Thomas Hunsaker

2.1k total citations
14 papers, 755 citations indexed

About

Thomas Hunsaker is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Thomas Hunsaker has authored 14 papers receiving a total of 755 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Cancer Research and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Thomas Hunsaker's work include Melanoma and MAPK Pathways (4 papers), Cancer, Hypoxia, and Metabolism (3 papers) and Developmental Biology and Gene Regulation (3 papers). Thomas Hunsaker is often cited by papers focused on Melanoma and MAPK Pathways (4 papers), Cancer, Hypoxia, and Metabolism (3 papers) and Developmental Biology and Gene Regulation (3 papers). Thomas Hunsaker collaborates with scholars based in United States, France and Switzerland. Thomas Hunsaker's co-authors include Jane E. Johnson, Philip J. Ebert, Amy W. Helms, Katherine Gowan, Yuji Nakada, R. Michael Henke, Ron Firestein, Marie Evangelista, David Peterson and Georgia Hatzivassiliou and has published in prestigious journals such as Neuron, Development and Cancer Cell.

In The Last Decade

Thomas Hunsaker

11 papers receiving 750 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas Hunsaker United States	8	566	182	149	121	110	14	755
Cesare Lancini Netherlands	12	837 1.5×	193 1.1×	266 1.8×	107 0.9×	128 1.2×	16	1.1k
Fernando C. Baltanás Spain	17	372 0.7×	45 0.2×	100 0.7×	125 1.0×	76 0.7×	33	655
Amy K. Weaver United States	8	506 0.9×	103 0.6×	39 0.3×	281 2.3×	60 0.5×	9	758
Hiroki Ishii Japan	14	664 1.2×	200 1.1×	40 0.3×	224 1.9×	264 2.4×	33	1.0k
Cindy Degerny France	14	697 1.2×	92 0.5×	35 0.2×	58 0.5×	137 1.2×	27	910
Luca Tiberi Italy	17	857 1.5×	118 0.6×	207 1.4×	156 1.3×	248 2.3×	28	1.2k
Shuyue Ren United States	13	380 0.7×	115 0.6×	43 0.3×	90 0.7×	54 0.5×	27	577
Janna Enderich Germany	11	713 1.3×	149 0.8×	103 0.7×	125 1.0×	61 0.6×	11	999
Maria F. Pazyra‐Murphy United States	18	591 1.0×	50 0.3×	81 0.5×	221 1.8×	124 1.1×	26	881
Caroline B. Wigley United Kingdom	19	303 0.5×	159 0.9×	255 1.7×	337 2.8×	169 1.5×	33	898

Countries citing papers authored by Thomas Hunsaker

Since Specialization

Citations

This map shows the geographic impact of Thomas Hunsaker's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Hunsaker with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Hunsaker more than expected).

Fields of papers citing papers by Thomas Hunsaker

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Hunsaker. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Hunsaker. The network helps show where Thomas Hunsaker may publish in the future.

Co-authorship network of co-authors of Thomas Hunsaker

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Hunsaker. A scholar is included among the top collaborators of Thomas Hunsaker based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Hunsaker. Thomas Hunsaker is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Ravichandran, Mirunalini, Jeff Lau, Jennifer A. Lacap, et al.. (2025). Data from RIT1<sup>M90I</sup> Is a Driver of Lung Adenocarcinoma Tumorigenesis and Resistance to Targeted Therapy. 1 indexed citations

Ravichandran, Mirunalini, Julie Weng, Luca Gerosa, et al.. (2025). Abstract 6849: RIT1M90I is a driver of lung adenocarcinoma tumorigenesis and resistance to targeted therapy. Cancer Research. 85(8_Supplement_1). 6849–6849.

Metcalfe, Ciara, Thomas Hunsaker, Tomislav Hafner, et al.. (2025). Abstract ND02: GDC-2992: A heterobifunctional Androgen Receptor (AR) antagonist and degrader for the treatment of AR wild-type and mutant prostate cancer. Cancer Research. 85(8_Supplement_2). ND02–ND02.

Shanahan, Fergus, Logan Brooks, Eva Lin, et al.. (2024). Computational Modeling of Drug Response Identifies Mutant-Specific Constraints for Dosing panRAF and MEK Inhibitors in Melanoma. Cancers. 16(16). 2914–2914.

Fischer, Holger, Mohammed Ullah, Cecile C. de la Cruz, et al.. (2020). Entrectinib, a TRK/ROS1 inhibitor with anti-CNS tumor activity: differentiation from other inhibitors in its class due to weak interaction with P-glycoprotein. Neuro-Oncology. 22(6). 819–829. 65 indexed citations

Cruz, Cecile C. de la, Thomas Hunsaker, Faye Vazvaei, et al.. (2019). Abstract 3894: Determination of the efficacious Entrectinib exposures required for pathway inhibition and anti-tumor activity in a subcutaneous and intracranialTPM3-NTRK1mutant tumor model. Cancer Research. 79(13_Supplement). 3894–3894. 3 indexed citations

Yen, Ivana, Fergus Shanahan, Mark Merchant, et al.. (2018). Pharmacological Induction of RAS-GTP Confers RAF Inhibitor Sensitivity in KRAS Mutant Tumors. Cancer Cell. 34(4). 611–625.e7. 47 indexed citations

Daemen, Anneleen, Bonnie Liu, Mandy Kwong, et al.. (2018). Pan-Cancer Metabolic Signature Predicts Co-Dependency on Glutaminase and De Novo Glutathione Synthesis Linked to a High-Mesenchymal Cell State. Cell Metabolism. 28(3). 383–399.e9. 56 indexed citations

Merchant, Mark, Jocelyn Chan, Jin Cheng, et al.. (2014). 387 Combination of the ERK inhibitor GDC-0994 with the MEK inhibitor cobimetinib significantly enhances anti-tumor activity in KRAS and BRAF mutant tumor models. European Journal of Cancer. 50. 124–124. 7 indexed citations

10.

Lahtela, Jenni, Laura Corson, Annabrita Hemmes, et al.. (2013). A high-content cellular senescence screen identifies candidate tumor suppressors, including EPHA3. Cell Cycle. 12(4). 625–634. 15 indexed citations

11.

McCleland, Mark L., Adam S. Adler, Yonglei Shang, et al.. (2012). An Integrated Genomic Screen Identifies LDHB as an Essential Gene for Triple-Negative Breast Cancer. Cancer Research. 72(22). 5812–5823. 150 indexed citations

12.

Nakada, Yuji, Thomas Hunsaker, R. Michael Henke, & Jane E. Johnson. (2004). Distinct domains within Mash1 and Math1 are required for function in neuronal differentiation versus neuronal cell-type specification. Development. 131(6). 1319–1330. 87 indexed citations

13.

Ebert, Philip J., John R. Timmer, Yuji Nakada, et al.. (2003). Zic1 represses Math1 expression via interactions with the Math1 enhancer and modulation of Math1 autoregulation. Development. 130(9). 1949–1959. 79 indexed citations

14.

Gowan, Katherine, et al.. (2001). Crossinhibitory Activities of Ngn1 and Math1 Allow Specification of Distinct Dorsal Interneurons. Neuron. 31(2). 219–232. 245 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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