Thomas C. Friedrich

10.2k total citations · 1 hit paper
153 papers, 4.7k citations indexed

About

Thomas C. Friedrich is a scholar working on Infectious Diseases, Epidemiology and Virology. According to data from OpenAlex, Thomas C. Friedrich has authored 153 papers receiving a total of 4.7k indexed citations (citations by other indexed papers that have themselves been cited), including 68 papers in Infectious Diseases, 60 papers in Epidemiology and 58 papers in Virology. Recurrent topics in Thomas C. Friedrich's work include HIV Research and Treatment (57 papers), Immune Cell Function and Interaction (42 papers) and Mosquito-borne diseases and control (25 papers). Thomas C. Friedrich is often cited by papers focused on HIV Research and Treatment (57 papers), Immune Cell Function and Interaction (42 papers) and Mosquito-borne diseases and control (25 papers). Thomas C. Friedrich collaborates with scholars based in United States, Uganda and Japan. Thomas C. Friedrich's co-authors include David H. O’Connor, Nancy A. Wilson, David I. Watkins, Jason T. Weinfurter, Austin L. Hughes, Levi Yant, Eva G. Rakasz, Jeffrey D. Lifson, Gemma E. May and Andrea M. Weiler and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Medicine and Nature Communications.

In The Last Decade

Thomas C. Friedrich

150 papers receiving 4.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Persistent SARS-CoV-2 infection: significance and implications

2024 69 citations Heather Machkovech, Peter Halfmann et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas C. Friedrich United States	41	2.0k	2.0k	1.7k	1.7k	643	153	4.7k
Miles W. Carroll United Kingdom	42	1.4k 0.7×	2.2k 1.1×	2.1k 1.2×	1.7k 1.0×	1.5k 2.4×	134	5.8k
Shan Lu United States	40	1.9k 1.0×	2.4k 1.2×	1.7k 1.0×	1.6k 1.0×	1.6k 2.5×	168	5.3k
Théodora Hatziioannou United States	40	3.7k 1.8×	2.3k 1.2×	2.9k 1.7×	2.4k 1.4×	1.9k 2.9×	65	7.3k
Margreet Bakker Netherlands	35	2.4k 1.2×	861 0.4×	2.3k 1.3×	1.0k 0.6×	731 1.1×	128	4.6k
Wing-Pui Kong United States	32	928 0.5×	1.9k 1.0×	2.4k 1.4×	2.4k 1.4×	1.4k 2.1×	40	5.4k
Charla Andrews United States	20	3.1k 1.5×	2.2k 1.1×	2.0k 1.1×	1.4k 0.8×	634 1.0×	30	4.8k
Elizabeth Bailes United Kingdom	26	3.1k 1.5×	847 0.4×	1.8k 1.1×	1.6k 0.9×	731 1.1×	33	4.4k
Hana Golding United States	49	2.7k 1.3×	4.3k 2.2×	2.0k 1.2×	3.5k 2.0×	1.8k 2.8×	184	8.2k
Yingying Li United States	36	2.1k 1.0×	929 0.5×	1.5k 0.9×	1.1k 0.6×	903 1.4×	88	4.1k

Countries citing papers authored by Thomas C. Friedrich

Since Specialization

Citations

This map shows the geographic impact of Thomas C. Friedrich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas C. Friedrich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas C. Friedrich more than expected).

Fields of papers citing papers by Thomas C. Friedrich

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas C. Friedrich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas C. Friedrich. The network helps show where Thomas C. Friedrich may publish in the future.

Co-authorship network of co-authors of Thomas C. Friedrich

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas C. Friedrich. A scholar is included among the top collaborators of Thomas C. Friedrich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas C. Friedrich. Thomas C. Friedrich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Chu, Winston T., Hui Wang, Marcelo A. Castro, et al.. (2025). Computed Tomography Radiomics and Machine Learning for Prediction of Histology-Based Hepatic Steatosis Scores. Diagnostics. 15(18). 2310–2310.

Machkovech, Heather, Nancy A. Wilson, Christina M. Newman, et al.. (2025). Amplicon sequencing of pasteurized retail dairy enables genomic surveillance of H5N1 avian influenza virus in United States cattle. PLoS ONE. 20(6). e0325203–e0325203. 1 indexed citations

Shi, Yujia, Saswati Bhattacharya, Andrea M. Weiler, et al.. (2023). Control of maternal Zika virus infection during pregnancy is associated with lower antibody titers in a macaque model. Frontiers in Immunology. 14. 1267638–1267638. 3 indexed citations

Jaeger, Anna S., Kasen K. Riemersma, John J. Baczenas, et al.. (2023). Gain without pain: adaptation and increased virulence of Zika virus in vertebrate host without fitness cost in mosquito vector. Journal of Virology. 97(10). e0116223–e0116223. 4 indexed citations

Braun, Katarina M., Luis A. Haddock, Chelsea M. Crooks, et al.. (2023). Avian H7N9 influenza viruses are evolutionarily constrained by stochastic processes during replication and transmission in mammals. Virus Evolution. 9(1). vead004–vead004. 4 indexed citations

Jaeger, Anna S., Andrea M. Weiler, James Weger‐Lucarelli, et al.. (2023). Wolbachia-mediated resistance to Zika virus infection in Aedes aegypti is dominated by diverse transcriptional regulation and weak evolutionary pressures. PLoS neglected tropical diseases. 17(10). e0011674–e0011674. 5 indexed citations

Braun, Katarina M., Gage K. Moreno, David H. O’Connor, et al.. (2022). Anti-membrane Antibodies Persist at Least One Year and Discriminate Between Past Coronavirus Disease 2019 Infection and Vaccination. The Journal of Infectious Diseases. 226(11). 1897–1902. 8 indexed citations

Braun, Katarina M., Gage K. Moreno, Molly A. Accola, et al.. (2021). Viral Sequencing to Investigate Sources of SARS-CoV-2 Infection in US Healthcare Personnel. Clinical Infectious Diseases. 73(6). e1329–e1336. 38 indexed citations

Braun, Katarina M., Gage K. Moreno, Peter Halfmann, et al.. (2021). Transmission of SARS-CoV-2 in domestic cats imposes a narrow bottleneck. PLoS Pathogens. 17(2). e1009373–e1009373. 62 indexed citations

10.

Hangartner, Lars, Eva G. Rakasz, Rebecca Nedellec, et al.. (2021). Effector function does not contribute to protection from virus challenge by a highly potent HIV broadly neutralizing antibody in nonhuman primates. Science Translational Medicine. 13(585). 26 indexed citations

11.

Caì, Yíngyún, Shuǐqìng Yú, Yīng Fāng, et al.. (2021). Development and Characterization of a cDNA-Launch Recombinant Simian Hemorrhagic Fever Virus Expressing Enhanced Green Fluorescent Protein: ORF 2b’ Is Not Required for In Vitro Virus Replication. Viruses. 13(4). 632–632. 7 indexed citations

12.

Sutton, Matthew S., Amy Ellis, Alexis J. Balgeman, et al.. (2019). CD8β Depletion Does Not Prevent Control of Viral Replication or Protection from Challenge in Macaques Chronically Infected with a Live Attenuated Simian Immunodeficiency Virus. Journal of Virology. 93(15). 7 indexed citations

13.

Ameel, Cassaundra L., Amy Ellis, Alexis J. Balgeman, et al.. (2018). Preexisting Simian Immunodeficiency Virus Infection Increases Susceptibility to Tuberculosis in Mauritian Cynomolgus Macaques. Infection and Immunity. 86(12). 17 indexed citations

14.

Sutton, Matthew S., Amy Ellis, Ryan V. Moriarty, et al.. (2018). Acute-Phase CD4 ⁺ T Cell Responses Targeting Invariant Viral Regions Are Associated with Control of Live Attenuated Simian Immunodeficiency Virus. Journal of Virology. 92(21). 8 indexed citations

15.

Lipsitch, Marc, William Barclay, Rahul Raman, et al.. (2016). Viral factors in influenza pandemic risk assessment. eLife. 5. 66 indexed citations

16.

Caì, Yíngyún, Elena Postnikova, John G. Bernbaum, et al.. (2014). Simian Hemorrhagic Fever Virus Cell Entry Is Dependent on CD163 and Uses a Clathrin-Mediated Endocytosis-Like Pathway. Journal of Virology. 89(1). 844–856. 41 indexed citations

17.

Greene, Justin, Benjamin J. Burwitz, Melisa L. Budde, et al.. (2010). Extralymphoid CD8 ⁺ T Cells Resident in Tissue from Simian Immunodeficiency Virus SIVmac239Δnef-Vaccinated Macaques Suppress SIVmac239 Replication Ex Vivo. Journal of Virology. 84(7). 3362–3372. 23 indexed citations

18.

Sacha, Jonah B., Juan P. Giraldo‐Vela, Matthew B. Buechler, et al.. (2009). Gag- and Nef-specific CD4 ⁺ T cells recognize and inhibit SIV replication in infected macrophages early after infection. Proceedings of the National Academy of Sciences. 106(24). 9791–9796. 73 indexed citations

19.

Sacha, Jonah B., Matthew R. Reynolds, Matthew B. Buechler, et al.. (2008). Differential Antigen Presentation Kinetics of CD8 ⁺ T-Cell Epitopes Derived from the Same Viral Protein. Journal of Virology. 82(18). 9293–9298. 22 indexed citations

20.

Friedrich, Thomas C., et al.. (2007). Einfühlung und phänomenologische Reduktion : Grundlagentexte zu Architektur, Design und Kunst. Lit eBooks. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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