Taro Misaki

749 total citations
28 papers, 503 citations indexed

About

Taro Misaki is a scholar working on Nephrology, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Taro Misaki has authored 28 papers receiving a total of 503 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Nephrology, 10 papers in Molecular Biology and 6 papers in Pathology and Forensic Medicine. Recurrent topics in Taro Misaki's work include Renal Diseases and Glomerulopathies (14 papers), Oral microbiology and periodontitis research (6 papers) and Streptococcal Infections and Treatments (6 papers). Taro Misaki is often cited by papers focused on Renal Diseases and Glomerulopathies (14 papers), Oral microbiology and periodontitis research (6 papers) and Streptococcal Infections and Treatments (6 papers). Taro Misaki collaborates with scholars based in Japan and United States. Taro Misaki's co-authors include Kazuhiko Nakano, Ryota Nomura, Shuhei Naka, Naro Ohashi, Akira Hishida, Yasuyuki Nagasawa, Seigo Ito, Michiyo Matsumoto‐Nakano, Yoshihide Fujigaki and Akiko Fukunaga and has published in prestigious journals such as PLoS ONE, Scientific Reports and International Journal of Molecular Sciences.

In The Last Decade

Taro Misaki

26 papers receiving 501 citations

Peers

Taro Misaki
Taro Misaki
Citations per year, relative to Taro Misaki Taro Misaki (= 1×) peers Kiyoshi Nakazono

Countries citing papers authored by Taro Misaki

Since Specialization
Citations

This map shows the geographic impact of Taro Misaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Taro Misaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Taro Misaki more than expected).

Fields of papers citing papers by Taro Misaki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Taro Misaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Taro Misaki. The network helps show where Taro Misaki may publish in the future.

Co-authorship network of co-authors of Taro Misaki

This figure shows the co-authorship network connecting the top 25 collaborators of Taro Misaki. A scholar is included among the top collaborators of Taro Misaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Taro Misaki. Taro Misaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ito, Seigo, Taro Misaki, Shuhei Naka, et al.. (2025). cnm-positive Streptococcus mutans, a major pathogen in dental caries, may exacerbate IgA nephropathy via the tonsils. Clinical and Experimental Nephrology. 29(10). 1400–1408.
2.
Naka, Shuhei, Taro Misaki, Yasuyuki Nagasawa, et al.. (2024). Contribution of collagen-binding protein Cnm of Streptococcus mutans to induced IgA nephropathy-like nephritis in rats. Communications Biology. 7(1). 1141–1141. 3 indexed citations
3.
Misaki, Taro, Shuhei Naka, Hitoshi Suzuki, et al.. (2023). cnm-positive Streptococcus mutans is associated with galactose-deficient IgA in patients with IgA nephropathy. PLoS ONE. 18(3). e0282367–e0282367. 6 indexed citations
4.
Ito, Seigo, Taro Misaki, Yasuyuki Nagasawa, et al.. (2023). Porphyromonas gingivalis infection in the oral cavity is associated with elevated galactose-deficient IgA1 and increased nephritis severity in IgA nephropathy. Clinical and Experimental Nephrology. 28(3). 192–200. 2 indexed citations
5.
Naka, Shuhei, Taro Misaki, Yasuyuki Nagasawa, et al.. (2022). Cnm of Streptococcus mutans is important for cell surface structure and membrane permeability. Frontiers in Cellular and Infection Microbiology. 12. 994014–994014. 11 indexed citations
6.
Nomura, Ryota, Yasuyuki Nagasawa, Taro Misaki, et al.. (2022). Distribution of periodontopathic bacterial species between saliva and tonsils. Odontology. 111(3). 719–727. 7 indexed citations
7.
Misaki, Taro, Shuhei Naka, Yasuyuki Nagasawa, et al.. (2022). Simultaneous Presence of Campylobacter rectus and Cnm-Positive Streptococcus mutans in the Oral Cavity Is Associated with Renal Dysfunction in IgA Nephropathy Patients: 5-Year Follow-Up Analysis. ˜The œNephron journals/Nephron journals. 147(3-4). 134–143. 5 indexed citations
8.
Ito, Daisuke, et al.. (2022). A rare case of proliferative glomerulonephritis with monoclonal IgG2 kappa deposit: a case report. BMC Nephrology. 23(1). 396–396.
9.
Naka, Shuhei, Taro Misaki, Seigo Ito, et al.. (2021). Streptococcus mutans induces IgA nephropathy-like glomerulonephritis in rats with severe dental caries. Scientific Reports. 11(1). 5784–5784. 26 indexed citations
10.
Misaki, Taro, Akiko Fukunaga, & Kazuhiko Nakano. (2020). Dental caries status is associated with arteriosclerosis in patients on hemodialysis. Clinical and Experimental Nephrology. 25(1). 87–93. 7 indexed citations
11.
Ito, Seigo, Taro Misaki, Shuhei Naka, et al.. (2019). Specific strains of Streptococcus mutans, a pathogen of dental caries, in the tonsils, are associated with IgA nephropathy. Scientific Reports. 9(1). 20130–20130. 38 indexed citations
12.
Misaki, Taro, Akiko Fukunaga, Yoshitaka Shimizu, Akira Ishikawa, & Kazuhiko Nakano. (2019). Possible link between dental diseases and arteriosclerosis in patients on hemodialysis. PLoS ONE. 14(12). e0225038–e0225038. 14 indexed citations
13.
Misaki, Taro, Shuhei Naka, Yasuyuki Nagasawa, et al.. (2018). <b><i>Campylobacter rectus</i></b> in the Oral Cavity Correlates with Proteinuria in Immunoglobulin A Nephropathy Patients. ˜The œNephron journals/Nephron journals. 139(2). 143–149. 17 indexed citations
14.
Misaki, Taro, et al.. (2016). Presence of Streptococcus mutans strains harbouring the cnm gene correlates with dental caries status and IgA nephropathy conditions. Scientific Reports. 6(1). 36455–36455. 42 indexed citations
15.
Misaki, Taro, et al.. (2014). A case of anaphylactoid reaction to acetate in acetate-containing bicarbonate dialysate. CEN Case Reports. 4(1). 81–84. 3 indexed citations
16.
Wang, Hong‐Hui, Taro Misaki, Vanessa Taupin, et al.. (2014). GIV/Girdin Links Vascular Endothelial Growth Factor Signaling to Akt Survival Signaling in Podocytes Independent of Nephrin. Journal of the American Society of Nephrology. 26(2). 314–327. 44 indexed citations
17.
Suzuki, Sayuri, Hirotaka Fukasawa, Taro Misaki, et al.. (2012). The Amelioration of Renal Damage in Skp2-Deficient Mice Canceled by p27 Kip1 Deficiency in Skp2−/− p27−/− Mice. PLoS ONE. 7(4). e36249–e36249. 14 indexed citations
18.
Suzuki, Sayuri, Hirotaka Fukasawa, Taro Misaki, et al.. (2011). Up-regulation of Cks1 and Skp2 with TNFα/NF-κB signaling in chronic progressive nephropathy. Genes to Cells. 16(11). 1110–1120. 16 indexed citations
19.
Fukasawa, Hirotaka, Tatsuo Yamamoto, Yoshihide Fujigaki, et al.. (2010). Reduction of transforming growth factor‐β type II receptor is caused by the enhanced ubiquitin‐dependent degradation in human renal cell carcinoma. International Journal of Cancer. 127(7). 1517–1525. 29 indexed citations
20.
Misaki, Taro, Tatsuo Yamamoto, Sayuri Suzuki, et al.. (2009). Decrease in Tumor Necrosis Factor-α Receptor-Associated Death Domain Results from Ubiquitin-Dependent Degradation in Obstructive Renal Injury in Rats. American Journal Of Pathology. 175(1). 74–83. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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