Thomas P. Cujec

1.6k total citations
16 papers, 863 citations indexed

About

Thomas P. Cujec is a scholar working on Molecular Biology, Virology and Infectious Diseases. According to data from OpenAlex, Thomas P. Cujec has authored 16 papers receiving a total of 863 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 8 papers in Virology and 5 papers in Infectious Diseases. Recurrent topics in Thomas P. Cujec's work include HIV Research and Treatment (8 papers), RNA Research and Splicing (6 papers) and HIV/AIDS drug development and treatment (5 papers). Thomas P. Cujec is often cited by papers focused on HIV Research and Treatment (8 papers), RNA Research and Splicing (6 papers) and HIV/AIDS drug development and treatment (5 papers). Thomas P. Cujec collaborates with scholars based in United States and Japan. Thomas P. Cujec's co-authors include B. Matija Peterlin, Koh Fujinaga, Jon Meyer, Hiroshi Okamoto, Ran Taube, Junmin Peng, David H. Price, Holly Chamberlin, David O. Morgan and Judit Garriga and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Genes & Development.

In The Last Decade

Thomas P. Cujec

16 papers receiving 838 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas P. Cujec United States 11 613 490 187 174 87 16 863
Mary Bakhanashvili Israel 18 576 0.9× 382 0.8× 324 1.7× 96 0.6× 234 2.7× 48 961
Markus‐Frederik Bohn United States 12 543 0.9× 222 0.5× 98 0.5× 128 0.7× 143 1.6× 23 788
Brendan Bell Canada 15 687 1.1× 230 0.5× 131 0.7× 162 0.9× 63 0.7× 30 938
Ronald Rooke France 14 191 0.3× 270 0.6× 258 1.4× 462 2.7× 175 2.0× 31 924
Holly L. MacArthur United States 9 299 0.5× 170 0.3× 225 1.2× 33 0.2× 59 0.7× 10 606
Douglas A. Dedera United States 12 419 0.7× 225 0.5× 136 0.7× 142 0.8× 121 1.4× 14 776
Elisa Franzolin Italy 13 474 0.8× 135 0.3× 85 0.5× 130 0.7× 70 0.8× 14 702
Matthew Woods Canada 13 250 0.4× 123 0.3× 83 0.4× 168 1.0× 65 0.7× 22 488
Marianna Dioszegi United States 10 244 0.4× 201 0.4× 119 0.6× 134 0.8× 116 1.3× 12 572
Wilhelm Bannwarth Switzerland 8 323 0.5× 135 0.3× 37 0.2× 220 1.3× 85 1.0× 11 705

Countries citing papers authored by Thomas P. Cujec

Since Specialization
Citations

This map shows the geographic impact of Thomas P. Cujec's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas P. Cujec with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas P. Cujec more than expected).

Fields of papers citing papers by Thomas P. Cujec

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas P. Cujec. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas P. Cujec. The network helps show where Thomas P. Cujec may publish in the future.

Co-authorship network of co-authors of Thomas P. Cujec

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas P. Cujec. A scholar is included among the top collaborators of Thomas P. Cujec based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas P. Cujec. Thomas P. Cujec is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Cohen, Michael P., et al.. (2022). A fully automated high-throughput plasmid purification workstation for the generation of mammalian cell expression-quality DNA. SLAS TECHNOLOGY. 27(4). 227–236. 1 indexed citations
2.
Crissman, John, et al.. (2020). RNase H-dependent PCR enables highly specific amplification of antibody variable domains from single B-cells. PLoS ONE. 15(11). e0241803–e0241803. 4 indexed citations
3.
Wu, Xiufeng, Diana I. Ruiz, Flora Huang, et al.. (2016). Comparing domain interactions within antibody Fabs with kappa and lambda light chains. mAbs. 8(7). 1276–1285. 23 indexed citations
4.
Okamoto, Hiroshi, Thomas P. Cujec, Hisashi Yamanaka, & Naoyuki Kamatani. (2008). Molecular aspects of rheumatoid arthritis: role of transcription factors. FEBS Journal. 275(18). 4463–4470. 78 indexed citations
5.
Cujec, Thomas P., et al.. (2002). Selection of v-Abl Tyrosine Kinase Substrate Sequences from Randomized Peptide and Cellular Proteomic Libraries Using mRNA Display. Chemistry & Biology. 9(2). 253–264. 47 indexed citations
6.
Okamoto, Hiroshi, Thomas P. Cujec, B. Matija Peterlin, & Takashi Okamoto. (2000). HIV-1 Replication Is Inhibited by a Pseudo-substrate Peptide That Blocks Tat Transactivation. Virology. 270(2). 337–344. 13 indexed citations
7.
Okamoto, Hiroshi, Thomas P. Cujec, Mika Okamoto, et al.. (2000). Inhibition of the RNA-Dependent Transactivation and Replication of Human Immunodeficiency Virus Type 1 by a Fluoroquinoline Derivative K-37. Virology. 272(2). 402–408. 33 indexed citations
8.
Serna, Ivana de la, Thomas P. Cujec, Yinggang Shi, & Brett M. Tyler. (2000). Non-coordinate regulation of 5S rRNA genes and the gene encoding the 5S rRNA-binding ribosomal protein homolog in Neurospora crassa. Molecular and General Genetics MGG. 263(6). 987–994. 3 indexed citations
9.
Fujinaga, Koh, Ran Taube, Thomas P. Cujec, et al.. (1999). Interactions between Tat and TAR and Human Immunodeficiency Virus Replication Are Facilitated by Human Cyclin T1 but Not Cyclins T2a or T2b. Virology. 255(1). 182–189. 71 indexed citations
10.
Fujinaga, Koh, et al.. (1999). Interactions between human cyclin T, Tat, and the transactivation response element (TAR) are disrupted by a cysteine to tyrosine substitution found in mouse cyclin T. Proceedings of the National Academy of Sciences. 96(4). 1285–1290. 114 indexed citations
11.
Fujinaga, Koh, Thomas P. Cujec, Junmin Peng, et al.. (1998). The Ability of Positive Transcription Elongation Factor b To Transactivate Human Immunodeficiency Virus Transcription Depends on a Functional Kinase Domain, Cyclin T1, and Tat. Journal of Virology. 72(9). 7154–7159. 144 indexed citations
12.
Cujec, Thomas P., Hiroshi Okamoto, Koh Fujinaga, et al.. (1997). The HIV transactivator TAT binds to the CDK-activating kinase and activates the phosphorylation of the carboxy-terminal domain of RNA polymerase II. Genes & Development. 11(20). 2645–2657. 177 indexed citations
13.
Cujec, Thomas P., Helen Cho, Edio Maldonado, et al.. (1997). The Human Immunodeficiency Virus Transactivator Tat Interacts with the RNA Polymerase II Holoenzyme. Molecular and Cellular Biology. 17(4). 1817–1823. 106 indexed citations
14.
Cujec, Thomas P. & Brett M. Tyler. (1996). Functional promoter elements common to ribosomal protein and ribosomal RNA genes in Neurospora crassa. Molecular and General Genetics MGG. 253(1-2). 205–216. 7 indexed citations
15.
Cujec, Thomas P. & Brett M. Tyler. (1996). Nutritional and Growth Control of Ribosomal Protein mRNA and rRNA in Neurospora crassa. Nucleic Acids Research. 24(5). 943–950. 9 indexed citations
16.
Alonso, Alicia, Thomas P. Cujec, & B. Matija Peterlin. (1994). Effects of human chromosome 12 on interactions between Tat and TAR of human immunodeficiency virus type 1. Journal of Virology. 68(10). 6505–6513. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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