipase A2γ Prevents Obesity and Insulin Resistance during High Fat Feeding by Mitochondrial Uncoupling and Increased Adipocyte Fatty Acid Oxidation. Journal of Biological Chemistry. 285(47). 36495–36510. 65 indexed citations

20.

Love‐Gregory, Latisha, Richard Sherva, Lingwei Sun, et al.. (2008). Variants in the CD36 gene associate with the metabolic syndrome and high-density lipoprotein cholesterol. Human Molecular Genetics. 17(11). 1695–1704. 156 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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