Thomas Karonitsch

2.1k total citations
41 papers, 984 citations indexed

About

Thomas Karonitsch is a scholar working on Rheumatology, Immunology and Oncology. According to data from OpenAlex, Thomas Karonitsch has authored 41 papers receiving a total of 984 indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Rheumatology, 19 papers in Immunology and 13 papers in Oncology. Recurrent topics in Thomas Karonitsch's work include Rheumatoid Arthritis Research and Therapies (18 papers), Systemic Lupus Erythematosus Research (12 papers) and Cytokine Signaling Pathways and Interactions (11 papers). Thomas Karonitsch is often cited by papers focused on Rheumatoid Arthritis Research and Therapies (18 papers), Systemic Lupus Erythematosus Research (12 papers) and Cytokine Signaling Pathways and Interactions (11 papers). Thomas Karonitsch collaborates with scholars based in Austria, Germany and Slovakia. Thomas Karonitsch's co-authors include Josef S Smolen, G Steiner, Hans P. Kiener, K Dalwigk, Michael Bonelli, Stephan Blüml, Georg Schett, Jochen Zwerina, Clemens Scheinecker and Bernhard Manger and has published in prestigious journals such as PLoS ONE, The American Journal of Medicine and Annals of the Rheumatic Diseases.

In The Last Decade

Thomas Karonitsch

39 papers receiving 966 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Karonitsch Austria 19 464 355 236 151 142 41 984
Nida Meednu United States 17 268 0.6× 411 1.2× 373 1.6× 165 1.1× 58 0.4× 25 964
José Ramón Lamas Spain 20 422 0.9× 423 1.2× 289 1.2× 115 0.8× 81 0.6× 54 1.0k
Zhizhong Ye China 19 429 0.9× 363 1.0× 327 1.4× 124 0.8× 98 0.7× 58 1.1k
Jianping Guo China 18 322 0.7× 357 1.0× 320 1.4× 120 0.8× 91 0.6× 50 964
Peter van Lent Netherlands 15 312 0.7× 284 0.8× 358 1.5× 114 0.8× 42 0.3× 21 908
Hirotsugu Ide Japan 16 312 0.7× 344 1.0× 203 0.9× 279 1.8× 100 0.7× 36 993
Toru Yago Japan 17 480 1.0× 465 1.3× 446 1.9× 267 1.8× 130 0.9× 60 1.2k
Taiichiro Miyashita Japan 18 310 0.7× 299 0.8× 277 1.2× 144 1.0× 73 0.5× 50 845
Beate M. Rüger Austria 16 147 0.3× 318 0.9× 225 1.0× 181 1.2× 87 0.6× 28 970
Sladjana Andrejević Serbia 15 257 0.6× 185 0.5× 89 0.4× 41 0.3× 56 0.4× 40 618

Countries citing papers authored by Thomas Karonitsch

Since Specialization
Citations

This map shows the geographic impact of Thomas Karonitsch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Karonitsch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Karonitsch more than expected).

Fields of papers citing papers by Thomas Karonitsch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Karonitsch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Karonitsch. The network helps show where Thomas Karonitsch may publish in the future.

Co-authorship network of co-authors of Thomas Karonitsch

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Karonitsch. A scholar is included among the top collaborators of Thomas Karonitsch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Karonitsch. Thomas Karonitsch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Karonitsch, Thomas, Victoria Saferding, Markus Kieler, et al.. (2023). Amino Acids Fueling Fibroblast‐Like Synoviocyte Activation and Arthritis By Regulating Chemokine Expression and Leukocyte Migration. Arthritis & Rheumatology. 76(4). 531–540. 3 indexed citations
3.
Müller, Lena, Teresa Preglej, Leonhard X. Heinz, et al.. (2023). AB0056 CYTOKINE-DIRECTED CELLULAR CROSSTALK IMPRINTS SYNOVIAL PATHOTYPES IN RHEUMATOID ARTHRITIS. Annals of the Rheumatic Diseases. 82. 1207–1207. 1 indexed citations
4.
Mandl, Péter, Selma Tobudic, Thomas Karonitsch, et al.. (2022). Response to SARS-CoV-2 vaccination in systemic autoimmune rheumatic disease depends on immunosuppressive regimen: a matched, prospective cohort study. Annals of the Rheumatic Diseases. 81(7). 1017–1022. 12 indexed citations
5.
Karonitsch, Thomas, et al.. (2019). The immunobiology of mTOR in autoimmunity. Journal of Autoimmunity. 110. 102373–102373. 98 indexed citations
6.
Bonelli, Michael, K Dalwigk, Alexander Platzer, et al.. (2019). IRF1 is critical for the TNF-driven interferon response in rheumatoid fibroblast-like synoviocytes. Experimental & Molecular Medicine. 51(7). 1–11. 58 indexed citations
7.
Platzer, Alexander, Thomas Nußbaumer, Thomas Karonitsch, Josef S Smolen, & Daniel Aletaha. (2019). Analysis of gene expression in rheumatoid arthritis and related conditions offers insights into sex-bias, gene biotypes and co-expression patterns. PLoS ONE. 14(7). e0219698–e0219698. 13 indexed citations
8.
Mandl, Péter, Silvia Hayer, Thomas Karonitsch, et al.. (2016). Nicotinic acetylcholine receptors modulate osteoclastogenesis. Arthritis Research & Therapy. 18(1). 63–63. 40 indexed citations
9.
Herdy, Barbara, Thomas Karonitsch, Gregory I. Vladimer, et al.. (2015). The RNA‐binding protein HuR/ELAVL1 regulates IFN‐β mRNA abundance and the type I IFN response. European Journal of Immunology. 45(5). 1500–1511. 44 indexed citations
10.
Karonitsch, Thomas & Martin Aringer. (2014). Biologika bei SLE. Wiener Medizinische Wochenschrift. 165(1-2). 40–45. 1 indexed citations
11.
Bonelli, Michael, Lisa Göschl, Stephan Blüml, et al.. (2014). CD4+CD25-Foxp3+ T cells: a marker for lupus nephritis?. Arthritis Research & Therapy. 16(2). R104–R104. 40 indexed citations
12.
Bonelli, Michael, Lisa Göschl, Stephan Blüml, et al.. (2012). Abatacept (CTLA‐4IG) treatment reduces the migratory capacity of monocytes in patients with rheumatoid arthritis. Arthritis & Rheumatism. 65(3). 599–607. 49 indexed citations
13.
Karonitsch, Thomas, K Dalwigk, Carl W. Steiner, et al.. (2011). Interferon signals and monocytic sensitization of the interferon‐γ signaling pathway in the peripheral blood of patients with rheumatoid arthritis. Arthritis & Rheumatism. 64(2). 400–408. 29 indexed citations
14.
Kiener, Hans P. & Thomas Karonitsch. (2011). The synovium as a privileged site in rheumatoid arthritis: Cadherin-11 as a dominant player in synovial pathology. Best Practice & Research Clinical Rheumatology. 25(6). 767–777. 22 indexed citations
15.
Tomás, Inmaculada, Enijad Sahinbegovic, Elmar Aigner, et al.. (2010). Validation of a radiographic scoring system for haemochromatosis arthropathy. Annals of the Rheumatic Diseases. 69(12). 2145–2151. 19 indexed citations
16.
Sahinbegovic, Enijad, Inmaculada Tomás, Elmar Aigner, et al.. (2010). Hereditary Hemochromatosis as a Risk Factor for Joint Replacement Surgery. The American Journal of Medicine. 123(7). 659–662. 32 indexed citations
17.
Tomás, Inmaculada, Gisela Ruiz Heiland, Johanna Strehl, et al.. (2010). CCL17/thymus and activation–related chemokine in Churg‐Strauss syndrome. Arthritis & Rheumatism. 62(11). 3496–3503. 66 indexed citations
18.
Karonitsch, Thomas, Eva Feierl, Carl W. Steiner, et al.. (2009). Activation of the interferon‐γ signaling pathway in systemic lupus erythematosus peripheral blood mononuclear cells. Arthritis & Rheumatism. 60(5). 1463–1471. 77 indexed citations
19.
Grisar, Johannes, C. W. Steiner, Michael Bonelli, et al.. (2008). Systemic lupus erythematosus patients exhibit functional deficiencies of endothelial progenitor cells. Lara D. Veeken. 47(10). 1476–1483. 34 indexed citations
20.
Feierl, Eva, Josef S Smolen, Thomas Karonitsch, et al.. (2007). Engulfed cell remnants, and not cells undergoing apoptosis, constitute the LE-cell phenomenon. Autoimmunity. 40(4). 315–321. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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