Thomas E. Callis

6.9k total citations · 2 hit papers
26 papers, 5.3k citations indexed

About

Thomas E. Callis is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cancer Research. According to data from OpenAlex, Thomas E. Callis has authored 26 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 12 papers in Cardiology and Cardiovascular Medicine and 11 papers in Cancer Research. Recurrent topics in Thomas E. Callis's work include MicroRNA in disease regulation (10 papers), Circular RNAs in diseases (8 papers) and Congenital heart defects research (6 papers). Thomas E. Callis is often cited by papers focused on MicroRNA in disease regulation (10 papers), Circular RNAs in diseases (8 papers) and Congenital heart defects research (6 papers). Thomas E. Callis collaborates with scholars based in United States, Netherlands and China. Thomas E. Callis's co-authors include Jian‐Fu Chen, Da‐Zhi Wang, Scott M. Hammond, J. Michael Thomson, Frank L. Conlon, Elizabeth M. Mandel, Da‐Zhi Wang, Mariko Tatsuguchi, Zhong‐Liang Deng and Heeyoung Seok and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Thomas E. Callis

26 papers receiving 5.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation

2005 2.3k citations Jian‐Fu Chen, Elizabeth M. Mandel et al. Nature Genetics profile →
MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice

2009 679 citations Thomas E. Callis, Kumar Pandya et al. Journal of Clinical Investigation profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas E. Callis United States	20	4.2k	3.3k	836	366	257	26	5.3k
Andrew H Williams United States	9	3.0k 0.7×	2.1k 0.6×	403 0.5×	250 0.7×	313 1.2×	27	3.6k
Kathryn N. Ivey United States	26	4.6k 1.1×	3.0k 0.9×	596 0.7×	882 2.4×	174 0.7×	35	5.8k
Laurent Metzinger France	27	2.0k 0.5×	737 0.2×	287 0.3×	180 0.5×	362 1.4×	51	2.6k
Brad A. Amendt United States	37	3.6k 0.9×	751 0.2×	243 0.3×	242 0.7×	209 0.8×	116	4.7k
Bin Zhou United States	31	2.8k 0.7×	722 0.2×	1.1k 1.3×	564 1.5×	119 0.5×	85	3.8k
Erika Gustafsson Sweden	25	2.5k 0.6×	635 0.2×	746 0.9×	591 1.6×	166 0.6×	34	4.7k
Reed Hickey United States	17	1.6k 0.4×	483 0.1×	561 0.7×	451 1.2×	262 1.0×	19	2.7k
Diego Franco Spain	35	3.2k 0.7×	607 0.2×	1.4k 1.7×	588 1.6×	76 0.3×	146	4.1k
Caroline M. Alexander United States	38	2.5k 0.6×	1.0k 0.3×	141 0.2×	266 0.7×	500 1.9×	74	4.9k
Tiziana Santini Italy	21	4.7k 1.1×	4.0k 1.2×	92 0.1×	115 0.3×	171 0.7×	36	5.1k

Countries citing papers authored by Thomas E. Callis

Since Specialization

Citations

This map shows the geographic impact of Thomas E. Callis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas E. Callis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas E. Callis more than expected).

Fields of papers citing papers by Thomas E. Callis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas E. Callis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas E. Callis. The network helps show where Thomas E. Callis may publish in the future.

Co-authorship network of co-authors of Thomas E. Callis

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas E. Callis. A scholar is included among the top collaborators of Thomas E. Callis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas E. Callis. Thomas E. Callis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Morales, Ana, Ren Sheng, Thomas E. Callis, et al.. (2023). Real-World Genetic Testing Utilization Among Patients With Cardiomyopathy. Circulation Genomic and Precision Medicine. 17(1). e004028–e004028. 2 indexed citations

Dellefave‐Castillo, Lisa, Allison L. Cirino, Thomas E. Callis, et al.. (2022). Assessment of the Diagnostic Yield of Combined Cardiomyopathy and Arrhythmia Genetic Testing. JAMA Cardiology. 7(9). 966–966. 36 indexed citations

Wang, Da Zhi, Sara R. Marques, Erin Kaltenbrun, et al.. (2021). The BMP pathway acts to directly regulate Tbx20 in the developing heart. UNC Libraries. 1 indexed citations

Nassar, Amin H., Sarah Abou Alaiwi, Saud H. AlDubayan, et al.. (2019). Prevalence of pathogenic germline cancer risk variants in high-risk urothelial carcinoma. Genetics in Medicine. 22(4). 709–718. 38 indexed citations

Kapplinger, Jamie D., Andrew S. Tseng, Benjamin A. Salisbury, et al.. (2015). Enhancing the Predictive Power of Mutations in the C-Terminus of the KCNQ1-Encoded Kv7.1 Voltage-Gated Potassium Channel. Journal of Cardiovascular Translational Research. 8(3). 187–197. 14 indexed citations

Kapplinger, Jamie D., Andrew P. Landstrom, J. Martijn Bos, et al.. (2014). Distinguishing Hypertrophic Cardiomyopathy-Associated Mutations from Background Genetic Noise. Journal of Cardiovascular Translational Research. 7(3). 347–361. 39 indexed citations

Seok, Heeyoung, Mariko Tatsuguchi, Thomas E. Callis, et al.. (2011). miR-155 Inhibits Expression of the MEF2A Protein to Repress Skeletal Muscle Differentiation. Journal of Biological Chemistry. 286(41). 35339–35346. 81 indexed citations

Kapplinger, Jamie D., Andrew P. Landstrom, Benjamin A. Salisbury, et al.. (2011). Distinguishing Arrhythmogenic Right Ventricular Cardiomyopathy/Dysplasia–Associated Mutations From Background Genetic Noise. Journal of the American College of Cardiology. 57(23). 2317–2327. 210 indexed citations

Callis, Thomas E., Brian C. Jensen, Karen E. Weck, & Monte S. Willis. (2010). Evolving molecular diagnostics for familial cardiomyopathies: at the heart of it all. Expert Review of Molecular Diagnostics. 10(3). 329–351. 26 indexed citations

10.

Callis, Thomas E., Kumar Pandya, Heeyoung Seok, et al.. (2009). MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice. Journal of Clinical Investigation. 119(9). 2772–2786. 679 indexed citations breakdown →

11.

Townley-Tilson, W. H. Davin, Thomas E. Callis, & Da‐Zhi Wang. (2009). MicroRNAs 1, 133, and 206: Critical factors of skeletal and cardiac muscle development, function, and disease. The International Journal of Biochemistry & Cell Biology. 42(8). 1252–1255. 298 indexed citations

12.

Tang, Ruhang, Xi‐Long Zheng, Thomas E. Callis, et al.. (2008). Myocardin inhibits cellular proliferation by inhibiting NF-κB(p65)-dependent cell cycle progression. Proceedings of the National Academy of Sciences. 105(9). 3362–3367. 97 indexed citations

13.

Callis, Thomas E. & Da‐Zhi Wang. (2008). Taking microRNAs to heart. Trends in Molecular Medicine. 14(6). 254–260. 92 indexed citations

14.

Chen, Jian‐Fu, Elizabeth P. Murchison, Ruhang Tang, et al.. (2008). Targeted deletion of Dicer in the heart leads to dilated cardiomyopathy and heart failure. Proceedings of the National Academy of Sciences. 105(6). 2111–2116. 453 indexed citations

15.

Chen, Jian‐Fu, Thomas E. Callis, & Da‐Zhi Wang. (2008). microRNAs and muscle disorders. Journal of Cell Science. 122(1). 13–20. 124 indexed citations

16.

Callis, Thomas E., Zhong‐Liang Deng, Jian‐Fu Chen, & Da‐Zhi Wang. (2008). Muscling Through the microRNA World. Experimental Biology and Medicine. 233(2). 131–138. 110 indexed citations

17.

Callis, Thomas E., Jian‐Fu Chen, & Da‐Zhi Wang. (2007). MicroRNAs in Skeletal and Cardiac Muscle Development. DNA and Cell Biology. 26(4). 219–225. 140 indexed citations

18.

Tatsuguchi, Mariko, Heeyoung Seok, Thomas E. Callis, et al.. (2007). Expression of microRNAs is dynamically regulated during cardiomyocyte hypertrophy. Journal of Molecular and Cellular Cardiology. 42(6). 1137–1141. 361 indexed citations

19.

Chen, Jian‐Fu, Elizabeth M. Mandel, J. Michael Thomson, et al.. (2005). The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation. Nature Genetics. 38(2). 228–233. 2258 indexed citations breakdown →

20.

Callis, Thomas E., Dongsun Cao, & Da‐Zhi Wang. (2005). Bone Morphogenetic Protein Signaling Modulates Myocardin Transactivation of Cardiac Genes. Circulation Research. 97(10). 992–1000. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact