Thomas Hackenbeck

723 total citations
12 papers, 463 citations indexed

About

Thomas Hackenbeck is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, Thomas Hackenbeck has authored 12 papers receiving a total of 463 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 6 papers in Cancer Research and 4 papers in Genetics. Recurrent topics in Thomas Hackenbeck's work include Cancer, Hypoxia, and Metabolism (6 papers), Renal and related cancers (3 papers) and Cell death mechanisms and regulation (2 papers). Thomas Hackenbeck is often cited by papers focused on Cancer, Hypoxia, and Metabolism (6 papers), Renal and related cancers (3 papers) and Cell death mechanisms and regulation (2 papers). Thomas Hackenbeck collaborates with scholars based in Germany, United Kingdom and France. Thomas Hackenbeck's co-authors include Michael S. Wiesener, Kai‐Uwe Eckardt, Christina Warnecke, Karl X. Knaup, Kerstin Amann, Carsten Willam, Johannes Schödel, Ruth Schietke, Xiaoqing Wu and Bernd Klanke and has published in prestigious journals such as PLoS ONE, The FASEB Journal and Kidney International.

In The Last Decade

Thomas Hackenbeck

12 papers receiving 458 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Hackenbeck Germany 11 271 231 66 59 59 12 463
Carl Simon Shelley United States 10 279 1.0× 286 1.2× 62 0.9× 127 2.2× 16 0.3× 13 651
Sally A. Nicholas United Kingdom 10 270 1.0× 94 0.4× 20 0.3× 37 0.6× 18 0.3× 12 426
Adrianna Moszyńska Poland 10 320 1.2× 336 1.5× 41 0.6× 69 1.2× 21 0.4× 14 549
Hiromichi Nagano Japan 8 228 0.8× 67 0.3× 43 0.7× 49 0.8× 16 0.3× 9 426
Colleen Byrnes United States 16 404 1.5× 116 0.5× 79 1.2× 48 0.8× 14 0.2× 42 989
Suk-Ran Yoon South Korea 11 270 1.0× 65 0.3× 21 0.3× 26 0.4× 16 0.3× 12 573
Julie Nijmeh United States 13 187 0.7× 74 0.3× 101 1.5× 30 0.5× 32 0.5× 21 475
Vidalba Rocher-Ros United Kingdom 3 222 0.8× 174 0.8× 37 0.6× 54 0.9× 10 0.2× 4 674
Aleksandra Cabaj Poland 8 190 0.7× 225 1.0× 33 0.5× 43 0.7× 20 0.3× 11 394
Jon Lecanda Spain 12 294 1.1× 81 0.4× 49 0.7× 55 0.9× 10 0.2× 16 528

Countries citing papers authored by Thomas Hackenbeck

Since Specialization
Citations

This map shows the geographic impact of Thomas Hackenbeck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Hackenbeck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Hackenbeck more than expected).

Fields of papers citing papers by Thomas Hackenbeck

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Hackenbeck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Hackenbeck. The network helps show where Thomas Hackenbeck may publish in the future.

Co-authorship network of co-authors of Thomas Hackenbeck

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Hackenbeck. A scholar is included among the top collaborators of Thomas Hackenbeck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Hackenbeck. Thomas Hackenbeck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Bartel, Alexander, José Horacio Grau, Julia Bitzegeio, et al.. (2024). Timely Monitoring of SARS-CoV-2 RNA Fragments in Wastewater Shows the Emergence of JN.1 (BA.2.86.1.1, Clade 23I) in Berlin, Germany. Viruses. 16(1). 102–102. 13 indexed citations
2.
Knaup, Karl X., Thomas Hackenbeck, Bernt Popp, et al.. (2018). Biallelic Expression of Mucin-1 in Autosomal Dominant Tubulointerstitial Kidney Disease: Implications for Nongenetic Disease Recognition. Journal of the American Society of Nephrology. 29(9). 2298–2309. 18 indexed citations
3.
Schley, Gunnar, Holger Scholz, Andre Kraus, et al.. (2015). Hypoxia inhibits nephrogenesis through paracrine Vegfa despite the ability to enhance tubulogenesis. Kidney International. 88(6). 1283–1292. 24 indexed citations
4.
Knaup, Karl X., Thomas Hackenbeck, Tilman Jobst‐Schwan, et al.. (2013). Hypoxia regulates the sperm associated antigen 4 (SPAG4) via HIF, which is expressed in renal clear cell carcinoma and promotes migration and invasion in vitro. Molecular Carcinogenesis. 53(12). 970–978. 25 indexed citations
5.
Jobst‐Schwan, Tilman, Karl X. Knaup, Rikke Nielsen, et al.. (2013). Renal uptake of the antiapoptotic protein survivin is mediated by megalin at the apical membrane of the proximal tubule. American Journal of Physiology-Renal Physiology. 305(5). F734–F744. 14 indexed citations
6.
Schietke, Ruth, Thomas Hackenbeck, Maxine Tran, et al.. (2012). Renal Tubular HIF-2α Expression Requires VHL Inactivation and Causes Fibrosis and Cysts. PLoS ONE. 7(1). e31034–e31034. 69 indexed citations
7.
Hackenbeck, Thomas, Regina Huber, Ruth Schietke, et al.. (2010). The GTPase RAB20 is a HIF target with mitochondrial localization mediating apoptosis in hypoxia. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1813(1). 1–13. 24 indexed citations
8.
Schödel, Johannes, Karl X. Knaup, Thomas Hackenbeck, et al.. (2010). Hypoxia‐inducible protein 2 is a novel lipid droplet protein and a specific target gene of hypoxia‐inducible factor‐1. The FASEB Journal. 24(11). 4443–4458. 123 indexed citations
9.
Hackenbeck, Thomas, Karl X. Knaup, Ruth Schietke, et al.. (2009). HIF-1 or HIF-2 induction is sufficient to achieve cell cycle arrest in NIH3T3 mouse fibroblasts independent from hypoxia. Cell Cycle. 8(9). 1386–1395. 55 indexed citations
10.
Warnecke, Christina, Alexander Weidemann, Ruth Schietke, et al.. (2008). The specific contribution of hypoxia-inducible factor-2α to hypoxic gene expression in vitro is limited and modulated by cell type-specific and exogenous factors. Experimental Cell Research. 314(10). 2016–2027. 42 indexed citations
11.
Lechler, Philipp, Xiaoqing Wu, Wanja M. Bernhardt, et al.. (2007). The Tumor Gene Survivin Is Highly Expressed in Adult Renal Tubular Cells. American Journal Of Pathology. 171(5). 1483–1498. 49 indexed citations
12.
Flohr, Aljoscha M., et al.. (2003). DNase I treatment of cDNA first strands prevents RT-PCR amplification of contaminating DNA sequences. BioTechniques. 35(5). 920–926. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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