Thomas Sander

4.5k total citations
67 papers, 2.9k citations indexed

About

Thomas Sander is a scholar working on Psychiatry and Mental health, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Thomas Sander has authored 67 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Psychiatry and Mental health, 35 papers in Cellular and Molecular Neuroscience and 31 papers in Molecular Biology. Recurrent topics in Thomas Sander's work include Epilepsy research and treatment (34 papers), Neuroscience and Neuropharmacology Research (22 papers) and Genetics and Neurodevelopmental Disorders (19 papers). Thomas Sander is often cited by papers focused on Epilepsy research and treatment (34 papers), Neuroscience and Neuropharmacology Research (22 papers) and Genetics and Neurodevelopmental Disorders (19 papers). Thomas Sander collaborates with scholars based in Germany, United States and Austria. Thomas Sander's co-authors include Armin Heils, Ortrud K. Steinlein, Hans Rommelspacher, Helmut Harms, Margret R. Hoehe, Andrew Escayg, Lutz G. Schmidt, Peter Dufeu, Silke Kühn and Jürgen Gallinat and has published in prestigious journals such as PLoS ONE, Annals of Neurology and Biological Psychiatry.

In The Last Decade

Thomas Sander

67 papers receiving 2.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Sander Germany 30 1.6k 1.2k 1.0k 640 306 67 2.9k
Adolfo Sequeira United States 33 958 0.6× 1.5k 1.3× 492 0.5× 509 0.8× 434 1.4× 52 3.4k
Thomas Sander Germany 28 843 0.5× 961 0.8× 560 0.5× 565 0.9× 369 1.2× 52 2.2k
Nader D. Halim United States 12 752 0.5× 882 0.8× 508 0.5× 308 0.5× 635 2.1× 13 2.5k
Michiko Fujimoto Japan 30 641 0.4× 1.2k 1.0× 521 0.5× 382 0.6× 544 1.8× 72 2.7k
Qi Xu China 32 637 0.4× 1.3k 1.1× 410 0.4× 438 0.7× 304 1.0× 133 2.9k
Hendrik Bielau Germany 25 807 0.5× 852 0.7× 653 0.6× 161 0.3× 446 1.5× 47 3.4k
Claudette Boni France 27 1.2k 0.8× 817 0.7× 337 0.3× 218 0.3× 237 0.8× 44 2.4k
Osamu Shirakawa Japan 32 1.2k 0.7× 968 0.8× 556 0.5× 281 0.4× 552 1.8× 117 2.9k
William A. Alaynick United States 22 628 0.4× 1.3k 1.1× 296 0.3× 476 0.7× 344 1.1× 33 2.9k
Tomoko Toyota Japan 31 818 0.5× 1.5k 1.2× 344 0.3× 850 1.3× 398 1.3× 87 2.8k

Countries citing papers authored by Thomas Sander

Since Specialization
Citations

This map shows the geographic impact of Thomas Sander's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Sander with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Sander more than expected).

Fields of papers citing papers by Thomas Sander

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Sander. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Sander. The network helps show where Thomas Sander may publish in the future.

Co-authorship network of co-authors of Thomas Sander

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Sander. A scholar is included among the top collaborators of Thomas Sander based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Sander. Thomas Sander is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hempelmann, Anne, Joana Cobilanschi, Armin Heils, et al.. (2007). Lack of evidence of an allelic association of a functional GABRB3 exon 1a promoter polymorphism with idiopathic generalized epilepsy. Epilepsy Research. 74(1). 28–32. 13 indexed citations
2.
Brevern, Michael von, Anupama Shankar, Anna Wiste, et al.. (2006). Migrainous Vertigo: Mutation Analysis of the Candidate Genes CACNA1A, ATP1A2, SCN1A, and CACNB4. Headache The Journal of Head and Face Pain. 46(7). 1136–1141. 58 indexed citations
3.
Lorenz, Susanne, Tim Becker, Hiltrud Muhle, et al.. (2006). Association of BRD2 polymorphisms with photoparoxysmal response. Neuroscience Letters. 400(1-2). 135–139. 44 indexed citations
4.
Lang, Undine E., Thomas Sander, Falk W. Lohoff, et al.. (2006). Association of the met66 allele of brain-derived neurotrophic factor (BDNF) with smoking. Psychopharmacology. 190(4). 433–439. 79 indexed citations
5.
Hempelmann, Anne, Armin Heils, & Thomas Sander. (2006). Confirmatory evidence for an association of the connexin-36 gene with juvenile myoclonic epilepsy. Epilepsy Research. 71(2-3). 223–228. 42 indexed citations
6.
Lenzen, Kirsten P., Armin Heils, Susanne Lorenz, Anne Hempelmann, & Thomas Sander. (2005). Association analysis of the Arg220His variation of the human gene encoding the GABA δ subunit with idiopathic generalized epilepsy. Epilepsy Research. 65(1-2). 53–57. 17 indexed citations
7.
Schmitt, Bernhard, Gabriele Wohlrab, Thomas Sander, Ortrud K. Steinlein, & Beatrice Latal. (2005). Neonatal seizures with tonic clonic sequences and poor developmental outcome. Epilepsy Research. 65(3). 161–168. 31 indexed citations
8.
Lenzen, Kirsten P., Armin Heils, Susanne Lorenz, Anne Hempelmann, & Thomas Sander. (2005). Association Analysis of Malic Enzyme 2 Gene Polymorphisms with Idiopathic Generalized Epilepsy. Epilepsia. 46(10). 1637–1641. 10 indexed citations
9.
Haug, Karsten, Thomas Sander, Uwe Runge, et al.. (2002). A Splice-Site Mutation in GABRG2 Associated With Childhood Absence Epilepsy and Febrile Convulsions. Archives of Neurology. 59(7). 1137–1137. 215 indexed citations
10.
Sander, Thomas, Mohammad R. Toliat, Armin Heils, et al.. (2002). Association of the 867Asp variant of the human anion exchanger 3 gene with common subtypes of idiopathic generalized epilepsy. Epilepsy Research. 51(3). 249–255. 59 indexed citations
11.
12.
Escayg, Andrew, Michel De Waard, David D. Lee, et al.. (2000). Coding and Noncoding Variation of the Human Calcium-Channel β4-Subunit Gene CACNB4 in Patients with Idiopathic Generalized Epilepsy and Episodic Ataxia. The American Journal of Human Genetics. 66(5). 1531–1539. 289 indexed citations
13.
Haug, Karsten, Kerstin Hallmann, Steve Horvath, et al.. (2000). No evidence for association between the KCNQ3 gene and susceptibility to idiopathic generalized epilepsy. Epilepsy Research. 42(1). 57–62. 8 indexed citations
14.
Sander, Thomas, Helmut Harms, Peter Dufeu, et al.. (1998). Serotonin transporter gene variants in alcohol-dependent subjects with dissocial personality disorder. Biological Psychiatry. 43(12). 908–912. 119 indexed citations
15.
Hoehe, Margret R., Birgit Wendel, Ingrid Grunewald, et al.. (1998). Brief research communication: serotonin transporter (5-HTT) gene polymorphisms are not associated with susceptibility to mood disorders. American Journal of Medical Genetics. 81(1). 1–3. 112 indexed citations
16.
Volz, Armin, Klemens Kaupmann, Andreas Ziegler, et al.. (1998). Mapping, genomic structure, and polymorphisms of the human GABA B R1 receptor gene: evaluation of its involvement in idiopathic generalized epilepsy. Neurogenetics. 2(1). 47–54. 43 indexed citations
17.
Sander, Thomas, Helmut Harms, Jan Podschus, et al.. (1997). Allelic association of a dopamine transporter gene polymorphism in alcohol dependence with withdrawal seizures or delirium. Biological Psychiatry. 41(3). 299–304. 122 indexed citations
18.
Heinz, Andreas, Thomas Sander, Helmut Harms, et al.. (1996). Lack of Allelic Association of Dopamine D1 and D2 (TaqIA) Receptor Gene Polymorphisms with Reduced Dopaminergic Sensitivity in Alcoholism. Alcoholism Clinical and Experimental Research. 20(6). 1109–1113. 51 indexed citations
19.
Sander, Thomas, Thomas Hildmann, Diéter Janz, et al.. (1996). Exclusion of linkage between idiopathic generalized epilepsies and the GABAA receptor α1 and γ2 subunit gene cluster on chromosome 5. Epilepsy Research. 23(3). 235–244. 9 indexed citations
20.
Sander, Thomas, Thomas Hildmann, Thomas F. Wienker, et al.. (1996). Common subtypes of idiopathic generalized epilepsies: Lack of linkage to D20S19 close to candidate loci (EBN1, EEGV1) on chromosome 20. American Journal of Medical Genetics. 67(1). 31–39. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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