Thomas M. Hammond

1.4k total citations
37 papers, 905 citations indexed

About

Thomas M. Hammond is a scholar working on Molecular Biology, Plant Science and Cell Biology. According to data from OpenAlex, Thomas M. Hammond has authored 37 papers receiving a total of 905 indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 14 papers in Plant Science and 7 papers in Cell Biology. Recurrent topics in Thomas M. Hammond's work include Fungal and yeast genetics research (22 papers), DNA Repair Mechanisms (11 papers) and Protist diversity and phylogeny (8 papers). Thomas M. Hammond is often cited by papers focused on Fungal and yeast genetics research (22 papers), DNA Repair Mechanisms (11 papers) and Protist diversity and phylogeny (8 papers). Thomas M. Hammond collaborates with scholars based in United States, United Kingdom and Sweden. Thomas M. Hammond's co-authors include Nancy P. Keller, Patrick Shiu, Hua Xiao, Marilyn J. Roossinck, Daren W. Brown, Michael B. Collins, Tami McDonald, Tony D. Perdue, Patricia J. Pukkila and Erin C. Boone and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Genetics.

In The Last Decade

Thomas M. Hammond

36 papers receiving 885 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas M. Hammond United States	19	549	518	199	180	69	37	905
Stefano F. F. Torriani Switzerland	18	1.1k 2.1×	315 0.6×	32 0.2×	568 3.2×	86 1.2×	31	1.4k
William Quaedvlieg Netherlands	12	979 1.8×	453 0.9×	17 0.1×	898 5.0×	31 0.4×	12	1.2k
Andrzej Chlebicki Poland	9	261 0.5×	116 0.2×	13 0.1×	201 1.1×	21 0.3×	68	392
Barbara Schuetze Germany	8	133 0.2×	563 1.1×	19 0.1×	95 0.5×	12 0.2×	8	765
H. Butin Germany	16	561 1.0×	273 0.5×	40 0.2×	564 3.1×	7 0.1×	87	871
R. C. Close New Zealand	15	464 0.8×	111 0.2×	46 0.2×	112 0.6×	24 0.3×	45	603
Jay William Pscheidt United States	15	492 0.9×	108 0.2×	124 0.6×	325 1.8×	8 0.1×	54	672
Catalina Salgado‐Salazar United States	15	517 0.9×	216 0.4×	12 0.1×	499 2.8×	9 0.1×	47	643
Ðenita Hadziabdic United States	15	311 0.6×	156 0.3×	129 0.6×	163 0.9×	98 1.4×	65	623

Countries citing papers authored by Thomas M. Hammond

Since Specialization

Citations

This map shows the geographic impact of Thomas M. Hammond's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas M. Hammond with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas M. Hammond more than expected).

Fields of papers citing papers by Thomas M. Hammond

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas M. Hammond. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas M. Hammond. The network helps show where Thomas M. Hammond may publish in the future.

Co-authorship network of co-authors of Thomas M. Hammond

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas M. Hammond. A scholar is included among the top collaborators of Thomas M. Hammond based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas M. Hammond. Thomas M. Hammond is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Boone, Erin C., et al.. (2023). A DEAD-box RNA helicase mediates meiotic silencing by unpaired DNA. G3 Genes Genomes Genetics. 13(8). 1 indexed citations

Lohmar, Jessica M., et al.. (2022). Gene drive by Fusarium SKC1 is dependent on its competing allele. Fungal Genetics and Biology. 163. 103749–103749. 2 indexed citations

Witz, Guillaume, et al.. (2021). Recombination-independent recognition of DNA homology for meiotic silencing in Neurospora crassa. Proceedings of the National Academy of Sciences. 118(33). 10 indexed citations

Svedberg, Jesper, Aaron A. Vogan, David J. Jacobson, et al.. (2021). An introgressed gene causes meiotic drive in Neurospora sitophila. Proceedings of the National Academy of Sciences. 118(17). 27 indexed citations

Hammond, Thomas M., et al.. (2020). A Nonhomologous End-Joining Mutant for Neurospora sitophila Research. Fungal Genetics Reports. 64(1). 2 indexed citations

Svedberg, Jesper, Pennapa Manitchotpisit, Daren W. Brown, et al.. (2019). Identification of rfk-1 , a Meiotic Driver Undergoing RNA Editing in Neurospora. Genetics. 212(1). 93–110. 20 indexed citations

Svedberg, Jesper, Sara Hosseini, Jun Chen, et al.. (2018). Datasets deposited for Svedberg et al. (2018). "Convergent evolution of complex genomic rearrangements in two fungal meiotic drive elements". Figshare. 1 indexed citations

Svedberg, Jesper, Sara Hosseini, Jun Chen, et al.. (2018). Convergent evolution of complex genomic rearrangements in two fungal meiotic drive elements. Nature Communications. 9(1). 4242–4242. 30 indexed citations

Proctor, Robert H., et al.. (2016). A Meiotic Drive Element in the Maize Pathogen Fusarium verticillioides Is Located Within a 102 kb Region of Chromosome V. G3 Genes Genomes Genetics. 6(8). 2543–2552. 8 indexed citations

10.

Hammond, Thomas M.. (2016). Sixteen Years of Meiotic Silencing by Unpaired DNA. Advances in genetics. 97. 1–42. 28 indexed citations

11.

Smith, Zachary J., et al.. (2016). A mus-51 RIP allele for transformation of Neurospora crassa. Fungal Genetics Reports. 62(1). 1–7. 1 indexed citations

12.

Smith, Zachary J., Hua Xiao, Erin C. Boone, et al.. (2014). Efficient Detection of Unpaired DNA Requires a Member of the Rad54-Like Family of Homologous Recombination Proteins. Genetics. 198(3). 895–904. 22 indexed citations

13.

Shiu, Patrick, et al.. (2014). A Critical Component of Meiotic Drive in Neurospora Is Located Near a Chromosome Rearrangement. Genetics. 197(4). 1165–1174. 24 indexed citations

14.

Hammond, Thomas M., et al.. (2013). Identification of Small RNAs Associated with Meiotic Silencing by Unpaired DNA. Genetics. 194(1). 279–284. 27 indexed citations

15.

Hammond, Thomas M., Hua Xiao, Erin C. Boone, et al.. (2013). Novel Proteins Required for Meiotic Silencing by Unpaired DNA and siRNA Generation in Neurospora crassa. Genetics. 194(1). 91–100. 24 indexed citations

16.

Hammond, Thomas M., et al.. (2012). Molecular dissection of Neurospora Spore killer meiotic drive elements. Proceedings of the National Academy of Sciences. 109(30). 12093–12098. 51 indexed citations

17.

Scherm, Barbara, Virgilio Balmas, Emanuela Azara, et al.. (2011). Altered trichothecene biosynthesis in TRI6 ‐silenced transformants of Fusarium culmorum influences the severity of crown and foot rot on durum wheat seedlings. Molecular Plant Pathology. 12(8). 759–771. 47 indexed citations

18.

Alexander, William G., Namboori B. Raju, Hua Xiao, et al.. (2007). DCL-1 colocalizes with other components of the MSUD machinery and is required for silencing. Fungal Genetics and Biology. 45(5). 719–727. 55 indexed citations

19.

Hammond, Thomas M., et al.. (2007). Aspergillus Mycoviruses Are Targets and Suppressors of RNA Silencing. Eukaryotic Cell. 7(2). 350–357. 116 indexed citations

20.

McDonald, Tami, Daren W. Brown, Nancy P. Keller, & Thomas M. Hammond. (2005). RNA Silencing of Mycotoxin Production in Aspergillus and Fusarium Species. Molecular Plant-Microbe Interactions. 18(6). 539–545. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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