Thomas Fath

3.5k total citations
59 papers, 2.4k citations indexed

About

Thomas Fath is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Thomas Fath has authored 59 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 26 papers in Cell Biology and 17 papers in Cellular and Molecular Neuroscience. Recurrent topics in Thomas Fath's work include Cellular Mechanics and Interactions (18 papers), Alzheimer's disease research and treatments (16 papers) and Cardiomyopathy and Myosin Studies (12 papers). Thomas Fath is often cited by papers focused on Cellular Mechanics and Interactions (18 papers), Alzheimer's disease research and treatments (16 papers) and Cardiomyopathy and Myosin Studies (12 papers). Thomas Fath collaborates with scholars based in Australia, United States and Germany. Thomas Fath's co-authors include Roland Brandt, Peter W. Gunning, Lars M. Ittner, Yazi D. Ke, Jürgen Götz, Galina Schevzov, Estelle Sontag, Janet van Eersel, Holly Stefen and Mian Bi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Thomas Fath

58 papers receiving 2.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Fath Australia 24 1.1k 887 588 545 381 59 2.4k
Chadwick M. Hales United States 27 1.6k 1.5× 790 0.9× 765 1.3× 442 0.8× 465 1.2× 47 2.8k
Jonathon L. Burman United States 12 2.2k 2.0× 506 0.6× 583 1.0× 593 1.1× 514 1.3× 14 3.6k
Suman Jayadev United States 28 1.4k 1.2× 773 0.9× 183 0.3× 632 1.2× 280 0.7× 86 2.6k
Jiro Takano Japan 22 1.1k 1.0× 1.2k 1.4× 610 1.0× 766 1.4× 273 0.7× 26 2.5k
Ko Miyoshi Japan 28 1.3k 1.2× 345 0.4× 638 1.1× 720 1.3× 267 0.7× 47 2.5k
Young Ho Suh South Korea 34 1.5k 1.4× 577 0.7× 402 0.7× 798 1.5× 186 0.5× 77 2.8k
Adam I. Fogel United States 13 2.4k 2.2× 633 0.7× 749 1.3× 493 0.9× 766 2.0× 15 4.2k
Neelam Shahani United States 26 1.5k 1.4× 588 0.7× 262 0.4× 696 1.3× 385 1.0× 39 2.6k
Christian Bjerggaard Vægter Denmark 25 1.0k 0.9× 818 0.9× 298 0.5× 1.0k 1.9× 604 1.6× 49 2.7k
Kazuhiko Tagawa Japan 25 1.4k 1.3× 385 0.4× 493 0.8× 644 1.2× 133 0.3× 52 2.0k

Countries citing papers authored by Thomas Fath

Since Specialization
Citations

This map shows the geographic impact of Thomas Fath's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Fath with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Fath more than expected).

Fields of papers citing papers by Thomas Fath

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Fath. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Fath. The network helps show where Thomas Fath may publish in the future.

Co-authorship network of co-authors of Thomas Fath

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Fath. A scholar is included among the top collaborators of Thomas Fath based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Fath. Thomas Fath is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hoven, Julia van der, Sian Genoud, Holly Stefen, et al.. (2024). Engineered Cyclotide Blocks Neuronal Excitotoxicity. Journal of Medicinal Chemistry. 68(5). 5211–5221. 2 indexed citations
2.
Hoven, Julia van der, Sian Genoud, Holly Stefen, et al.. (2024). Design of peptide therapeutics as protein–protein interaction inhibitors to treat neurodegenerative diseases. RSC Advances. 14(47). 34637–34642. 1 indexed citations
3.
Ippati, Stefania, Yuanyuan Deng, Julia van der Hoven, et al.. (2021). Rapid initiation of cell cycle reentry processes protects neurons from amyloid-β toxicity. Proceedings of the National Academy of Sciences. 118(12). 23 indexed citations
4.
Martin, Adam D., Jonathan P. Wojciechowski, Eric Y. Du, et al.. (2019). Decoupling the effects of hydrophilic and hydrophobic moieties at the neuron–nanofibre interface. Chemical Science. 11(5). 1375–1382. 9 indexed citations
5.
Bi, Mian, Amadeus Gladbach, Janet van Eersel, et al.. (2017). Tau exacerbates excitotoxic brain damage in an animal model of stroke. Nature Communications. 8(1). 473–473. 133 indexed citations
6.
Fröhlich, Dominik, Alexandra K. Suchowerska, Georg von Jonquières, et al.. (2016). In vivo characterization of the aspartyl-tRNA synthetase DARS: Homing in on the leukodystrophy HBSL. Neurobiology of Disease. 97(Pt A). 24–35. 21 indexed citations
7.
Patel, Shrujna, et al.. (2016). Tropomyosins in the healthy and diseased nervous system. Brain Research Bulletin. 126(Pt 3). 311–323. 23 indexed citations
8.
Couttas, Timothy A., Nupur Kain, Alexandra K. Suchowerska, et al.. (2016). Loss of ceramide synthase 2 activity, necessary for myelin biosynthesis, precedes tau pathology in the cortical pathogenesis of Alzheimer's disease. Neurobiology of Aging. 43. 89–100. 69 indexed citations
9.
Suchowerska, Alexandra K., et al.. (2015). Amyotrophic lateral sclerosis-associated mutant profilin 1 increases dendritic arborisation and spine formation in primary hippocampal neurons. Neuroscience Letters. 609. 223–228. 13 indexed citations
10.
Schevzov, Galina, Anthony J. Kee, Bin Wang, et al.. (2015). Regulation of cell proliferation by ERK and signal-dependent nuclear translocation of ERK is dependent on Tm5NM1-containing actin filaments. Molecular Biology of the Cell. 26(13). 2475–2490. 56 indexed citations
11.
Curthoys, Nikki M., Andrea R. Connor, Melissa Desouza, et al.. (2013). Tropomyosins induce neuritogenesis and determine neurite branching patterns in B35 neuroblastoma cells. Molecular and Cellular Neuroscience. 58. 11–21. 29 indexed citations
12.
Fath, Thomas. (2012). Tropomodulins and tropomyosins – organizers of cellular microcompartments. BioMolecular Concepts. 4(1). 89–101. 5 indexed citations
13.
Schevzov, Galina, Nikki M. Curthoys, Peter W. Gunning, & Thomas Fath. (2012). Functional Diversity of Actin Cytoskeleton in Neurons and its Regulation by Tropomyosin. International review of cell and molecular biology. 298. 33–94. 30 indexed citations
14.
Dale, Russell C., Vera Merheb, Sekhar Pillai, et al.. (2012). Antibodies to surface dopamine-2 receptor in autoimmune movement and psychiatric disorders. Brain. 135(11). 3453–3468. 262 indexed citations
15.
Schevzov, Galina, et al.. (2011). Tropomyosin isoforms and reagents. PubMed. 1(4). 135–164. 125 indexed citations
16.
Hook, Jeff, Frances A. Lemckert, Galina Schevzov, Thomas Fath, & Peter W. Gunning. (2011). Functional identity of the Gamma Tropomyosin gene. PubMed. 1(1). 49–59. 27 indexed citations
17.
Hundelt, Monika, Thomas Fath, Joaquı́n Jordán, et al.. (2009). Altered phosphorylation but no neurodegeneration in a mouse model of tau hyperphosphorylation. Neurobiology of Aging. 32(6). 991–1006. 25 indexed citations
18.
Götz, Jürgen, Della David, Frédéric J. Hoerndli, et al.. (2008). Functional Genomics Dissects Pathomechanisms in Tauopathies: Mitosis Failure and Unfolded Protein Response. Neurodegenerative Diseases. 5(3-4). 179–181. 9 indexed citations
19.
Fath, Thomas, Yazi D. Ke, Peter W. Gunning, Jürgen Götz, & Lars M. Ittner. (2008). Primary support cultures of hippocampal and substantia nigra neurons. Nature Protocols. 4(1). 78–85. 164 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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