Thomas Hägglöf

6.0k total citations
17 papers, 492 citations indexed

About

Thomas Hägglöf is a scholar working on Immunology, Molecular Biology and Epidemiology. According to data from OpenAlex, Thomas Hägglöf has authored 17 papers receiving a total of 492 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Immunology, 5 papers in Molecular Biology and 3 papers in Epidemiology. Recurrent topics in Thomas Hägglöf's work include Immune Cell Function and Interaction (9 papers), T-cell and B-cell Immunology (8 papers) and Immunotherapy and Immune Responses (3 papers). Thomas Hägglöf is often cited by papers focused on Immune Cell Function and Interaction (9 papers), T-cell and B-cell Immunology (8 papers) and Immunotherapy and Immune Responses (3 papers). Thomas Hägglöf collaborates with scholars based in United States, Sweden and Greece. Thomas Hägglöf's co-authors include Mikael C. I. Karlsson, Saikiran K. Sedimbi, Elizabeth A. Leadbetter, Thiago Y. Oliveira, Emilie K. Grasset, Michel C. Nussenzweig, Christian Gaebler, Gaëlle Breton, Marina Caskey and Pilar Mendoza and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Thomas Hägglöf

15 papers receiving 488 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Hägglöf United States 11 314 138 90 63 44 17 492
Willianne Hoepel Netherlands 11 182 0.6× 79 0.6× 71 0.8× 36 0.6× 29 0.7× 15 343
Amy Cross United Kingdom 14 184 0.6× 104 0.8× 82 0.9× 54 0.9× 62 1.4× 22 491
Marco De Zuani Czechia 13 199 0.6× 117 0.8× 62 0.7× 66 1.0× 56 1.3× 20 419
Kate Gibson United Kingdom 4 236 0.8× 124 0.9× 47 0.5× 103 1.6× 106 2.4× 5 495
Yoko Mizoguchi Japan 12 158 0.5× 64 0.5× 68 0.8× 70 1.1× 48 1.1× 35 327
Ottavia M. Delmonte United States 14 455 1.4× 120 0.9× 130 1.4× 93 1.5× 81 1.8× 33 680
Michael A. Oropallo United States 8 325 1.0× 102 0.7× 51 0.6× 41 0.7× 46 1.0× 11 476
Gabriela Trentin Scortegagna Brazil 5 255 0.8× 85 0.6× 44 0.5× 82 1.3× 28 0.6× 10 371
Guillermo Juárez‐Vega Mexico 12 160 0.5× 94 0.7× 137 1.5× 53 0.8× 19 0.4× 25 363
Manuela Testi Italy 13 157 0.5× 67 0.5× 55 0.6× 23 0.4× 36 0.8× 23 479

Countries citing papers authored by Thomas Hägglöf

Since Specialization
Citations

This map shows the geographic impact of Thomas Hägglöf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Hägglöf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Hägglöf more than expected).

Fields of papers citing papers by Thomas Hägglöf

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Hägglöf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Hägglöf. The network helps show where Thomas Hägglöf may publish in the future.

Co-authorship network of co-authors of Thomas Hägglöf

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Hägglöf. A scholar is included among the top collaborators of Thomas Hägglöf based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Hägglöf. Thomas Hägglöf is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Johnson, L’Aurelle A., et al.. (2025). Emerging involvement of CXCL13 in cancer development and progression. Cytokine & Growth Factor Reviews. 87. 73–88.
2.
Zhou, Pengcheng, Mohamed ElTanbouly, Julia Merkenschlager, et al.. (2024). Affinity maturation of antibody responses is mediated by differential plasma cell proliferation. Science. 387(6732). 413–420. 6 indexed citations
3.
Hägglöf, Thomas, et al.. (2023). RIPK1 deficiency prevents thymic NK1.1 expression and subsequent iNKT cell development. Frontiers in Immunology. 14. 1103591–1103591. 2 indexed citations
4.
Hägglöf, Thomas, et al.. (2022). T-bet+ B cells accumulate in adipose tissue and exacerbate metabolic disorder during obesity. Cell Metabolism. 34(8). 1121–1136.e6. 48 indexed citations
5.
Hägglöf, Thomas, Melissa Cipolla, Spencer T. Chen, et al.. (2022). Continuous germinal center invasion contributes to the diversity of the immune response. Cell. 186(1). 147–161.e15. 41 indexed citations
6.
Hägglöf, Thomas, et al.. (2022). Diet-induced obesity promotes CD11c+ T-bet+ B cell expansion in liver and adipose tissue. The Journal of Immunology. 208(Supplement_1). 160.09–160.09. 1 indexed citations
7.
Breton, Gaëlle, Pilar Mendoza, Thomas Hägglöf, et al.. (2021). Persistent cellular immunity to SARS-CoV-2 infection. The Journal of Experimental Medicine. 218(4). 81 indexed citations
8.
Hägglöf, Thomas, et al.. (2021). Receptor Interacting Protein Kinase Pathways Regulate Innate B Cell Developmental Checkpoints But Not Effector Function in Mice. Frontiers in Immunology. 12. 758407–758407. 1 indexed citations
9.
Sedimbi, Saikiran K., Thomas Hägglöf, Shan Wang, et al.. (2020). Combined proinflammatory cytokine and cognate activation of invariant natural killer T cells enhances anti-DNA antibody responses. Proceedings of the National Academy of Sciences. 117(16). 9054–9063. 11 indexed citations
10.
Hägglöf, Thomas, et al.. (2020). RIP Kinase death pathways regulate B cell homeostasis and splenic architecture. The Journal of Immunology. 204(1_Supplement). 153.12–153.12.
11.
Svedin, Emma, Michael Hühn, Pär Larsson, et al.. (2017). A Link Between a Common Mutation in CFTR and Impaired Innate and Adaptive Viral Defense. The Journal of Infectious Diseases. 216(10). 1308–1317. 11 indexed citations
12.
Hägglöf, Thomas, Saikiran K. Sedimbi, Jennifer L. Yates, et al.. (2016). Neutrophils license iNKT cells to regulate self-reactive mouse B cell responses. Nature Immunology. 17(12). 1407–1414. 28 indexed citations
13.
Smith, Laura E., Anna‐Maria Georgoudaki, Arnika Kathleen Wagner, et al.. (2016). Sensitivity of dendritic cells to NK-mediated lysis depends on the inflammatory environment and is modulated by CD54/CD226-driven interactions. Journal of Leukocyte Biology. 100(4). 781–789. 10 indexed citations
14.
Grasset, Emilie K., Amanda Duhlin, Hanna E. Agardh, et al.. (2015). Sterile inflammation in the spleen during atherosclerosis provides oxidation-specific epitopes that induce a protective B-cell response. Proceedings of the National Academy of Sciences. 112(16). E2030–8. 53 indexed citations
15.
Vomhof‐DeKrey, Emilie E., Jennifer L. Yates, Thomas Hägglöf, et al.. (2015). Cognate interaction with iNKT cells expands IL-10–producing B regulatory cells. Proceedings of the National Academy of Sciences. 112(40). 12474–12479. 29 indexed citations
16.
Sedimbi, Saikiran K., Thomas Hägglöf, & Mikael C. I. Karlsson. (2013). IL-18 in inflammatory and autoimmune disease. Cellular and Molecular Life Sciences. 70(24). 4795–4808. 121 indexed citations
17.
Enoksson, Sara Lind, Emilie K. Grasset, Thomas Hägglöf, et al.. (2011). The inflammatory cytokine IL-18 induces self-reactive innate antibody responses regulated by natural killer T cells. Proceedings of the National Academy of Sciences. 108(51). E1399–407. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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