Tal Keren‐Raifman
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- Neuroscience and Neuropharmacology Research 7
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- Ion channel regulation and function 14
- Receptor Mechanisms and Signaling 13
- Protein Kinase Regulation and GTPase Signaling 3
- Retinal Development and Disorders 1
- Signaling Pathways in Disease 1
- Ubiquitin and proteasome pathways 1
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- Cardiac electrophysiology and arrhythmias 6
- Co-authors
- Nathan DascalTatiana IvaninaCarmen DessauerVladlen Z. SlepakSagit PelegShai BerlinKonstantin LevayAmal Kanti Bera
- Partner nations
- IsraelUnited StatesAustria
In The Last Decade
Tal Keren‐Raifman
18 papers receiving 464 citations
Peers
Comparison fields: 5 of 50
- Cellular and Molecular Neuroscience 289
- Molecular Biology 430
- Cardiology and Cardiovascular Medicine 103
- Physiology 9
- Sensory Systems 7
Countries citing papers authored by Tal Keren‐Raifman
This map shows the geographic impact of Tal Keren‐Raifman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tal Keren‐Raifman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tal Keren‐Raifman more than expected).
Fields of papers citing papers by Tal Keren‐Raifman
This network shows the impact of papers produced by Tal Keren‐Raifman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tal Keren‐Raifman. The network helps show where Tal Keren‐Raifman may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Tal Keren‐Raifman, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 9 | |
| 2 | 2019 | 9 | |
| 3 | 2015 | 12 | |
| 4 | 2013 | 7 | |
| 5 | 2012 | 11 | |
| 6 | 2011 | 32 | |
| 7 | 2009 | 34 | |
| 8 | 2009 | 39 | |
| 9 | 2008 | 16 | |
| 10 | 2007 | 30 | |
| 11 | 2004 | 61 | |
| 12 | 2003 | 40 | |
| 13 | 2002 | 17 | |
| 14 | 2001 | 24 | |
| 15 | 2001 | 38 | |
| 16 | 2000 | 2 | |
| 17 | 2000 | 46 | |
| 18 | 1999 | 44 |
About Tal Keren‐Raifman
Tal Keren‐Raifman is a scholar working on Cellular and Molecular Neuroscience, Cardiology and Cardiovascular Medicine and Molecular Biology, having authored 18 papers that have together received 471 indexed citations. Recurring topics across this work include Ion channel regulation and function (14 papers), Receptor Mechanisms and Signaling (13 papers), Neuroscience and Neuropharmacology Research (7 papers), Cardiac electrophysiology and arrhythmias (6 papers), Protein Kinase Regulation and GTPase Signaling (3 papers), Retinal Development and Disorders (1 paper), Signaling Pathways in Disease (1 paper) and Ubiquitin and proteasome pathways (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (289 citations), Molecular Biology (430 citations) and Cardiology and Cardiovascular Medicine (103 citations). Tal Keren‐Raifman has collaborated with scholars based in Israel, United States and Austria. Frequent co-authors include Nathan Dascal, Tatiana Ivanina, Carmen Dessauer, Vladlen Z. Slepak, Sagit Peleg, Shai Berlin, Konstantin Levay, Amal Kanti Bera, Ida Rishal and Moran Rubinstein. Their work appears in journals such as Journal of Biological Chemistry, Biochemistry, iScience, Scientific Reports and PLoS Computational Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.